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Human AMH Protein expressed in HEK-293 Cells - ABIN2714759
Signorile, Petraglia, Baldi: Anti-mullerian hormone is expressed by endometriosis tissues and induces cell cycle arrest and apoptosis in endometriosis cells. in Journal of experimental & clinical cancer research : CR 2014
nuclear receptor subfamily 5, group A, member 1b is a new candidate for sex determination and differentiation in a way similar to steroidogenic factor 1 (show NR5A1 Proteins), possibly involving AMH
zebrafish Anti-Mullerian hormone (Amh) is regulated by sox9a, sox9b, and cyp19a1a during gonad development
amh is a candidate gene down-regulating cyp19a1a, leading to "juvenile ovary-to-testis" transformation.
It was shown that the male-to-female sex reversal phenotype in hotei medaka mutants is not a direct consequence of anti-Mullerian hormone signaling in supporting cells, but is instead mediated by germ cells.
These findings underscore the importance of screening young females with AMH for possible occult primary ovarian insufficiency.
Women with unexplained infertility did not show evidence of decreased ovarian reserve as measured by AMH and antral follicle count.
An undetectable AMH level in women aged <40 at the end of chemotherapy for early stage breast cancer gave a good prediction that ovarian function would not return.
In granulosa cells from women with PCOS, the regulation of AMH and AMHR2 (show AMHR2 Proteins) expression is altered in a way that promotes the overexpression of the AMH/AMHR2 (show AMHR2 Proteins) system, and could contribute to the follicular arrest observed in these patients.
genetic variants of AMH or AMHR2 (show AMHR2 Proteins) were not found to be associated with a higher risk for polycystic ovaries syndrome.
Strong positive age-independent relationship between AMH level and the rate of euploid blastocysts I in vitro fertilization/intracytoplasmic sperm injection was found.
The ratio of inactive proAMH precursor to receptor-competent AMHN,C differs in women with polycystic ovary syndrome relative to unaffected controls indicating that AMH signaling mechanisms may be altered in women with PCOS.
Study found 24 rare AMH variants in patients with PCOS and control subjects; 18 variants were specific to women with PCOS. Seventeen of 18 (94%) PCOS-specific variants had significantly reduced AMH signaling, whereas none of 6 variants observed in control subjects showed significant defects in signaling.
In the obese polycystic ovary syndrome group, anti-mullerian hormone was associated with ghrelin (show GHRL Proteins) levels independent of age, insulin (show INS Proteins), and total testosterone. There was no association between total ghrelin (show GHRL Proteins) and anti-mullerian hormone levels in non-obese women with polycystic ovary syndrome, non-obese controls, or obese controls.
Data suggest that young men and young women initially have similar levels of circulating levels of AMH, but women lose their AMH in parallel with the decrease in their ovarian reserve. Circulating AMH in men is negatively associated with all-cause mortality; men with higher levels of AMH may outlive men with lower levels of AMH. [EDITORIAL]
AMH increases GnRH-dependent LH pulsatility and secretion, supporting a central action of AMH on GnRH neurons.
AMH and FOXL2 (show FOXL2 Proteins) collaboratively work to reserve ovarian follicles. AMH is an endogenous target gene of FOXL2 (show FOXL2 Proteins).
Up-regulation of SOX9 (show SOX9 Proteins) in sertoli cells from testiculopathic patients accounts for increasing anti-mullerian hormone expression via impaired androgen receptor (show AR Proteins) signaling.
Male mice require AMH to undergo normal social development.
Data show that Purkinje cells express receptors for Mullerian inhibiting substance (MIS), and that MIS(-/-) male mice have female-like numbers of Purkinje cells and a female-like size to other parts of their cerebellum.
MIS may be involved in anterograde rather than autocrine or retrograde regulation of neurons.
FSH (show BRD2 Proteins) and cAMP stimulate AMH transcription by granulosa cells. FSH (show BRD2 Proteins) and LH have an additive effect, which may be important in polycystic ovary syndrome.
This suggests that MIS is one of the determinants of "boy"-specific behavior.
Role of anti-Mullerian hormone (AMH) as a regulator and marker of ovarian function.
Administration of MIS to male mice induced IEX-1S mRNA in the prostate in vivo, suggesting that MIS may function as an endogenous hormonal regulator of NF-kappaB (show NFKB1 Proteins) signaling and growth in the prostate gland.
This study showed that antral follicle counts (AFC) and AMH levels were repeatable when determined at an unknown stage of follicular growth and expected day of follicular wave emergence, but repeatability was greater for AMH than AFC.
The effects of castration and other surgical intervention on the blood levels of anti-Muellerian hormone, inhibin A (show INHA Proteins), gonadotropins, and gonadotropin receptors in bull calves are reported.
Results from these studies indicate that AMH signaling plays a role in both regulating granulosa cell proliferation and preventing granulosa cells from 5- to 8-mm follicles from undergoing premature differentiation before follicle selection.
These findings indicate the followings: AMH mRNA levels decrease in both dominant and secondary follicles during follicular deviation; granulosa cells from heathy follicles express more AMH mRNA compared to subordinate follicles undergoing atresia and FSH (show BRD2 Proteins) stimulates AMH and AMHR2 (show AMHR2 Proteins) mRNA expression in granulosa cells of co-dominant follicles.
Measurement of AMH concentration in the plasma of cows can help to predict their capacity for embryo production in response to gonadotrophin treatment.
In first study to investigate the blood profile and immunohuistochemistry of anti-Mullerian hormone in bovine granulosa-theca cell tumors, the findings indicated that anti-Mullerian hormone is a novel biomarker for granulosa-theca cell tumors in cattle.
Regulation of anti-Mullerian hormone production in the cow.
Intrafollicular AMH was not a marker of cystic development in the cow, but low AMH concentrations in cysts were associated with luteinization.
AMH expression is modulated by androgens in bovine granulosa cells from small follicles.
Anti-Mullerian hormone is a member of the transforming growth factor-beta gene family which mediates male sexual differentiation. Anti-Mullerian hormone causes the regression of Mullerian ducts which would otherwise differentiate into the uterus and fallopian tubes. Some mutations in the anti-Mullerian hormone result in persistent Mullerian duct syndrome.
, anti-Mullerian hormone
, anti-mullerian hormone
, Mullerian inhibiting factor
, Mullerian inhibiting substance
, anti-Muellerian hormone
, muellerian-inhibiting substance
, Mullerian inhibitory substance
, Anti - Mullerian hormone (Mulerian inhibiting substance)