Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species and application
anti-Human KIF3A Antibodies:
anti-Mouse (Murine) KIF3A Antibodies:
anti-Rat (Rattus) KIF3A Antibodies:
Go to our pre-filtered search.
Human Monoclonal KIF3A Primary Antibody for IF, WB - ABIN968567
Hirokawa: Kinesin and dynein superfamily proteins and the mechanism of organelle transport. in Science (New York, N.Y.) 1998
Show all 3 references for ABIN968567
Cow (Bovine) Polyclonal KIF3A Primary Antibody for WB - ABIN2778192
Whitehead, Wang, Bost-Usinger, Hoang, Frazer, Burnside: Photoreceptor localization of the KIF3A and KIF3B subunits of the heterotrimeric microtubule motor kinesin II in vertebrate retina. in Experimental eye research 1999
Inhibition of kif3a in the human dental follicle cells (hDFCs) and human dental pulp cells (hDPCs) is disrupted primary cilia formation and/or function, and it was shown that kif3a is important in the differentiation of hDFCs and hDPCs through the Wnt (show WNT2 Antibodies) pathway.
High KIF3A expression is associated with glioblastoma.
These data suggest that GLI (show GLI1 Antibodies) interactions with KIF3A-KIF3B (show KIF3B Antibodies)-KAP3 (show KIFAP3 Antibodies) complexes are essential for proper GLI (show GLI1 Antibodies) transcriptional activity.
KIF3A and OVOL1 (show OVOL1 Antibodies) are involved in the development of Atopic dermatitis in the Chinese pediatric population.
both motor domains of KIF3A/B coordinate for processive motility and move at different speeds
Ablation of this phosphorylation abolished herpes simplex virus 1 US3-mediated downregulation of CD1d (show CD1D Antibodies) expression, suggesting that phosphorylation of KIF3A is the primary mechanism of viral suppression of CD1d (show CD1D Antibodies) expression.
Findings support the notion that upregulation of KIF3a is causal of aberrant activation of Wnt (show WNT2 Antibodies) signaling in advanced prostate cancer through the KIF3a-DVL2 (show DVL2 Antibodies)-beta-catenin (show CTNNB1 Antibodies) axis.
POPX2 (show PPM1F Antibodies) affects trafficking by determining the phosphorylation status of KIF3A at serine 690.
Association of KIF3A, but not OVOL1 and ACTL9, with atopic eczema in Italian patients
in addition to its ciliogenic roles, Kif3a recruits p150(Glued (show DCTN1 Antibodies)) to the subdistal appendages of mother centrioles, critical for centrosomes to function as microtubule-organizing centres.
Longitudinal microcomputed tomography (muCT) imaging and histopathological analyses revealed an increased rate of cyst formation, increased proportion of cysts with proliferating cells, higher frequency of atypical cysts as well as the development of neoplasms in Vhl (show VHL Antibodies)/Kif3a/Trp53 (show TP53 Antibodies) mutant kidneys compared to Kif3a/Trp53 (show TP53 Antibodies) or Vhl (show VHL Antibodies)/Kif3a mutant kidneys.
Data suggest biocatalysis by Kif3a homodimer, Kif3c (show KIF3C Antibodies) homodimer, or Kif3a/Kif3c (show KIF3C Antibodies) heterodimer vary; microtubule interaction is very fast for Kif3a homodimer; Kif3c (show KIF3C Antibodies) homodimer interaction is much slower; Kif3a/Kif3c (show KIF3C Antibodies) heterodimer interaction is intermediate.
KIF1-binding protein (show KIAA1279 Antibodies) interacts with KIF3A in haploid male germ cells, promoting spermatid elongation.
Vhl (show VHL Antibodies) and Kif3a deletion accelerates renal cyst formation
The results of this study suggested thatactivity-dependent cargo loading, in which phosphorylation of the KIF3A C terminus upregulates the loading and transport of N-cadherin (show CDH2 Antibodies) in homeostatic synaptic plasticity.
The results demonstrate that visual pigments transport to the retinal outer segment despite removal of KIF3 and IFT88 (show IFT88 Antibodies), and KIF3-mediated anterograde IFT is responsible for photoreceptor transition zone and axoneme formation.
Processivity of the kinesin-2 (show KIF2A Antibodies) KIF3A results from rear head gating and not front head gating.
A ciliopathy with hydrocephalus, isolated craniosynostosis, hypertelorism, and clefting caused by deletion of Kif3a
KIF3AB exhibits novel ATPase (show DNAH8 Antibodies) activity.
single-molecule motility from different kinesin-1 and kinesin-2 (show KIF2A Antibodies) neck-linker chimeras stepping along microtubules, was studied.
mouse homolog is a molecular motor protein involved in the development of primary cilia
kinesin family protein 3A
, kinesin-like protein KIF3A
, microtubule plus end-directed kinesin motor 3A
, DNA sequence AF180004
, DNA sequence AF180009
, N-4 kinesin
, kinesin-II subunit