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This locus represents a small inducible cytokine. Additionally we are shipping CCL3 Proteins (106) and CCL3 Kits (98) and many more products for this protein.
Showing 10 out of 278 products:
Human Polyclonal CCL3 Primary Antibody for EIA, WB - ABIN453560
Hirashima, Ono, Nakao, Nishi, Kimura, Nomiyama, Hamada, Yoshida, Shimada: Nucleotide sequence of the third cytokine LD78 gene and mapping of all three LD78 gene loci to human chromosome 17. in DNA sequence : the journal of DNA sequencing and mapping 1993
Show all 2 references for ABIN453560
Mouse (Murine) Polyclonal CCL3 Primary Antibody for IP, WB - ABIN223561
Neff, Zgraggen, Neff, Jamnicki-Abegg, Suter, Schimmer, Booy, Joch, Pasch, Ward, Beck-Schimmer: Inflammatory response of tracheobronchial epithelial cells to endotoxin. in American journal of physiology. Lung cellular and molecular physiology 2005
Show all 2 references for ABIN223561
Human Polyclonal CCL3 Primary Antibody for IHC (p), WB - ABIN652466
Wang, Lin, Izumi, Jiang, Lai, Xu, Fang, Lu, Li, Xia, Chang: Increased infiltrated macrophages in benign prostatic hyperplasia (BPH): role of stromal androgen receptor in macrophage-induced prostate stromal cell proliferation. in The Journal of biological chemistry 2012
Show all 2 references for ABIN652466
Human Monoclonal CCL3 Primary Antibody for ELISA, WB - ABIN520044
Quek, Ho, Loprieno, Ellis, Elliott, Liu: A multiplex assay to measure RNA transcripts of prostate cancer in urine. in PLoS ONE 2012
Human Monoclonal CCL3 Primary Antibody for EIA, Func - ABIN115769
Xia, Qin, Wu, Mackay, Hyman: Immunohistochemical study of the beta-chemokine receptors CCR3 and CCR5 and their ligands in normal and Alzheimer's disease brains. in The American journal of pathology 1998
Human Monoclonal CCL3 Primary Antibody for EIA, WB - ABIN114723
Randolph, Furie: A soluble gradient of endogenous monocyte chemoattractant protein-1 promotes the transendothelial migration of monocytes in vitro. in Journal of immunology (Baltimore, Md. : 1950) 1995
Human Polyclonal CCL3 Primary Antibody for ELISA, WB - ABIN185352
Gonzalez, Kulkarni, Bolivar, Mangano, Sanchez, Catano, Nibbs, Freedman, Quinones, Bamshad, Murthy, Rovin, Bradley, Clark, Anderson, Oconnell, Agan, Ahuja, Bologna, Sen, Dolan, Ahuja: The influence of CCL3L1 gene-containing segmental duplications on HIV-1/AIDS susceptibility. in Science (New York, N.Y.) 2005
The residues on the N-loop and beta-sheets of MIP-1a are close to both CCR1 and CCR5 (show CCR5 Antibodies), and those in the C-terminal helix region are close to CCR5 (show CCR5 Antibodies).
CCL2 (show CCL2 Antibodies), 3, 14 have roles in stimulating macrophage bone marrow homing, proliferation, and polarization in multiple myeloma
An increased MIP-1alpha level in nasopharyngeal aspirates from patients with acute respiratory symptoms during the first wheezing episode caused by viral infections might predict recurrent wheezing.
Tregs from subjects with established type 1 diabetes were impaired in their ability to produce CCL3 and CCL4 (show CCL4 Antibodies).
Pleural and serum MIP1a were lower in patients with lung cancer compared to lung metastasis, pneumonia, tuberculosis, and transudate pleural effusion.
AEG-1 (show MTDH Antibodies) mediates CCL3/CCR5 (show CCR5 Antibodies)-induced EMT (show ITK Antibodies) development via both Erk1/2 (show MAPK1/3 Antibodies) and Akt (show AKT1 Antibodies) signaling pathway in CM patients, which indicates CCL3/CCR5 (show CCR5 Antibodies)-AEG-1 (show MTDH Antibodies)-EMT (show ITK Antibodies) pathway could be suggested as a useful target to affect the progression of CM.
The EP in TKA differed from EP in aseptically failed THA by lower CCL3 and DC-STAMP (show TM7SF4 Antibodies) mRNA and protein expression. EP of all studied inflammatory and osteoclastogenic molecules were similar in knee and hip OA.
A significantly higher percentage of healthy asymptomatic persons possibly exposed to asbestos had detectable levels of serum CCL3 compared to healthy unexposed control subjects.
APOE (show APOE Antibodies) varepsilon4 genotype specifically modulates astrocyte secretion of potent microglial chemotactic agents, including CCL3
Macrophage inflammatory protein-1alpha shows predictive value as a risk marker for subjects and sites vulnerable to bone loss in a longitudinal model of aggressive periodontitis.
these data suggest that the chemokine (show CCL1 Antibodies) CCL3 is an hippocampal neuromodulator able to regulate synaptic plasticity mechanisms involved in learning and memory functions.
Ccl3 mRNAs increased within 5 h after injury in mouse cortical slices.
Previous exposure to noninjurious ventilation induces a state of tolerance to ventilator-induced lung injury. Down regulation of the chemokine (show CCL1 Antibodies) gene Ccl3 could be the mechanism responsible for this effect.
the tissue fibrinolytic system contributes to the induction of macrophage recruitment and CCL3 at the bone injury site, thereby, leading to the enhancement of the repair process.
The chemokines monocyte chemotactic protein 1 (MCP1 (show CCL2 Antibodies)), MIP1alpha, MIP1beta (show CCL4 Antibodies), interferon gamma (show IFNG Antibodies)-induced protein 10 (IP-10 (show CXCL10 Antibodies)), and eotaxin (show CCL11 Antibodies) were induced in Saa1 (show SAA1 Antibodies) TG mice.
In experimental autoimmune encephalomyelitis there was a significant increase in activation of astrocytes with increased MIP-1alpha expression.
Aged mice had similar levels of IL-1beta (show IL1B Antibodies), TNF (show TNF Antibodies), IFN-gamma (show IFNG Antibodies), IL-17 (show IL17A Antibodies), and granulocyte colony-stimulating factor (show CSF3 Antibodies) following S. pneumoniae infection, compared with young mice, but increased levels of the chemokines CXCL9 (show CXCL9 Antibodies), CXCL12 (show CXCL12 Antibodies), CCL3, CCL4 (show CCL4 Antibodies), CCL5 (show CCL5 Antibodies), CCL11 (show CCL11 Antibodies), and CCL17 (show CCL17 Antibodies).
Negative modulation of PGE2 signaling reduced infection-induced anti-inflammatory cytokine polarization and enhanced inflammatory chemokines, CCL3 and CCL5 (show CCL5 Antibodies).
Granzyme M (show GZMM Antibodies) regulates the release of natural killer cell MIP-1alpha to initiate innate immune responses.
variation in the CCL18 (show CCL18 Antibodies)-CCL3-CCL4 (show CCL4 Antibodies) chemokine (show CCL1 Antibodies) gene cluster influences HIV Type 1 transmission and AIDS disease progression
CCL3, CCL4 (show CCL4 Antibodies) and CCL5 (show CCL5 Antibodies) gene expression was evaluated in response to simian-human immunodeficiency virus (SHIV) infection in a rhesus macaque model.
Stress decreased MIP-1alpha gene expression in both CeA (show CEACAM3 Antibodies) lesioned and non-lesioned animals. But, the CeA (show CEACAM3 Antibodies) lesions increased the tissue expression of MIP-1alpha mRNA prior to and after stress exposure.
This locus represents a small inducible cytokine. The encoded protein, also known as macrophage inflammatory protein 1 alpha, plays a role in inflammatory responses through binding to the receptors CCR1, CCR4 and CCR5. Polymorphisms at this locus may be associated with both resistance and susceptibility to infection by human immunodeficiency virus type 1.
C-C motif chemokine 3
, macrophage inflammatory protein 1 alpha
, G0/G1 switch regulatory protein 19-1
, PAT 464.1
, macrophage inflammatory protein 1-alpha
, small inducible cytokine A3 (homologous to mouse Mip-1a)
, tonsillar lymphocyte LD78 alpha protein
, Macrophage inflammatory protein 1 alpha (Small inducible cytokine A3)
, small inducible cytokine A3
, small-inducible cytokine A3
, MIP-1 alpha
, MIP1 (a)
, heparin-binding chemotaxis protein
, macrophage inflammatory protein-1alpha
, chemokine (C-C motif) ligand 3-like 1
, CC chemokine ligand 3
, chemokine (C-C motif) ligand 3-like 3
, CCL3-like 1