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The protein encoded by IL23R is a subunit of the receptor for IL23A/IL23. Additionally we are shipping Interleukin 23 Receptor Antibodies (124) and Interleukin 23 Receptor Proteins (13) and many more products for this protein.
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Four SNPs in IFNGR2, IL12RB1 (show IL12RB1 ELISA Kits), IL12RB2 (show IL12RB2 ELISA Kits), and IL23R were found to be associated with the MAP infection status of the resource population.
The interleukin-23 receptor gene polymorphism may not contribute to the susceptibility of development of primary immune thrombocytopenia in Egyptian children.
this study shows that there is no significant difference in mucosal IL-23R expression in Ulcerative Colitis patients with moderate-to-severe disease activity compared to those in remission
In this study, we were unable to establish a correlation between the IL-23R SNPs investigated and HLA-B27-associated acute anterior uveitis as well as idiopathic intermediate uveitis.
These results suggest that IL23R may contribute to the development of intracerebral hemorrhage.
The association of IL-23R and Ankylosing Spondylitis (AS) that is seen in Caucasian patients with AS is not present in Chinese patients with AS.
Differential splicing generates antagonistic soluble IL-23R (sIL (show PMEL ELISA Kits)-23R) variants, which might limit IL-23 (show IL23A ELISA Kits)-mediated immune responses. Here, ectodomain shedding of IL-23R was identified as an alternative pathway for the generation of sIL (show PMEL ELISA Kits)-23R.
The IL23R polymorphisms rs10889677, rs7517847, and the IL12B (show IL12B ELISA Kits) polymorphism rs3212227 are not associated with multiple sclerosis risk.
meta-analysis supports that two polymorphisms (rs11209026 and rs7517847) in the IL-23 (show IL23A ELISA Kits) gene may be considered to be protective factors against developing UC among Caucasian populations.
The results of the current study disclosers1343151 variant of IL23R as a susceptibility gene in CD.
Lower LCN2 (show LCN2 ELISA Kits) levels in Crohn's Disease patients carrying IBD risk-increasing IL23R variants may result from a restricted upregulation of LCN2 (show LCN2 ELISA Kits) due to an impaired Th17 immune response
Differential splicing generates antagonistic soluble IL-23R (sIL (show STIL ELISA Kits)-23R) variants, which might limit IL-23 (show IL23A ELISA Kits)-mediated immune responses. Here, ectodomain shedding of IL-23R was identified as an alternative pathway for the generation of sIL (show STIL ELISA Kits)-23R.
Estrogen and progesterone decrease let-7f microRNA expression and increase IL-23/IL-23 (show IL23A ELISA Kits) receptor signaling and IL-17A (show IL17A ELISA Kits) production in patients with severe asthma.
Data show the contribution of IL-23/IL-23 (show IL23A ELISA Kits) receptor and IL-7/IL-7 (show IL7 ELISA Kits) receptor signaling in Th17 and Th1 (show HAND1 ELISA Kits) cell dynamics during experimental autoimmune encephalomyelitis (EAE).
Study provides evidence that IL-23 (show IL23A ELISA Kits)/IL-23R plays a critical role in brain ischemic injury
IL-23 (show IL23A ELISA Kits) plus T-cell receptor signalling results in significant upregulation of IL-23 receptor expressed predominantly on CD4 (show CD4 ELISA Kits)(hi)CD8 (show CD8A ELISA Kits)(hi)CD3 (show CD3E ELISA Kits)(+)alphabetaTCR(+) thymocytes, and leads to RORgammat-dependent apoptosis.
Homeostatic IL-23 receptor signaling limits Th17 response through IL-22 (show IL22 ELISA Kits)-mediated containment of commensal microbiota.
Characterization of potent IL-23R small-peptide modulator, 2305 (teeeqqly), that decreases inflammatory response.
Although they share a common subunit, IL-23 (show IL23A ELISA Kits) and IL-12 (show IL12A ELISA Kits) receptors are not expressed on the same cell populations
IL-23R+ innate lymphoid cells induce colitis via interleukin-22 (show IL22 ELISA Kits)-dependent mechanism.
the balance between the enhancing and inhibitory effects of gamma-delta T cells is regulated by their level of IL-23R expression.
The protein encoded by this gene is a subunit of the receptor for IL23A/IL23. This protein pairs with the receptor molecule IL12RB1/IL12Rbeta1, and both are required for IL23A signaling. This protein associates constitutively with Janus kinase 2 (JAK2), and also binds to transcription activator STAT3 in a ligand-dependent manner.
, interleukin 23 receptor
, interleukin-23 receptor-like
, IL-23 receptor
, interleukin 23 receptor-like