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Human WNT5A ELISA Kit for Sandwich ELISA - ABIN481725
Schulte, Müller, Neumann, Oberhäuser, Faust, Güdelhöfer, Brandt, Krone, Laudes: Pro-inflammatory wnt5a and anti-inflammatory sFRP5 are differentially regulated by nutritional factors in obese human subjects. in PLoS ONE 2012
Show all 9 references for ABIN481725
Mouse (Murine) WNT5A ELISA Kit for Sandwich ELISA - ABIN837310
Dawes, Sugiyama, Lovicu, Harris, Shelley, McAvoy: Interactions between lens epithelial and fiber cells reveal an intrinsic self-assembly mechanism. in Developmental biology 2014
Show all 5 references for ABIN837310
There is an important role of Wnt5a in kidney development. Disrupted Wnt5a results in kidney cysts in zebrafish and pleiotropic abnormal kidney development in mice.
Wnt5a acts as a chemoattractant in the emerging limb bud where it contributes to the establishment of cell polarity that is likely to underlie the oriented cell behaviours
the CaMKK (show CAMKK2 ELISA Kits)-CaMKIalpha (show CAMK1 ELISA Kits) cascade is required for the axonal growth effect of Wnt5a during neuronal polarization.
Decreased expressions of Wnt1 (show WNT1 ELISA Kits) and Wnt5a.
indicate that restricted expression of Wnt5a in the caudal (show CAD ELISA Kits) SpM (show NPC1 ELISA Kits) is essential for normal OFT morphogenesis, and uncover a novel function of spatially regulated cell cohesion by Wnt5a in driving the deployment of SHF cells from the SpM (show NPC1 ELISA Kits) into the OFT
Findings indicate wingless-related MMTV integration site 5A protein (WNT5a) as a key regulator of follicle development and gonadotropin responsiveness.
The oxidative stress of diabetes mellitus impairs Wnt (show WNT2 ELISA Kits) signaling and causes cardiac outlet defects that are rescued by SOD1 (show SOD1 ELISA Kits) overexpression
The results in a potent increase in odontoblastic cell differentiation, indicating the unique involvement of Atg5 (show ATG5 ELISA Kits), autophagy and Wnt5 signaling in CS/BMP-4 (show BMP4 ELISA Kits)-induced differentiation of ES cells into odontoblast-like cells, at a relatively early stage.
the Wnt5a/Frizzled-2 (show FZD2 ELISA Kits) axis suppresses beta-catenin (show CTNNB1 ELISA Kits) signaling in hepatocytes in an autocrine manner, thereby contributing to timely conclusion of the liver regeneration process
effects on hypoxic pulmonary hypertension by regulating pulmonary vascular remodeling and right ventricular hypertrophy via reduction in beta-catenin (show CTNNB1 ELISA Kits)/cyclin D1 (show CCND1 ELISA Kits) signaling
Wnt5a regulates the pathogenesis of gp120 (show ITIH4 ELISA Kits)-induced pain.
Study identified a novel Wnt5a/Fzd4 (show FZD4 ELISA Kits) signaling pathway that contributes to the regulation of dendrite morphogenesis
Xenopus adult stem cells originate from the larval absorptive cells expressing Ror2 (show ROR2 ELISA Kits), which require Wnt5a/Ror2 (show ROR2 ELISA Kits) signaling for their dedifferentiation accompanied by changes in cell morphology.
Data show that PAPC (show PCDH8 ELISA Kits) signaling via RhoA (show RHOA ELISA Kits) and Wnt5a/Ror2 (show ROR2 ELISA Kits) activity are required to keep cells aligned in apical-basal orientation during invagination of the ear placode.
Data show that Rspo3 (show RSPO3 ELISA Kits) binds syndecan 4 and that together they activate Wnt5a/PCP (show PRCP ELISA Kits) signaling.
In an examination of signaling pathways in developing Xenopus lung, wnt5a was expressed in the mesenchyme layer of the entire lungs through stages 39-41.
Wnt5a reduces cell surface levels and promotes ubiquitination and degradation of SDC4 (show SDC4 ELISA Kits) in dorsal mesodermal cells from Xenopus gastrulae.
Transcriptional regulation of XPAPC (show PCDH8 ELISA Kits) by XWnt-5A requires the receptor tyrosine kinase Ror2 (show ROR2 ELISA Kits).
Xwnt-5a plays an instructive role in larval tail regeneration via Wnt (show WNT2 ELISA Kits)/JNK (show MAPK8 ELISA Kits) signaling
these findings suggest that miR154 functions as a tumor suppressor in osteosarcoma by partially suppressing Wnt5a expression.
Studied the roles of WNT4 (show WNT4 ELISA Kits) and WNT5A in human decidulization and their relationship with preeclampsia.
High WNT5A and WNT5B expression strongly associates with unmutated IGHV.
Wnt5a is increased and beta-catenin (show CTNNB1 ELISA Kits) is decreased in parenchyma from idiopathic pulmonary fibrosis patients.
results corroborated previous findings of Ryk (show RYK ELISA Kits)-mediated Wnt5a effect, and suggested a role for Ror2 (show ROR2 ELISA Kits) in the Wnt5a machinery in glioblastoma
WNT5A participates in the biology of odontogenic tumors and inflammatory cyst but not in developmental cyst.
Mutation in WNT5A may serve as a susceptibility factor for Congenital Anomalies Of Kidney And Urinary Tract.
Wnt5a-Ror2 (show ROR2 ELISA Kits) signaling enhances expression and secretion of CXCL16 (show CXCL16 ELISA Kits) in mesenchymal stem cells thereby activating CXCR6 (show CXCR6 ELISA Kits) expressed on tumor cells to promote proliferation.
The homozygous genotypes (CC for rs524153 and GG for rs504849) were associated with a lower WNT5A transcriptional level compared with the transcriptional level of those with wild-type genotypes.
Epigenetic inactivation of the WNT5A promoter B involves both DNA methylation (show HELLS ELISA Kits) and histone modifications. Differential expression of the WNT5A alternative promoters A and B is a characteristic of osteosarcomas.
WNT5A is a negative regulator of FSH (show BRD2 ELISA Kits)-stimulated granulosa cell steroidogenesis in cattle.
Wnt-5A may be associated with cartilage destruction by promoting the expression of matrix metalloproteinases.
The WNT gene family consists of structurally related genes which encode secreted signaling proteins. These proteins have been implicated in oncogenesis and in several developmental processes, including regulation of cell fate and patterning during embryogenesis. This gene encodes a member of the WNT family that signals through both the canonical and non-canonical WNT pathways. This protein is a ligand for the seven transmembrane receptor frizzled-5 and the tyrosine kinase orphan receptor 2. This protein plays an essential role in regulating developmental pathways during embryogenesis. This protein may also play a role in oncogenesis. Mutations in this gene are the cause of autosomal dominant Robinow syndrome. Alternate splicing results in multiple transcript variants.
wingless-type MMTV integration site family, member 5A
, protein Wnt-5a
, protein Wnt-5a-like
, WNT-5A protein
, Wnt-5a protein
, wingless-related MMTV integration site 5A
, wingless-type MMTV integration site 5A