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Human Monoclonal NEUROD Primary Antibody for ELISA, WB - ABIN1724757
Shimoda, Chen, Noguchi, Matsumoto, Grayburn: Neurogenic differentiation 1 directs differentiation of cytokeratin 19-positive human pancreatic nonendocrine cells into insulin-producing cells. in Transplantation proceedings 2010
Show all 2 references for ABIN1724757
Human Monoclonal NEUROD Primary Antibody for ELISA, WB - ABIN1724758
Huang, Kishida, Cao, Murakami-Tonami, Mu, Nakaguro, Koide, Takeuchi, Onishi, Kadomatsu: The neuronal differentiation factor NeuroD1 downregulates the neuronal repellent factor Slit2 expression and promotes cell motility and tumor formation of neuroblastoma. in Cancer research 2011
Show all 2 references for ABIN1724758
Human Polyclonal NEUROD Primary Antibody for ELISA, WB - ABIN1532158
Tamimi, Steingrimsson, Copeland, Dyer-Montgomery, Lee, Hernandez, Jenkins, Tapscott: The NEUROD gene maps to human chromosome 2q32 and mouse chromosome 2. in Genomics 1996
NeuroD1 (show NEUROD1 Antibodies) seemed not sufficient to induce and maintain neuronal differentiation. Induction of neuronal differentiation by overexpression of Neurog1 (show NEUROG1 Antibodies) initiated important steps for the development of glutamatergic neurons such as the spiral ganglion neurons
Study reports a family with autosomal dominant diabetes related to a new NEUROD1 mutation, one of very few meeting Maturity Onset Diabetes of the Young criteria.
RNAi of lentiviral vector target NeuroD can reduce the migration and invasion abilities of PANC-1 cells
this study concludes that the novel mechanism would regulate the expression of ALK (show ALK Antibodies) in neuroblastoma (show ARHGEF16 Antibodies) and that NeuroD1 (show NEUROD1 Antibodies) should be significantly involved in neuroblastoma (show ARHGEF16 Antibodies) tumorigenesis.
NEUROD1 is important for maintenance of the retina function and partial loss-of-function mutation in NEUROD1 is likely a rare cause of nonsyndromic ARRP.
Increased expression of NeuroD1 subsequently leads to regulation of expression and function of the nicotinic acetylcholine receptor subunit cluster of alpha3, alpha5, and beta4.
Transactivation of Ctbp (show CTBP2 Antibodies) was dependent on the histone H3 (show HIST3H3 Antibodies) lysine 9 (H3K9) demethylase (show MBD2 Antibodies) activity of LSD1 (show KDM1A Antibodies) facilitates subsequent H3K9 acetylation by the NeuroD1 (show NEUROD1 Antibodies)-associated histone acetyltransferase (show HAT Antibodies), P300/CBP-associated factor (show KAT2B Antibodies).
Gene expression profiling revealed that permissive lines are typified by lower expression of the early neurogenic transcription factor ASCL1 and, conversely, by higher expression of the late neurogenic transcription factor NEUROD1.
NeuroD1 (show NEUROD1 Antibodies) regulates survival and migration of neuroendocrine lung carcinomas via signaling molecules TrkB (show NTRK2 Antibodies) and NCAM (show NCAM1 Antibodies).
The overexpression of NeuroD (show NEUROD1 Antibodies) may contribute to the tumorogenesis and development of pancreatic carcinoma, and is closely correlated to the cancer cell proliferation, p53 (show TP53 Antibodies) signal pathway and neural invasion.
This gene encodes a member of the NeuroD family of basic helix-loop-helix (bHLH) transcription factors. The protein forms heterodimers with other bHLH proteins and activates transcription of genes that contain a specific DNA sequence known as the E-box. It regulates expression of the insulin gene, and mutations in this gene result in type II diabetes mellitus.
basic helix-loop-helix transcription factor
, beta-cell E-box transactivator 2
, class A basic helix-loop-helix protein 3
, neurogenic differentiation factor 1
, neurogenic helix-loop-helix protein NEUROD