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CCL5 is one of several CC cytokine genes clustered on the q-arm of chromosome 17. Additionally we are shipping CCL5 Proteins (114) and CCL5 Kits (101) and many more products for this protein.
Showing 10 out of 307 products:
Human Monoclonal CCL5 Primary Antibody for FACS, IHC - ABIN4900684
van Bladel, Roest, de Groot, Schutgens: Up-regulation of platelet activation in hemophilia A. in Haematologica 2011
Show all 7 references for ABIN4900684
Human Monoclonal CCL5 Primary Antibody for EIA, Func - ABIN118992
Conway, Sahni, Szkopek: Plasmonic interconnects versus conventional interconnects: a comparison of latency, crosstalk and energy costs. in Optics express 2009
Show all 6 references for ABIN118992
Human Monoclonal CCL5 Primary Antibody for FACS - ABIN2657999
von Luettichau, Nelson, Pattison, van de Rijn, Huie, Warnke, Wiedermann, Stahl, Sibley, Krensky: RANTES chemokine expression in diseased and normal human tissues. in Cytokine 1996
Show all 4 references for ABIN2657999
Human Polyclonal CCL5 Primary Antibody for IHC, IHC (fro) - ABIN446965
Sakamoto, Tsuruda, Hatakeyama, Imamura, Asada, Kitamura: Impact of age-dependent adventitia inflammation on structural alteration of abdominal aorta in hyperlipidemic mice. in PLoS ONE 2014
Show all 3 references for ABIN446965
Human Monoclonal CCL5 Primary Antibody for FACS - ABIN4898185
Ferrarini, Delfanti, Gianolini, Rizzi, Alfano, Lazzarin, Biswas: NF-kappa B modulates sensitivity to apoptosis, proinflammatory and migratory potential in short- versus long-term cultured human gamma delta lymphocytes. in Journal of immunology (Baltimore, Md. : 1950) 2008
Show all 3 references for ABIN4898185
Cow (Bovine) Polyclonal CCL5 Primary Antibody for FACS, IF (p) - ABIN674949
Pattappa, Peroglio, Sakai, Mochida, Benneker, Alini, Grad: CCL5/RANTES is a key chemoattractant released by degenerative intervertebral discs in organ culture. in European cells & materials 2014
Show all 3 references for ABIN674949
Human Monoclonal CCL5 Primary Antibody for FACS - ABIN4898184
Tikhonov, Deetz, Paca, Berg, Lukyanenko, Lim, Pauza: Human Vgamma2Vdelta2 T cells contain cytoplasmic RANTES. in International immunology 2006
Show all 2 references for ABIN4898184
Chicken Polyclonal CCL5 Primary Antibody for WB - ABIN2779989
Palaniappan, Singh, Singh, Singh, Ades, Briles, Hollingshead, Royal, Sampson, Stiles, Taub, Lillard: CCL5 modulates pneumococcal immunity and carriage. in Journal of immunology (Baltimore, Md. : 1950) 2006
Mouse (Murine) Polyclonal CCL5 Primary Antibody for EIA, WB - ABIN116261
Gonzalez-Rey, Chorny, Robledo, Delgado: Cortistatin, a new antiinflammatory peptide with therapeutic effect on lethal endotoxemia. in The Journal of experimental medicine 2006
MCP-1 (show CCL2 Antibodies) and RANTES levels (ng/mg total protein) of gingivitis group were higher than those of the MSH groups (P=0.005, P=0.0001, respectively).
There were no associations of CCL5 gene promoter polymorphism with the risk of diabetic microvascular complications (DMI); However, the 59029A polymorphism in CCR5 (show CCR5 Antibodies) might affect individual susceptibility for DMI [Meta-Analysis]
RANTES Gene Polymorphisms are Associated with HIV-1 Infections.
Through the self-production of CCL5, ovarian cancer stem-like cells are induced to differentiate into endothelial cells and participate in tumor angiogenesis.
monocytes and lymphocytes cooperate to enhance migration towards CXCR3 (show CXCR3 Antibodies) chemokines and CCL5 in COPD (show ARCN1 Antibodies)
In perennial allergic rhinitis patients, we found positive correlation between RANTES levels and nasal symptom score and eosinophil counts. In patients with non-allergic rhinitis with eosinophilia syndrome, the concentrations of MCP-1 (show CCL2 Antibodies) and RANTES were associated with nasal symptom score and percentage of eosinophils.
Oral lipid loading is linked to reduced circulating pro-inflammatory chemokines CCL2 (show CCL2 Antibodies), IP-10 (show CXCL10 Antibodies), and chemerin (show RARRES2 Antibodies) and to increased RANTES levels, suggesting that dietary lipids affect immune function.
Results indicated that cisplatin-induced CCL5 secretion derived from the cancer-associated fibroblasts may promote cisplatin resistance, which was mediated by regulation of the STAT3 (show STAT3 Antibodies) and PI3K (show PIK3CA Antibodies)/Akt (show AKT1 Antibodies) signal pathways.
Authors demonstrate that the inhibition of the CCL5 expression is the cause of the observed effects resulting from RKIP (show PEBP1 Antibodies) expression. Results underscore the significance of RKIP (show PEBP1 Antibodies) as important negative regulator of tumor microenvironment.
The N termini of CC chemokines are shown to be involved in receptor binding and oligomerization. We also report an alternative CCL3 (show CCL3 Antibodies) oligomer structure that reveals how conformational changes in CCL3 (show CCL3 Antibodies) N termini profoundly alter its surface properties and dimer-dimer interactions to affect GAG binding and oligomerization. Such complexity in oligomerization and GAG binding enables intricate, physiologically relevant regulatio
RANTES produced by renal tubular cells act as a key chemokine (show CCL1 Antibodies) in acute kidney injury and HIF-1alpha (show HIF1A Antibodies) regulated LncRNA-PRINS might be involved in RANTES production.
Mycobacterium chelonae activates the gene expressions of chemokine (C-C motif) ligand 2 (CCL2 (show CCL2 Antibodies)) and CCL5 in murine bone marrow-derived macrophages and in vivo mouse model.
Neutralization of circulating RANTES decreased liver neutrophilic infiltration
CCL5 signaling through CCR5 (show CCR5 Antibodies) may increase platelet counts during physiological stress.
An intriguing finding was that S1P (show S1PR1 Antibodies) induced c-Fos-inhibited CCL5 directly and also indirectly through inhibition of the IFN-beta (show IFNB1 Antibodies) amplification loop
platelet-derived CCL5 is an important link between platelet activation and neutrophil recruitment in acute colitis
Nuclear FAK (show PTK2 Antibodies) is associated with chromatin and exists in complex with transcription factors and their upstream regulators that control Ccl5 expression. Furthermore, FAK's immuno-modulatory nuclear activities may be specific to cancerous squamous epithelial cells, as normal keratinocytes do not have nuclear FAK (show PTK2 Antibodies).
that mast cells, recruited towards CCL2 and CCL5 expressed by epithelium induced to proliferate by Human Papillomavirus 16 E7 protein
PRDX6 (show PRDX6 Antibodies) promotes lung tumor development via its mediated and CCL5-associated activation of the JAK2 (show JAK2 Antibodies)/STAT3 (show STAT3 Antibodies) pathway.
tumours are characterized by expression of inflammatory chemokines (CCL2 (show CCL2 Antibodies), CCL5, CCL7 (show CCL7 Antibodies), CCL8 (show CCL8 Antibodies), CCL12 (show Ccl12 Antibodies), CXCL9 (show CXCL9 Antibodies), CXCL10 (show CXCL10 Antibodies) and CX3CL1 (show CX3CL1 Antibodies)), reflected by an enrichment of activated Foxp3 (show FOXP3 Antibodies)(-) and Foxp3 (show FOXP3 Antibodies)(+) T cells
These results represent an important molecular mechanism whereby H. parasuis induced RANTES in the inflammatory response.
Foot-and-mouth disease virus leader proteinase inhibits dsRNA-induced RANTES transcription.
we cloned the nucleotide sequence of the 5'-flanking region of the porcine RANTES (poRANTES) gene and characterized the regulatory elements that activate transcription
The Toll (show TLR4 Antibodies)-like receptor signaling pathway is involved in porcine reproductive and respiratory syndrome virus-induced RANTES activation.
These results suggest that porcine RANTES can play an important role in xenotransplant rejection, through participating in the interaction between porcine endothelial cells and human monocytes.
CCL5 but not CCL2 (show CCL2 Antibodies) mainly attract bovine classical monocytes and promote their differentiation into LPS (show IRF6 Antibodies)-hypo-responsive macrophages.
decrease in sensitivity of HIV variants to RANTES neutralization during the course of progressive infection, but not during follow-up of long term survivors; data suggest a role for RANTES neutralization sensitivity of HIV-1 in AIDS pathogenesis
In the pathogenesis of allergic rhinitis, substance P (show TAC1 Antibodies) induces expression of RANTES in nasal mucosa.
CCL3 (show CCL3 Antibodies), CCL4 (show CCL4 Antibodies) and CCL5 gene expression was evaluated in response to simian-human immunodeficiency virus (SHIV) infection in a rhesus macaque model.
This gene is one of several CC cytokine genes clustered on the q-arm of chromosome 17. Cytokines are a family of secreted proteins involved in immunoregulatory and inflammatory processes. The CC cytokines are proteins characterized by two adjacent cysteines. The cytokine encoded by this gene functions as a chemoattractant for blood monocytes, memory T helper cells and eosinophils. It causes the release of histamine from basophils and activates eosinophils. This cytokine is one of the major HIV-suppressive factors produced by CD8+ cells. It functions as one of the natural ligands for the chemokine receptor CCR5 and it suppresses in vitro replication of the R5 strains of HIV-1, which use CCR5 as a coreceptor.
chemokine (C-C motif) ligand 5
, small inducible cytokine A5
, C-C motif chemokine 5
, T-cell-specific protein RANTES
, regulated upon activation normal T-cell expressed and secreted
, small-inducible cytokine A5
, T-cell specific protein p288
, beta-chemokine RANTES
, eosinophil chemotactic cytokine
, regulated upon activation, normally T-expressed, and presumably secreted
, small inducible cytokine subfamily A (Cys-Cys), member 5
, chemokine ah294
, small inducible cytokine A5 RANTES
, chemokine CCL5/RANTES
, chemokine ligand 5