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Binds to one of the cis-acting domain of the insulin gene enhancer. Additionally we are shipping and many more products for this protein.
Isl2a is expressed in the lateral posterior mesoderm prior to angioblast migration. Isl2a-deficient angioblasts show disorganized migration to the midline to form axial vessels. Its expression is negatively regulated by Notch (show NOTCH1 Antibodies) activity. It positively regulates flt4 (show FLT4 Antibodies), a VEGF-C (show VEGFC Antibodies) receptor repressed by Notch (show NOTCH1 Antibodies) during angiogenesis. Isl2a may be an intermediate between Notch (show NOTCH1 Antibodies) signaling and genetic programs controlling angioblasts.
Prdm14 (show PRDM14 Antibodies) regulates caudal (show CAD Antibodies) rimary axon outgrowth through activation of islet2 expression.
primary motoneuron subtypes are likely to be specified by factors that act in parallel to or upstream of islet1 (show ISL1 Antibodies) and islet2
Binds to one of the cis-acting domain of the insulin gene enhancer. May be involved in subtype specialization of primary motoneurons.
insulin gene enhancer protein isl-2
, insulin gene enhancer protein isl-2a