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anti-Human GSTM1 Antibodies:
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Human Polyclonal GSTM1 Primary Antibody for ELISA, WB - ABIN268723
Lee, Kim, Woo, Hong, Cho: Increased frequencies of glutathione S-transferase (GSTM1 and GSTT1) gene deletions in Korean patients with acquired aplastic anemia. in Blood 2001
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Cow (Bovine) Polyclonal GSTM1 Primary Antibody for WB - ABIN2776984
Sobti, Kaur, Kaur, Singh, Gupta, Jain, Nakahara: Interaction of passive smoking with GST (GSTM1, GSTT1, and GSTP1) genotypes in the risk of cervical cancer in India. in Cancer genetics and cytogenetics 2006
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Human Polyclonal GSTM1 Primary Antibody for FACS, IHC (p) - ABIN390883
Kostrykina, Pechkovskii, Mishukova, Khripko, Zarubina, Selezneva, Sinkina, Terekhova, Lazarev, Petrova, Filipenko: Studying the association of polymorphic variants of GSTM1 and GSTT1 genes with breast cancer in female residents of Altai Krai. in Bulletin of experimental biology and medicine 2009
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Human Monoclonal GSTM1 Primary Antibody for ELISA, FACS - ABIN4314717
Başak, Başak, Doğuç, Aylak, Oğuztüzün, Bozer, Gültekin: Does maternal exposure to artificial food coloring additives increase oxidative stress in the skin of rats? in Human & experimental toxicology 2016
Human Monoclonal GSTM1 Primary Antibody for FACS, IHC - ABIN969180
Navarro, Peterson, Chen, Makar, Schwarz, King, Li, Li, Kestin, Lampe: Cruciferous vegetable feeding alters UGT1A1 activity: diet- and genotype-dependent changes in serum bilirubin in a controlled feeding trial. in Cancer prevention research (Philadelphia, Pa.) 2009
Human Polyclonal GSTM1 Primary Antibody for ELISA, WB - ABIN185334
Habalova, Salagovic, Kalina, Stubna: Combined analysis of polymorphisms in glutathione S-transferase M1 and microsomal epoxide hydrolase in lung cancer patients. in Neoplasma 2005
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results suggest that combined deletion polymorphisms of GSTT1 (show GSTT1 Antibodies) and GSTM1 can have implications in the prediction of the clinical course of the disease
On comparing the prevalence of GSTM1 null polymorphism among the group with subjects with tobacco habits and no oral lesions, oral leukoplakia, and oral squamous cell carcinoma, it was observed that there was a statistically significant association between GSTM1 null polymorphism and the different groups (P < 0.01).
The present case-control study found that GSTM1 and GSTT1 (show GSTT1 Antibodies) polymorphism are not associated with JOAG (show MYOC Antibodies) risk in North Indian population. The present meta-analysis suggested that there might be a significant association of GSTM1 null genotype with glaucoma (JOAG (show MYOC Antibodies) & POAG) risk.
Pesticide-exposed individuals with inherited susceptible metabolic genotypes (particularly, null genotype for GSTM1 and the PON1 (show PON1 Antibodies) 192R allele) appear to have an increased risk of genotoxic DNA damage.
182 Hungarian bladder cancer cases and 78 cancer-free controls were investigated. It was not possible to establish a particular impact of NAT2 (show SLC38A1 Antibodies)*6A and *7B genotypes (15 cases, 8%, 5 controls, 7%). GSTT1 (show GSTT1 Antibodies) exerted no marked influence on bladder cancer (negative 21% cases vs. 22% controls). The portion of GSTM1 negative bladder cancer patients was increased (63% cases vs. 54% controls).
double-null genotype of the GSTM1 gene is not associated with ALL development or its progression.
We assessed the effect of allelic deletions in the GSTM1 and GSTT1 (show GSTT1 Antibodies) genotypes on lung cancer overall survival through a systematic review of the scientific literature after applying predefined inclusion and exclusion criteria
GSTM1 genetic polymorphisms are not associated with the development of graft-versus-host disease after allogeneic hematopoietic stem cell transplantation.
Autism Spectrum Disorder status may be a potential effect modifier when assessing the association between GSTP1 (show GSTP1 Antibodies) rs1695 but not (GSTM1 or GSTT1 (show GSTT1 Antibodies)) and blood aluminum concentrations, among Jamaican children.
Combined GSTM1, GSTT1 (show GSTT1 Antibodies), GSTA1 (show GSTA1 Antibodies) and GSTP1 (show GSTP1 Antibodies) polymorphisms might be associated to the risk of clear cell renal cell carcinoma (show MOK Antibodies).
Six proteins were regulated at both basal and inducible levels exhibiting the largest dynamic range of Nrf2 (show NFE2L2 Antibodies) regulation: cytochrome CYP2A5, GSTM3 (show glutathione S-transferase mu 3 (brain) Antibodies), GSTM1, ENTPD5 (show ENTPD5 Antibodies),UDPGDH (show UGDH Antibodies), and EPHX1 (show EPHX1 Antibodies).
mitochondrial Gstb1 is induced under oxidative stress
the gene (GSTM1) encoding the detoxification enzyme glutathione S-transferase M1 is transcriptionally upregulated by Myb (show MYB Antibodies)
The Gstm1 gene was represented by two EST (show MAP3K8 Antibodies) clones with similar levels of downregulation.
We overexpressed Hoxa9 (show HOXA9 Antibodies) and Meis1 (show MEIS1 Antibodies) in primary hematopoietic cells. Arrays identified c-Myb (show MYB Antibodies) and a c-Myb (show MYB Antibodies) target (Gstm1) among the genes with the strongest response to Hoxa9 (show HOXA9 Antibodies)/Meis1 (show MEIS1 Antibodies).
Genetic variants that cause a decremental change in expression of Gstm1 may permit an environment of exaggerated oxidative stress, leading to susceptibility to vascular remodeling and atherosclerosis
The role of polymorphisms of glutathione S-transferases GSTM1, M3, P1, T1 and A1 in susceptibility to alcoholic liver disease. In addition, oxidant stress is proposed to be an important pathogenic factor in liver damage related to alcohol.
Cytosolic and membrane-bound forms of glutathione S-transferase are encoded by two distinct supergene families. At present, eight distinct classes of the soluble cytoplasmic mammalian glutathione S-transferases have been identified: alpha, kappa, mu, omega, pi, sigma, theta and zeta. This gene encodes a glutathione S-transferase that belongs to the mu class. The mu class of enzymes functions in the detoxification of electrophilic compounds, including carcinogens, therapeutic drugs, environmental toxins and products of oxidative stress, by conjugation with glutathione. The genes encoding the mu class of enzymes are organized in a gene cluster on chromosome 1p13.3 and are known to be highly polymorphic. These genetic variations can change an individual's susceptibility to carcinogens and toxins as well as affect the toxicity and efficacy of certain drugs.
GST class-mu 1
, glutathione S-transferase Mu 1
, glutathione S-transferase mu 1
, glutathione S-transferase M1
, GST HB subunit 4
, HB subunit 4
, S-(hydroxyalkyl)glutathione lyase
, glutathione S-alkyltransferase
, glutathione S-aralkyltransferase
, glutathione S-aryltransferase
, glutathione S-transferase M4
, glutathione S-transferase mu 4
, GST 3-3
, GST Yb1
, glutathione S-transferase Yb-1 subunit
, glutathione-S-transferase mu type 1 (Yb1)
, glutathione-S-transferase, mu type 1 (Yb1)
, GST class-mu 2
, GST, muscle
, S-(hydroxyalkyl)glutathione lyase M2
, glutathione S-alkyltransferase M2
, glutathione S-aralkyltransferase M2
, glutathione S-aryltransferase M2
, glutathione S-transferase 4
, glutathione S-transferase M2 (muscle)
, glutathione S-transferase Mu 2
, GST 1-1
, glutathione S-transferase GT8.7
, glutathione-S-transferase, mu 1