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PROS1 encodes a vitamin K-dependent plasma protein that functions as a cofactor for the anticoagulant protease, activated protein C (APC) to inhibit blood coagulation. Additionally we are shipping PROS1 Kits (15) and PROS1 Proteins (9) and many more products for this protein.
Showing 10 out of 73 products:
Cow (Bovine) Polyclonal PROS1 Primary Antibody for IHC, WB - ABIN2774131
Ten Kate, Platteel, Mulder, Terpstra, Nicolaes, Reitsma, van der Steege, van der Meer: PROS1 analysis in 87 pedigrees with hereditary protein S deficiency demonstrates striking genotype-phenotype associations. in Human mutation 2008
The immunoabsorption of PON1 (show PON1 Antibodies) from plasma significantly reduced protein S anti-coagulant activity.
A PROS1 c.1486_1490delGATTA mutation on exon 12. appeared to be the primary cause of thrombosis in the family of the present study.
analysis of the amino acid residues in the laminin G domains of protein S involved in tissue factor pathway inhibitor (show TFPI Antibodies) interaction
The present study highlights that the GAS6 (show GAS6 Antibodies)/ProS-TAM (show CCNA1 Antibodies) system correlates in several ways with disease activity in systemic lupus erythematosus
genetic polymorphism affects endogenous thrombin (show F2 Antibodies) potential among FV Leiden carriers
Identical large duplication mutation of PROS1 was detected in 3 unrelated patients with thrombophilia from hereditary protein S deficiency.
Anti-human protein S antibody induces tissue factor (show F3 Antibodies) expression through a direct interaction with PFKP (show PFKP Antibodies) and ERK1/2 (show MAPK1/3 Antibodies) activation in coronary artery endothelial cells.
Data indicate that the protein S sex hormone-binding globulin (SHBG (show SHBG Antibodies))-like domain was important for binding and enhancement of tissue factor pathway inhibitor (TFPI (show TFPI Antibodies)).
miR (show MLXIP Antibodies)-494 is involved in the mechanism of estrogen-mediated downregulation of PS expression
This is the first report of a large deletion of PROS1 from exon 1 through 12 in Polish patients with deep-vein thrombosis.
Activated protein C (show PROC Antibodies)(APC (show APC Antibodies)) combined with protein S(PS) had significant antithrombotic effect. APC (show APC Antibodies) combined with PS prolonged clotting time. Dependence on APC (show APC Antibodies)-cofactor activity of PS for expression of anticoagulant activity by APC (show APC Antibodies).
By revealing that neural stem-like cells act within the SVZ neurogenic niche as phagocytes and that the ProS/MerTK (show MERTK Antibodies) path represents an endogenous regulatory mechanism for SVZ cell phagocytic activity
Optimal TAM (show CCNA1 Antibodies) signaling requires coincident TAM (show CCNA1 Antibodies) ligand engagement of both its receptor and the phospholipid phosphatidylserine regulating TAM (show CCNA1 Antibodies) receptor tyrosine kinases Tyro3 (show TYRO3 Antibodies), Axl (show AXL Antibodies), and Mer (show ERH Antibodies) and their ligands Gas6 (show GAS6 Antibodies) and Protein S.
Data indicate that activated T cells express Pros1.
Results demonstrate that Protein S is a Mer (show ERH Antibodies) ligand, and is active in Mer (show ERH Antibodies)-driven phagocytosis in the retina.
A self-regulatory mechanism of Toll (show TLR4 Antibodies)-like receptor signalling through the suppression of Gas6 (show GAS6 Antibodies) and ProS expression is described.
Protein S controls hypoxic/ischemic blood-brain barrier disruption through the TAM (show CCNA1 Antibodies) receptor Tyro3 (show TYRO3 Antibodies) and sphingosine 1-phosphate receptor1.
results demonstrate that ProS is a pleiotropic anticoagulant with activated Protein C (show PROC Antibodies)-independent activities and highlight new roles for ProS in vascular development and homeostasis
Pregnancy causes a decrease in APC (show APC Antibodies) resistance in mice, which can be explained by the elevation of protein S levels and increased TFPI (show TFPI Antibodies) activity in plasma.
This gene encodes a vitamin K-dependent plasma protein that functions as a cofactor for the anticoagulant protease, activated protein C (APC) to inhibit blood coagulation. It is found in plasma in both a free, functionally active form and also in an inactive form complexed with C4b-binding protein. Mutations in this gene result in autosomal dominant hereditary thrombophilia. An inactive pseudogene of this locus is located at an adjacent region on chromosome 3.
vitamin K-dependent protein S
, protein Sa
, vitamin K-dependent plasma protein S
, vitamin K-dependent protein S preproprotein