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anti-Human YAP1 Antibodies:
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Human Monoclonal YAP1 Primary Antibody for IF, IHC (p) - ABIN564526
Lau, Murray, Houshmandi, Xu, Gutmann, Yu: Merlin is a potent inhibitor of glioma growth. in Cancer research 2008
Show all 17 Pubmed References
Human Polyclonal YAP1 Primary Antibody for ChIP, ICC - ABIN258559
Kapoor, Yao, Ying, Hua, Liewen, Wang, Zhong, Wu, Sadanandam, Hu, Chang, Chu, Al-Khalil, Jiang, Xia, Fletcher-Sananikone, Lim, Horwitz, Viale, Pettazzoni, Sanchez, Wang, Protopopov, Zhang, Heffernan et al.: Yap1 activation enables bypass of oncogenic Kras addiction in pancreatic cancer. ... in Cell 2014
Show all 12 Pubmed References
Human Monoclonal YAP1 Primary Antibody for FACS, IHC - ABIN969574
Fernandez-L, Northcott, Dalton, Fraga, Ellison, Angers, Taylor, Kenney: YAP1 is amplified and up-regulated in hedgehog-associated medulloblastomas and mediates Sonic hedgehog-driven neural precursor proliferation. in Genes & development 2009
Show all 2 Pubmed References
Human Polyclonal YAP1 Primary Antibody for ICC, IF - ABIN4366489
Li, Lim, Chen, McCabe, Kim, Zhang, Mao: Spinal expression of Hippo signaling components YAP and TAZ following peripheral nerve injury in rats. in Brain research 2013
Dog (Canine) Polyclonal YAP1 Primary Antibody for ELISA, WB - ABIN4219868
Zender, Spector, Xue, Flemming, Cordon-Cardo, Silke, Fan, Luk, Wigler, Hannon, Mu, Lucito, Powers, Lowe: Identification and validation of oncogenes in liver cancer using an integrative oncogenomic approach. in Cell 2006
Human Polyclonal YAP1 Primary Antibody for IF (p), IHC (p) - ABIN701485
Li, Shang, Shu, Zhang, Ji, Sun, Li, Xie: gga-miR-375 plays a key role in tumorigenesis post subgroup J avian leukosis virus infection. in PLoS ONE 2014
SRSF3 (show SRSF3 Antibodies)-regulated miR (show MLXIP Antibodies)-132/212 controls cell migration and invasion by targeting YAP1
findings indicate that HIF-2alpha (show EPAS1 Antibodies) increases cancer cell growth by up-regulating YAP1 activity
Studies indicate that the transcriptional co-activators YAP and TAZ (show TAZ Antibodies) recently emerged as key mediators of the biological effects that are observed in response to extracellular matrix (ECM (show MMRN1 Antibodies)) elasticity and cell shape.
Results uncover a novel PKCiota-AMOT (show AMOT Antibodies)-YAP1 signaling axis that promotes OSC (show LSS Antibodies) tumor growth.
YAP inhibition significantly enhances the antitumor efficacy of a pan-RAF inhibitor, LY3009120, in KRAS-mutant pancreatic cancer.
High YAP expression is associated with pancreatic ductal adenocarcinoma.
High YAP1 expression is associated with triple-negative breast cancer.
This identifies the YAP1/TEAD1 (show TEAD1 Antibodies) complex as the representative dysregulated profile of Hippo signaling in OS and provides proof-of-principle that targeting TEAD1 (show TEAD1 Antibodies) may be a therapeutic strategy of osteosarcoma.
a stiffness-dependent YAP/TAZ (show TAZ Antibodies)-mediated positive feedback loop that drives remodeling phenotypes in pulmonary artery smooth muscle cells via reduced COX-2 (show COX2 Antibodies) and prostaglandin activity.
Study found that LPS (show IRF6 Antibodies)-induced activation and nuclear accumulation of YAP accelerated human pulmonary micro-vascular endothelial cell apoptosis.
found that epidermal YAP activity drives GLI2 (show GLI2 Antibodies) nuclear accumulation in the skin of YAP2-5SA-DeltaC mice, which depends on epidermal beta-catenin (show CTNNB1 Antibodies) activation
Yap and Taz (show TAZ Antibodies) leads to impaired epicardial epithelial-to-mesenchymal transition (EMT (show ITK Antibodies)) and a reduction in epicardial cell proliferation and differentiation into coronary endothelial cells.
This study identifies YAP/TAZ as central mediators of VEGF signaling
These results suggest that YAP promotes muscle differentiation by activating the Abl (show ABL1 Antibodies)/Src (show SRC Antibodies)/MEKK3 (show MAP3K3 Antibodies)/MEK5 (show MAP2K5 Antibodies)/ERK5 (show MAPK7 Antibodies) kinase cascade.
data demonstrate that Yap1 is a required factor for proper differentiation of mouse embryonic stem cells, while remaining dispensable for self-renewal
these results identify a novel function of YAP in neocortical astrocytic differentiation and proliferation, and reveal a BMP2 (show BMP2 Antibodies)-YAP-SMAD1 (show SMAD1 Antibodies) pathway underlying astrocytic differentiation in the developing mouse neocortex.
Wnt/beta-catenin signaling via Axin2 is required for myogenesis and, together with YAP/Taz and Tead1, active in IIa/IIx muscle fibers
Epicardial YAP/TAZ (show TAZ Antibodies) orchestrate an immunosuppressive response following myocardial infarction
YAP negatively regulated an antiviral immune response. YAP deficiency resulted in enhanced innate immunity, a diminished viral load, and morbidity in vivo. YAP blocked dimerization of the transcription factor IRF3 (show IRF3 Antibodies) and impeded translocation of IRF3 (show IRF3 Antibodies) to the nucleus after viral infection.
During vascular regression, Yap/Taz is activated by blood circulation in the endothelial cells. This leads to induction of Ctgf and actin polymerization. Interference with Yap/Taz activation decreased Ctgf production, which decreased actin polymerization and vascular regression.
that Yap1 entered the nucleus and promoted transcription in response to blood flow.
that Yap1 integrates the anabolic demands of tissue growth during development and tumorigenesis by reprogramming nitrogen metabolism to stimulate nucleotide biosynthesis
Amotl2a (show AMOTL2 Antibodies) function in the control of lateral line primordium cell proliferation is mediated together by the Hippo pathway effector Yap1 and the Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies) effector Lef1 (show LEF1 Antibodies).
data reveals that Yap is required for pronephric duct integrity, maintenance of baso-lateral cell polarity, and ciliogenesis during zebrafish kidney development
Yap/Taz (show TAZ Antibodies)-Tead activity is necessary and sufficient for optic vesicle progenitors to adopt retinal pigment epithelium identity in zebrafish.
Yap is essential for fin regeneration and that its function is dependent on mechanical tension, conferred by a balancing act of cell density and cytoskeleton activity.
When transcriptional coactivators Yap and Taz (show TAZ Antibodies) were restricted from interacting with Tead transcription factors through expression of a dominant negative transgene, cardiac precursors failed to migrate completely to the midline.
Yap coordinately regulates cell proliferation and apoptosis and is required for dorsoventral axis formation, gastrulation, cardiogenesis, hematopoiesis, and somitogenesis.
zebrafish yap is required for the development of the brain, eyes, and neural crest during embryogenesis.
Yap1 has a crucial role in controlling the limb regenerative capacity in Xenopus
The encoded gene product presumably interacts with YY1 protein\; however, its exact function is not known. Alternative splicing results in multiple transcript variants encoding different isoforms.
Yes-associated protein 1, 65kDa
, Yes-associated protein 1, 65 kD
, 65 kDa Yes-associated protein
, yes-associated protein 2
, yorkie homolog
, yes-associated protein, 65 kDa
, YY1-associated protein 1
, hepatocellular carcinoma susceptibility protein
, hepatocellular carcinoma-associated protein 2