Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species and application
anti-Rat (Rattus) IL17C Antibodies:
anti-Human IL17C Antibodies:
anti-Mouse (Murine) IL17C Antibodies:
Go to our pre-filtered search.
Human Polyclonal IL17C Primary Antibody for IF (p), IHC (p) - ABIN747983
Huang, Yuan, Zhu, Yin, Hong, Dong, Jin, Dong: IL-17C/IL-17RE Augments T Cell Function in Autoimmune Hepatitis. in Journal of immunology (Baltimore, Md. : 1950) 2016
Human Monoclonal IL17C Primary Antibody for CyTOF, ELISA (Capture) - ABIN4899383
Chen, Park, Tang: Activity-dependent synaptic Wnt release regulates hippocampal long term potentiation. in The Journal of biological chemistry 2006
Helicobacter pylori infection was associated with a significant increase in IL-17C expression in gastric mucosa. The role of IL-17C in the pathogenesis of H. pylori-induced diseases remains to be determined.
Data indicate that epithelial IL-17C promotes neutrophilic inflammation in the tumor microenvironment and suggest that IL-17C links a pathologic microbiota, as present in COPD (show ARCN1 Antibodies) patients, with enhanced tumor growth.
IL-17C is an essential epithelial cell-derived cytokine.
Our study provides evidence that Staphylococcus aureus activates NOD2 (show NOD2 Antibodies) in keratinocytes, resulting in an increased expression of IL-17C, a mechanism that may be dysregulated in atopic dermatitis.
Interleukin-17C is present in the tissue around aseptic loosened implants.
IL-17C is involved in the innate immune response of respiratory epithelial cells and is suppressed by cigarette smoke.
Data suggest that activation of TLR5 (show TLR5 Antibodies) upregulates IL17C expression in cells from intestinal mucosa and colonic adenocarcinoma; IL17C expression is upregulated in intestinal mucosa cells from patients with ulcerative colitis compared with normal cells.
Keratinocyte overexpression of IL-17C promotes psoriasiform skin inflammation.
In our pilot study, we discovered significant changes in methylation patterns of genes IL-7 (show IL7 Antibodies), IL-13 (show IL13 Antibodies), IL-17C and TYK2 (show TYK2 Antibodies) between henodialysis patients and healthy subjects
IL-17C is an essential autocrine cytokine that regulates innate epithelial immune responses.
IL-17C+IL-6 (show IL6 Antibodies) knockout mice reveal that IL17C may have a role in psoriasis and skin inflammation, explaining why IL6 (show IL6 Antibodies) blockade treatment may not be effective
IL-17C is a critical factor that potentiates inflammatory responses and causes host injury during fungal infection.
IL-17A (show IL17A Antibodies)-mediated expression of epithelial IL-17C amplifies the release of chemokines by epithelial cells and thereby contributes to the recruitment of neutrophils and systemic inflammation during acute P. aeruginosa pneumonia.
Smooth muscle cell-derived IL-17C plays a proatherogenic role by supporting the recruitment of Il17a (show IL17A Antibodies)-positive Th17 cells to atherosclerotic lesions.
IL-17C and IL-17RE (show IL17RE Antibodies) are dispensable for immunity to candidiasis
IL-17C plays a critical role in maintaining mucosal barrier integrity; IL-17C deficiency renders mice more susceptible to mucosal barrier breakage and development of colitis.
IL-17C-interleukin-17 receptor E (show IL17RE Antibodies) induced the expression of a nuclear IkappaB family member, IkappaBzeta (show NFKBIZ Antibodies), in Th17 cells to potentiate the Th17 cell response
Intranasal administration of adenovirus expressing IL-17 (show IL17A Antibodies) results in bronchoalveolar lavage neutrophilia and Th1 (show HAND1 Antibodies)-type inflammatory gene expression in the lung.
The protein encoded by this gene is a T cell-derived cytokine that shares the sequence similarity with IL17. This cytokine was reported to stimulate the release of tumor necrosis factor alpha and interleukin 1 beta from a monocytic cell line. The expression of this cytokine was found to be restricted to activated T cells.
, Interleukin 17-3
, cytokine CX2