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anti-Human ATF3 Antibodies:
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Human Polyclonal ATF3 Primary Antibody for IF (p), IHC (p) - ABIN669561
Hu, Li, Li: MARVELD1 Inhibits Nonsense-Mediated RNA Decay by Repressing Serine Phosphorylation of UPF1. in PLoS ONE 2013
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Human Polyclonal ATF3 Primary Antibody for ICC, IF - ABIN4282009
Wu, Nguyen, Dziunycz, Chang, Brooks, Lefort, Hofbauer, Dotto: Opposing roles for calcineurin and ATF3 in squamous skin cancer. in Nature 2010
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Human Monoclonal ATF3 Primary Antibody for RNAi, ELISA - ABIN559974
Chen, Huang, Chu, Chen, Chou, Wang, Kulp, Teng, Wang, Chen: Energy restriction-mimetic agents induce apoptosis in prostate cancer cells in part through epigenetic activation of KLF6 tumor suppressor gene expression. in The Journal of biological chemistry 2011
Show all 3 Pubmed References
Human Polyclonal ATF3 Primary Antibody for IHC (p), IHC - ABIN315461
Maciag, Nandurdikar, Hong, Chakrapani, Diwan, Morris, Shami, Shiao, Anderson, Keefer, Saavedra: Activation of the c-Jun N-terminal kinase/activating transcription factor 3 (ATF3) pathway characterizes effective arylated diazeniumdiolate-based nitric oxide-releasing anticancer prodrugs. in Journal of medicinal chemistry 2011
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Human Monoclonal ATF3 Primary Antibody for ELISA, WB - ABIN559976
Hsueh, Kuo, Chen: Transcriptional regulators of the ?Np63: their role in limbal epithelial cell proliferation. in Journal of cellular physiology 2012
Human Polyclonal ATF3 Primary Antibody for FACS, WB - ABIN1536715
Mo, Dai, Kang, Cui, He et al.: Ectopic expression of human MutS homologue 2 on renal carcinoma cells is induced by oxidative stress with interleukin-18 promotion via p38 mitogen-activated protein kinase (MAPK) and c-Jun N-terminal ... in The Journal of biological chemistry 2012
Data show that Atf3 was detected in retinal ganglion cell axons in both the nerve fiber layer and the optic nerve on the injured side.
Data show that ATF3 may be an important mediator of optic nerve regeneration-promoting gene expression in fish, a role which merits further investigation.
The analysis showed higher urinary NGAL (show LCN2 Antibodies) and urinary ATF3 in patients with sepsis-AKI in comparison with patients with sepsis-non-AKI and healthy volunteers.
ATF3 mRNA and protein expression was significantly reduced in preterm Preeclamptic placentas. Therefore, reduced ATF3 may be centrally involved in the pathology of Preeclampsia.
Data suggest that suppression of nonsense-mediated RNA decay due to persistent DNA damage (from exposure to either mutagens, gamma rays, or oxidative stress) requires the activity of p38alpha (show MAPK14 Antibodies) MAPK (show MAPK1 Antibodies) (MAPK14 (show MAPK14 Antibodies), mitogen-activated protein kinase 14 (show MAPK14 Antibodies), MAP kinase p38 alpha (show MAPK14 Antibodies)); mRNA of ATF3 (activating transcription factor 3) is stabilized by persistent DNA damage in a p38alpha (show MAPK14 Antibodies) MAPK (show MAPK1 Antibodies)-dependent manner.
The microvascular injury phenotypes observed in vitro and in vivo were similar. ATF3 plays an important role in mediating brain microvascular responses to acute and chronic lipotoxic injury and may be an important preventative and therapeutic target for endothelial dysfunction in vascular cognitive impairment .
we have identified ATF3 as an important regulator of cisplatin cytotoxicity and that ATF3 inducers in combination with platins are a potential novel therapeutic approach for NSCLC.
reactivation of ATF3 is an important factor in determining sensitivity to pracinostat treatment, both in vitro and in vivo, and could serve as a potential biomarker of response and provide a rationale for therapeutic utility in HDACi-mediated treatments for bladder cancer.
these findings support roles for both cFOS (indirect) and ATF3 (direct) in effecting MMP13 (show MMP13 Antibodies) transcription in human chondrocytes.
We functionally validated several elements for metformin-induced promoter and enhancer activity. These include an enhancer in an ataxia telangiectasia mutated (ATM (show ATM Antibodies)) intron that has SNPs in linkage disequilibrium .Using ChIP-seq and siRNA knockdown, we further show that activating transcription factor 3 (ATF3), our top metformin upregulated AMPK (show PRKAA1 Antibodies)-dependent gene, could have an important role in gluconeogenesis repression.
ATF3 inhibition significantly increased the expression of proinflammatory cytokines interleukin 6 and interleukin 8 in cigarette smoke extract stimulated human bronchial epithelial cells.
ATF3 overexpression increased PFF-A-mediated cleaved PARP (show COL11A2 Antibodies).
This study shows, for the first time, that alpha-lactalbumin (show LALBA Antibodies) isolated in a rare 28kDa dimeric form induces cell death, while 14kDa (show SRP14 Antibodies) monomeric alpha-lactalbumin (show LALBA Antibodies) is inactive.
Results show a pro-regenerative ATF3 function during PNS nerve regeneration involving transcriptional activation of a neuropeptide-encoding regeneration-associated gene cluster.
Results reveal a critical role for ATF3 as a key regulator of the acinar cell transcriptional response during injury and may provide a link between chronic pancreatitis and PDAC.
The lung injury score and mortality were higher in ATF3 knock-out mice treated with Pseudomonas aeruginosa. Moreover, ATF3 was demonstrated to bind to lipopolysaccharide binding protein (show LBP Antibodies). These findings suggest ATF3 protects mice against acute lung injury induced by Pseudomonas aeruginosa partly due to the binding to lipopolysaccharide binding protein (show LBP Antibodies).
ATF3-KO mice escape from PE-dependent maladaptive cardiac remodeling by suppressing the IFNgamma-CXCL10 (show CXCL10 Antibodies)-CXCR3 (show CXCR3 Antibodies) axis at multiple levels.
ATF3 has a protective role in dampening the high fat-induced cardiac remodeling processes.
ATF3 protects against LPS (show TLR4 Antibodies)-induced acute lung injury by inhibiting TL1A (show TNFSF15 Antibodies) expression.
Myotube contraction increased ATF3 level, which modified chemokine (show CCL1 Antibodies) expression. In skeletal muscle after downhill running, ATF3 also modified chemokine (show CCL1 Antibodies) expression.
converges with CSL (show SMPX Antibodies) in negative control of cancer-associated fibroblast activation
Loss of activating transcription factor 3 (ATF3) in knockout mice promotes the emergence of keratins CK5 (show KRT5 Antibodies)+CK8 (show KRT8 Antibodies)+ epithelial cells.
ATF3 appears to affect gonadotropin-stimulated progesterone secretion at a step or steps downstream of PKA signaling and before cholesterol conversion to progesterone.
ATF3 induction by acute hypoxia is mediated by nitric oxide and the JNK (show MAPK8 Antibodies) pathway in endothelial cells
Data indicate increasing expression for CREB (show CREB1 Antibodies), ATF1 (show AFT1 Antibodies), and ATF3 during gastrulation.
This gene encodes a member of the mammalian activation transcription factor/cAMP responsive element-binding (CREB) protein family of transcription factors. This gene is induced by a variety of signals, including many of those encountered by cancer cells, and is involved in the complex process of cellular stress response. Multiple transcript variants encoding different isoforms have been found for this gene. It is possible that alternative splicing of this gene may be physiologically important in the regulation of target genes.
cyclic AMP-dependent transcription factor ATF-3
, activating transcription factor 3
, cAMP-dependent transcription factor ATF-3
, transcription factor LRG-21
, liver regeneration factor 1