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The analysis points to a critical role for Hoxa9 (show HOXA9 ELISA Kits) and PU.1 in distal regulation of c-myb expression in murine myeloid cells during iL-6 (show IL6 ELISA Kits)-induced cell differentiation.
Data suggest that the upregulations of Myb and Peg3 (show PEG3 ELISA Kits) are likely the key anti-cancer events of EGCG in vivo.
deficiency alters the expression of a crucial subset of TAL1 (show TAL1 ELISA Kits)- and NOTCH1 (show NOTCH1 ELISA Kits)-regulated genes, including the MYB and MYC (show MYC ELISA Kits) oncogenes, respectively.
MYB acts on MAPK (show MAPK1 ELISA Kits) signaling by directly regulating transcription of the gene encoding the negative modulator SPRY2 (show SPRY2 ELISA Kits).
This work provides important mechanistic insight into how spatiotemporal expression of the Rag genes is tightly controlled during B lymphocyte (show AKAP17A ELISA Kits) development to prevent mistimed dsDNA breaks and their deleterious consequences.
These results Myb as a critical component of the gene regulatory network controlling effector Treg cell differentiation and function.
Myb expands the ISC pool within which CRC (show SCRIB ELISA Kits) is initiated while co-operating with TSG (show TWSG1 ELISA Kits) loss
c-Myb regulates proliferation/differentiation of adventitial Sca1 (show ATXN1 ELISA Kits)+ vascular smooth muscle progenitor cells by transcriptional activation of myocardin (show MYOCD ELISA Kits).
the key role of the transcription factor c-Myb in regulating the T-bet-mediated anti-viral program, is reported.
p38 (show CRK ELISA Kits) and NOX1 (show NOX1 ELISA Kits) are essential for the protective effect of c-Myb and that NOX1 (show NOX1 ELISA Kits) acts upstream of p38 (show CRK ELISA Kits) activation.
Expression of the MYB-NFIB (show NFIB ELISA Kits) fusion oncogene (show RAB1A ELISA Kits) in mammary tissue resulted in hyperplastic glands that developed into adenocarcinoma.
A trend toward superior PFS was noted with the MYB/NFIB (show NFIB ELISA Kits) rearrangement, although this was not statistically significant. NGS revealed three tumors with 4q12 amplification, producing increased copies of axitinib-targeted genes PDGFR (show PDGFRB ELISA Kits)/KDR (show KDR ELISA Kits)/KIT.
Rearrangement of MYB did not affect OS.
Exosomes isolated from cultured AML (show RUNX1 ELISA Kits) or the plasma from mice bearing AML (show RUNX1 ELISA Kits) xenografts exhibited enrichment of miR (show MLXIP ELISA Kits)-150 and miR (show MLXIP ELISA Kits)-155. HSPCs cocultured with either of these exosomes exhibited impaired clonogenicity, through the miR (show MLXIP ELISA Kits)-150- and miR (show MLXIP ELISA Kits)-155-mediated suppression of the translation of transcripts encoding c-MYB
identification of SNPs within the IQCJ, NXPH1 (show NXPH1 ELISA Kits), PHF17 (show PHF17 ELISA Kits) and MYB genes partly explaining the large interindividual variability observed in plasma triglyceride levels in response to an n-3 fatty acid supplementation
The data indicate that MAZ is essential to bypass MYB promoter repression by RB family members and to induce MYB expression.
A mutant of c-Myb, D152V, specifically affects c-Myb's ability to regulate genes involved in differentiation, causing failure in c-Myb's ability to block differentiation.
l genetic alterations were limited to two fusion genes, EWSR1 (show EWSR1 ELISA Kits)-PATZ1 and SLMAP (show SLMAP ELISA Kits)-NTRK2 (show NTRK2 ELISA Kits), both in gangliogliomas. Alterations in BRAF (show BRAF ELISA Kits), FGFR1 (show FGFR1 ELISA Kits), or MYB account for most pathogenic alterations in low-grade neuroepithelial tumors
This gene encodes a transcription factor that is a member of the MYB family of transcription factor genes. The protein contains three domains, an N-terminal DNA-binding domain, a central transcriptional activation domain and a C-terminal domain involved in transcriptional repression. This protein plays an essential role in the regulation of hematopoiesis and may play a role in tumorigenesis. Alternative splicing results in multiple transcript variants.
, Avian myeloblastosis viral (v-myb) oncogene homolog
, v-myb myeloblastosis viral oncogene homolog (avian)
, transcriptional activator Myb-like
, gag-myb protein
, myb proto-oncogene protein
, myeloblastosis proto-oncogene product
, proto-oncogene c-Myb
, transcriptional activator Myb
, tumor-specific myb protein
, c-myb protein (140 AA)
, DNA-binding transcriptional regulator
, c-myb proto-oncogene