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anti-Human Oncostatin M Receptor Antibodies:
anti-Mouse (Murine) Oncostatin M Receptor Antibodies:
anti-Rat (Rattus) Oncostatin M Receptor Antibodies:
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Dog (Canine) Polyclonal Oncostatin M Receptor Primary Antibody for WB - ABIN2782612
Arita, South, Hans-Filho, Sakuma, Lai-Cheong, Clements, Odashiro, Odashiro, Hans-Neto, Hans, Holder, Bhogal, Hartshorne, Akiyama, Shimizu, McGrath: Oncostatin M receptor-beta mutations underlie familial primary localized cutaneous amyloidosis. in American journal of human genetics 2008
the RET (show RET Antibodies) p.S891A mutation combined with OSMR p.G513D may underlie a novel phenotype manifesting as familial medullary thyroid carcinoma and cutaneous amyloidosis
this study offers new findings on the molecular genetics and disease relevance of mutations in OSMR in Familial primary localized cutaneous amyloidosis.
Oncostatin M (show OSM Antibodies) and interleukin-31 (show IL31 Antibodies): Cytokines, receptors, signal transduction and physiology.
primary localized cutaneous amyloidosis has a missense mutation in oncostatin M receptor beta
The interleukin IL-31 (show IL31 Antibodies)/IL-31receptor axis contributes to tumor growth in human follicular lymphoma.
oncostatin M (show OSM Antibodies) is a cytokine possessing vigorous antiviral and immunostimulatory properties which is released by APC (show APC Antibodies) upon interaction with CD40L (show CD40LG Antibodies) present on activated CD4 (show CD4 Antibodies)+ T cells.
The disease severity of rheumatoid arthritis and systemic lupus erythematosus can be partially affected by the OSMR promoter polymorphisms.
We conclude that an OSMR/TGM2 (show TGM2 Antibodies)/integrin-alpha5beta1/fibronectin (show FN1 Antibodies) pathway is of biological significance in cervical squamous cell carcinoma
A unique loop structure in oncostatin M (show OSM Antibodies) determines binding affinity toward oncostatin M receptor and leukemia inhibitory factor receptor (show LIFR Antibodies).
enhanced production by beta-defensin-2 (show DEFB4 Antibodies) in T cells
In astrocytes but not microglia, phosphorylation of STAT1 (show STAT1 Antibodies) and STAT3 (show STAT3 Antibodies) occurred in response to OSM (show OSM Antibodies), whereas both microglia and astrocytes responded to hyper-IL-6 (IL-6 (show IL6 Antibodies) linked to the soluble IL-6 (show IL6 Antibodies) receptor).
OSM (show OSM Antibodies) signaling via OSMR in synovial fibroblasts has the potential to contribute significantly to joint destruction in inflammatory arthritis.
defects in OSM (show OSM Antibodies) signaling promote the deterioration of high-fat diet-induced obesity and related metabolic disorders
Data indicate that OSM (show OSM Antibodies) receptor beta subunit (show POLG Antibodies)-deficient (OSMRbeta(-/-)) mice exhibited phenotypic changes in adipose tissue macrophages (ATMs) to M1, increased proinflammatory cytokines in the adipose tissue, and systemic insulin (show INS Antibodies) resistance.
These data indicate that the transient RANKL (show TNFSF11 Antibodies) induction by intermittent PTH (show PTH Antibodies) administration, which is associated with its anabolic action, is changed to a prolonged induction in OSMR-deficient osteoblasts, resulting in bone destruction.
Bone formation can be stimulated independently of bone resorption and provide new insights into OSMR signaling.
Expression of oncostatin M receptor beta in a specific subset of nociceptive sensory neurons.
Mice deficient in OSM (show OSM Antibodies) receptor showed impaired liver regeneration with persistent parenchymal necrosis after carbon tetrachloride exposure. Recovery of liver mass from partial hepatectomy was also significantly delayed in OSMR(-/-) mice.
the up-regulation of p-STAT3 (show STAT3 Antibodies) and p-CREB (show CREB1 Antibodies) may be induced possibly through OSMR in inflammation
This gene encodes a member of the type I cytokine receptor family. The encoded protein heterodimerizes with interleukin 6 signal transducer to form the type II oncostatin M receptor and with interleukin 31 receptor A to form the interleukin 31 receptor, and thus transduces oncostatin M and interleukin 31 induced signaling events. Mutations in this gene have been associated with familial primary localized cutaneous amyloidosis. Alternatively spliced transcript variants encoding different isoforms have been found for this gene.
IL-31 receptor subunit beta
, IL-31R subunit beta
, interleukin-31 receptor subunit beta
, oncostatin-M specific receptor beta subunit
, oncostatin-M-specific receptor subunit beta
, oncostatin receptor
, oncostatin-M specific receptor subunit beta
, oncostatin M specific receptor