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Rat (Rattus) Endothelin 1 Primary Antibody for ELISA - ABIN366462
Huang, Tang, Liang, Wen, Zhang, Xuan, Jian, Lin, Huang: The effects of 17-methoxyl-7-hydroxy-benzene-furanchalcone on the pressure overload-induced progression of cardiac hypertrophy to cardiac failure. in PLoS ONE 2014
Show all 3 references for ABIN366462
Mouse (Murine) Endothelin 1 Primary Antibody for ELISA - ABIN1672858
Agapitov, Haynes: Role of endothelin in cardiovascular disease. in Journal of the renin-angiotensin-aldosterone system : JRAAS 2002
Show all 3 references for ABIN1672858
Human Endothelin 1 Primary Antibody for ELISA - ABIN837829
Lacedonia, Valerio, Palladino, Carpagnano, Correale, Biase, Barbaro: Role of vasoactive intestinal Peptide in chronic obstructive pulmonary disease with pulmonary hypertension. in Rejuvenation research 2014
Show all 2 references for ABIN837829
Pig (Porcine) Endothelin 1 Primary Antibody for ELISA - ABIN3155739
Gallinat, Efferz, Paul, Minor: One or 4 h of "in-house" reconditioning by machine perfusion after cold storage improve reperfusion parameters in porcine kidneys. in Transplant international : official journal of the European Society for Organ Transplantation 2014
Phylogenetic analysis of conserved grhl (show GHRL ELISA Kits)-binding sites in gene regulatory regions identified endothelin-1 (edn1) as a putative direct grhl3 (show GRHL3 ELISA Kits) target gene, and this was confirmed by chromatin precipitation assays in embryos.
EDN1 plays an important role in hepatocellular carcinoma progression by activating the PI3K/AKT pathway and is regulated by miR-1.
Data suggest that edn1/ednraa (show EDNRA ELISA Kits) (endothelin-1/endothelin-1 receptor type A (show EDNRA ELISA Kits)) signaling is involved in acid-base regulation and transepithelial proton secretion via vacuolar proton-translocating ATPase (show DNAH8 ELISA Kits) in zebrafish embryonic skin.
These findings point to complexity of regulation by edn1 and hand2 at the earliest stages of pharyngeal arch development, in which control of growth and morphogenesis can be genetically separated.
Endothelin 1 combines with Bone Morphogenetic Proteins to pattern the dorsal-ventral axis of the craniofacial skeleton.
activation of Endothelin-1 signaling in craniofacial patterning
Edn1 from the pharyngeal ectoderm signals through Ednra (show EDNRA ELISA Kits) proteins to direct early dorsoventral patterning of the skeletogenic neural crest.
The role of endothelin-1 and light in the regulation of melanopsins and the clock proteins in an embyronic cell line is reported.
This is the first report describing the cDNA encoding preproendothelin-1 in an amphibian species.
A new role for et-1 signaling during early neural crest specification is reported.
The paternal rs5370 polymorphism decreases the risk for preeclampsia and is not associated with placental expression of the EDN1 gene.
high percentages of HIF-1alpha (show HIF1A ELISA Kits) (100%), VEGF (show VEGFA ELISA Kits) (89.5%), iNOS (show NOS2 ELISA Kits) (78.9%), and ET-1 (84.2%) expressions were observed in congenital heart disease autopsy cases and this was found to be significant.
The level of ET-1 and eNOS (show NOS3 ELISA Kits) plasma was not significantly different between the CMS (show Cd2ap ELISA Kits) patients and healthy controls
Coronary occlusion patients in the poor coronary collateral circulation ( CCC ) group had a higher ET1 level than those in the good CCC group (0.59 +/- 0.48 vs. 0.39 +/- 0.32 pmol/L, p < 0.001), and the ET1 values increased with the descent of the Rentrop grades (p for trend < 0.001).
Activin A (show INHBA ELISA Kits) and preeclamptic serum upregulate ET-1, ICAM-1 (show ICAM1 ELISA Kits), and VCAM-1 (show VCAM1 ELISA Kits) in human umbilical vein endothelial cells, consistent with activin A (show INHBA ELISA Kits) contributing to the pathophysiology of preeclampsia.
ET1 gene and ECE-1 expression levels in human umbilical vein endothelial cells and ET-1 expression levels in the cell culture supernatants increased significantly in some experimental groups compared with those in the control group.
Serum and synovial fluid endothelin-1 concentrations are correlated with the development and progression of knee osteoarthritis.
This study demonstrated the pathogenesis mechanism during the development of dementia after ischemic stroke by investigating the relationship between miR (show MLXIP ELISA Kits)-125a and its target ET-1.
Urinary TGF-beta1 (show TGFB1 ELISA Kits) and ET-1 levels were associated with AGT (show AGXT ELISA Kits) level, which likely reflects an early interplay between tissue remodeling and RAAS in obesity-related kidney injury.
High circulating endothelin-1 levels are associated with the development of impaired glucose tolerance and type 2 diabetes in women.
sub-vasomotor concentration of ET-1 leads to vascular dysfunction by impairing endothelium-dependent NO-mediated dilation via p38 (show MAPK14 ELISA Kits) kinase-mediated production of superoxide from NADPH oxidase (show NOX1 ELISA Kits) following ETA receptor activation
ET1 was lowest in kidneys removed from live pigs, greater in kidneys from pigs with brain death, and greatest in kidneys from pigs with cardiac arrest.
ET-1 contributes to formation of oedema during experimental sepsis by a novel mechanism involving increased HBP (show HEBP1 ELISA Kits) release from neutrophils.
Endothelin-1 expression is upregulated following therapeutic hypothermia after cardiac arrest.
ET-1 produces contraction of the urinary bladder neck muscles via muscular ET(A (show EDNRA ELISA Kits)) receptors coupled to extracellular Ca(2 (show CA2 ELISA Kits)+) entry via VOC (L-type) and non-VOC channels.
Endothelin-1 elevation is not only a conserved phenomenon in a pig traumatic brain injury (TBI) model, but it is a likely target for understanding the observed enhanced vascular response to TBI.
interleukin-6 (show IL6 ELISA Kits), endothelin ET-1, and apoptotic Bak (show BAK1 ELISA Kits) and Bcl-XL (show BCL2L1 ELISA Kits) genes have roles in small bowel transplantation, in a swine model of ischemia and reperfusion injury
ET-1 elicits a different pattern of Src (show SRC ELISA Kits) family kinase (SFK) activation and might trigger a different pattern of SFK activation from that caused by ATP and UTP.
Compared with the untreated group, the levels of serum ET-1 after acute myocardial infarction and reperfusion were significantly decreased in the Xuefu Zhuyu-treated group.
study suggests a possible role for endothelin-1(resulting from the actions of endothelin-converting enzyme-1 (show ECE1 ELISA Kits)) acting via endothelin receptor A (show EDNRA ELISA Kits) in the control of luteolytic sensitivity in the pig
Data suggest that, in Fallopian tubes, endothelins (EDN1, EDN2 (show EDN2 ELISA Kits), EDN3 (show EDN3 ELISA Kits)) and EDN (show RNASE2 ELISA Kits)-converting enzymes (ECE1 (show ECE1 ELISA Kits), ECE2 (show ECE2 ELISA Kits)) are expressed in epithelial cells; EDN (show RNASE2 ELISA Kits) receptors (EDNRA (show EDNRA ELISA Kits), EDNRB (show EDNRB ELISA Kits)) are present in smooth-muscle. Expression of EDN1, EDN2 (show EDN2 ELISA Kits), and ECE2 (show ECE2 ELISA Kits) is highest on day of ovulation. EDN (show RNASE2 ELISA Kits)/ENDR signaling appears to participate Fallopian tube function. These studies were conducted in Holstein cows.
results suggest that ET-1-induced activation of proMMP-2 is mediated via cross-talk between NADPH oxidase (show NOX1 ELISA Kits)-PKCalpha (show PKCa ELISA Kits)-p(38)MAPK (show MAPK1 ELISA Kits) and NFkappaB-MT1MMP (show MMP14 ELISA Kits) signaling pathways along with a marked decrease in TIMP-2 (show TIMP2 ELISA Kits) expression in the cells
We demonstrated that (i) treatment of bovine pulmonary artery smooth muscle cells with ET-1 stimulates cPLA2 (show PLA2G4A ELISA Kits) activity in the cell membrane.
Differential cell-specific and spatiotemporal expression of the EDN1 system and NOS (show NOS ELISA Kits) in the bovine utero-placental unit may be associated with regulation of vascular and cellular functions during pregnancy.
Oxidized LDL is a stimulus of ET-1 production in cultured vascular endothelial cells.
Endothelin-1 decreases ethanolamine plasmalogen levels and evokes platelet-activating factor production in brain microvessels
prostaglandin F2alpha, endothelin-1, and angiotensin II may interact with each other in a local positive feedback manner to activate their secretion in the regressing corpus luteum, thus accelerating and completing luteolysis
LOX-1 (show OLR1 ELISA Kits) and CD40 (show CD40 ELISA Kits) synergistically, but through a distinct pathway, work to induce endothelin-1 expression in endothelial cells.
Elevated local expression of ET-1 and Ednra/Ednrb (show EDNRB ELISA Kits) during the peri (show PLIN1 ELISA Kits)-ovulatory period induces the high contractile activity of the oviduct to optimize gamete transport.
Suggest that the activation of a local positive feedback mechanism in the corpus luteum among ET-1, Ang II (show AGT ELISA Kits) and PGF2alpha might play a functional role in the paracrine modulation of luteolytic cascade.
Review and Meta-Analysis of ET-1 Transgenic Mice provides robust evidence that global ET-1 overexpression in mice lowers blood pressure in an age-dependent manner. Older ET-1+/+ mice have a somewhat more pronounced reduction of blood pressure.
evidence supports a model in which aldosterone activation of the mineralocorticoid receptor (show NR3C2 ELISA Kits) (MR) results in the MR-hormone complex binding at HRE at -671bp to open chromatin structure around other regulatory elements in the Edn1 gene
Decreased ET-1 levels were associated with greater activation of NLRP3 (show NLRP3 ELISA Kits) and IL-1beta (show IL1B ELISA Kits) in normal glucose. High glucose increased NLRP3 (show NLRP3 ELISA Kits) markers and activation compared to normal and low glucose
AMPAR-mediated glutamatergic neurotransmission may underlie the mechanism of ET1-ET(A (show EDNRA ELISA Kits))R signaling pathway in the regulation of anxiety.
indicate that endothelin-1 is critical in the development of cerebrovascular and cognitive impairments with experimental cerebral malaria
increased extrarenal vascular ET-1 production in response to HS intake is mediated by increased extracellular osmolarity and plays a critical role in regulating skin storage of Na(+)
PPARgamma (show PPARG ELISA Kits) regulates miR (show MLXIP ELISA Kits)-98 to modulate ET-1 expression and pulmonary endothelial cell proliferation in pulmonary hypertension.
these results indicated that I/R induced upregulation of ET1 and ETA in the kidneys, which was, at least in part, dependent on the production of inflammatory cytokines.
we have demonstrated that the expression of ET-1 and IL-25 (show IL25 ELISA Kits) was coordinately upregulated in the lesional keratinocytes of patients with AD and a murine AD model.
Short-term hyperinsulinaemia leads to increased vascular resistance in the equine digit and increased expression of ET-1 in the laminar tissue.
Plasma ET-1 levels of rabbits increased significantly in fluorinated groups compared with those in the control group.
Dynamic monitoring and comparison of plasma levels of ET, CGRP, NO, and MDA as well as SOD activity revealed that appropriate intervention of these factors may reduce reperfusion injury
In a saline lavage-induced lung injury model, both circulatory and pulmonary ET-1 levels increased.
Data suggest elevated levels of endothelin-1, as exhibited in cardiovascular diseases, facilitate development of ventricular arrhythmia by steepening action potential duration restitution and by increasing beat-to-beat variability of repolarization.
High levels of ET-1 are closely associated with BBB (show ALMS1 ELISA Kits) disruption. ET-1 may play an important role in the pathogenesis of secondary brain injury after ICH (show ACE ELISA Kits).
After subarachnoid hemorrhage, the contractile response to ET-1 was enhanced, and the ET(A (show EDNRA ELISA Kits)) receptor expression was upregulated in the basilar artery.
After acute pulmonary thromboembolism, thrombolytic and anti-inflammatory treatment could decrease acute lung injury induced by ET-1 and NF-kappaB (show NFKB1 ELISA Kits) activation. [endothelin-1; NFKB]
The increase in myocardial distensibility induced by endothelin-1 is absent in heart failure and is dependent on nitric oxide and prostaglandin release.
Endothelin-1 enhances nuclear Ca2 (show CA2 ELISA Kits)+ transients in atrial myocytes through Ins (show INS ELISA Kits)(1,4,5)P3-dependent Ca2 (show CA2 ELISA Kits)+ release from perinuclear Ca2 (show CA2 ELISA Kits)+ stores
The enhancement of gap junction intercellular communication is activated by endothelin-1 via modulating the expression of connexin43 (show GJA1 ELISA Kits), and plays an important role in the pathogenesis of cerebral vasospasm
The protein encoded by this gene is proteolytically processed to release a secreted peptide termed endothelin 1. This peptide is a potent vasoconstrictor and is produced by vascular endothelial cells. Endothelin 1 also can affect the central nervous system. Two transcript variants encoding different isoforms have been found for this gene.
, endothelin 1
, gene for endothelin
, preproendothelin 1