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anti-Human MEF2C Antibodies:
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Human Polyclonal MEF2C Primary Antibody for EIA, IHC (p) - ABIN358801
Janson, Chen, Li, Leifer: Functional regulatory regions of human transcription factor MEF2C. in Brain research. Molecular brain research 2001
Show all 5 references for ABIN358801
Human Polyclonal MEF2C Primary Antibody for EIA, IHC (p) - ABIN358804
Konig, Hinard, Arnaudeau, Holzer, Potter, Bader, Bernheim: Membrane hyperpolarization triggers myogenin and myocyte enhancer factor-2 expression during human myoblast differentiation. in The Journal of biological chemistry 2004
Show all 4 references for ABIN358804
Human Polyclonal MEF2C Primary Antibody for EIA - ABIN358292
Maeda, Gupta, Stewart: TEF-1 and MEF2 transcription factors interact to regulate muscle-specific promoters. in Biochemical and biophysical research communications 2002
Show all 3 references for ABIN358292
Human Polyclonal MEF2C Primary Antibody for IF, IHC (p) - ABIN390244
Maeda, Chapman, Stewart: Mammalian vestigial-like 2, a cofactor of TEF-1 and MEF2 transcription factors that promotes skeletal muscle differentiation. in The Journal of biological chemistry 2002
Show all 3 references for ABIN390244
Chicken Polyclonal MEF2C Primary Antibody for WB - ABIN2774663
Nagel, Meyer, Quentmeier, Kaufmann, Drexler, MacLeod: MEF2C is activated by multiple mechanisms in a subset of T-acute lymphoblastic leukemia cell lines. in Leukemia 2008
Our study provides new insights in MEF2C conservation and provides the first evidence of mef2cb regulation by both transcriptional and post transcriptional mechanisms.
By selectively inhibiting translational initiation of mef2ca and other mRNAs, eIF4EBP3L reprograms the translational profile of muscle, enabling it to adjust to new environmental conditions.
find no evidence that the phenotypic stability in the wild type is provided by redundancy between mef2ca and its co-ortholog mef2cb, or that it is related to the selector (homeotic) gene function of mef2ca
Mef2ca single mutants have delayed heart development, but form an apparently normal heart. Mef2cb single mutants have a functional heart and are viable adults.
Data show that mef2cb is expressed in the late ventricular region, and is necessary for late myocardial addition to the arterial pole.
the genetic interaction of Tbx5 (show TBX5 Antibodies) and Mef2c is not only required for MYH6 (show MYH6 Antibodies) expression but also essential for the early stages of heart development and survival
Mef2c and Mef2d (show MEF2D Antibodies) are required for proper cardiac gene expression.
The finding of a jugular pit (show IRF6 Antibodies) in this patient facilitated the diagnosis, and he is, to our knowledge, the third case of jugular pit (show IRF6 Antibodies) in association with 5q14.3 deletion incorporating the MEF2C locus.
MiR (show MLXIP Antibodies)-135b-5p and MiR (show MLXIP Antibodies)-499a-3p Promote Cell Proliferation and Migration in Atherosclerosis by Directly Targeting MEF2C
early B cell factor-1 (EBF1 (show EBF1 Antibodies)) was identified as a co-regulator of gene expression with MEF2C.
Data show that high myocyte enhancer factor 2C (MEF2C) mRNA expression leads to overexpression of MEF2C protein, and these findings provide the rationale for therapeutic targeting of MEF2C transcriptional activation in acute myeloid leukemia (show BCL11A Antibodies).
this is the first report of a Greek-Cypriot patient with a MEF2C-related phenotype highlighting the rich variability in phenotypic expression and the ethnic diversity associated with this condition.
BCL2 (show BCL2 Antibodies) inhibitors may be a therapeutic candidate in vivo for patients with ETP-ALL with high expression levels of MEF2C.
MEF2C regulates the expression of G2/M checkpoint genes (14-3-3gamma (show YWHAG Antibodies), Gadd45b (show GADD45B Antibodies) and p21 (show CDKN1A Antibodies)) and the sub-cellular localization of CYCLIN B1 (show CCNB1 Antibodies).
The overall effect of MEF2C in hepatocellular carcinoma progression regulation was dictated by its subcellular distribution.
MEF2C/alpha-2-macroglobulin (show A2M Antibodies) axis functions in endothelial cells as a negative feed-back mechanism that adapts sprouting activity to the oxygen concentration thus diminishing inappropriate and excess angiogenesis.
MEF2C and MEF2D interact with the E3 ligase F-box protein SKP2, which mediates their subsequent degradation through the ubiquitin-proteasome system.
The cDNA sequence was analyzed and the 5' upstream region of the mef2c gene was isolated from porcine genomic DNA.
analysis of sequence and variations of the bovine myocyte enhancer factor 2C (MEF2C) gene promoter in Bos taurus cattle
We conclude that ILK (show ILK Antibodies) negatively and independently of PI3K regulated MEF2C phosphorylation activity and MCK (show CKM Antibodies) mRNA expression in C2C12 cells
Data show that three transcriptional factors Gata4 (show GATA4 Antibodies), Mef2c, and Tbx5 (show TBX5 Antibodies) (abbreviated as GMT (show GAMT Antibodies)) significantly improved murine embryonic stem cells (ESCs (show NR2E3 Antibodies)) differentiated into cardiomyocytes.
MEF2C has a role in regulating outflow tract alignment and transcriptional control of Tdgf1 (show TDGF1 Antibodies)
These findings uncover a novel role for Mef2c/d in coordinating the transcriptional network that promotes early B-cell development.
our data indicate that robust hypertrophic MEF2 activation in the heart in vivo requires a background of adiponectin (show ADIPOQ Antibodies) signaling and that adiponectin (show ADIPOQ Antibodies) signaling in primary isolated CM directly enhances MEF2 activity through activation of p38 MAPK (show MAPK14 Antibodies)
SmarcA4 (show SMARCA4 Antibodies) is required for synapse development and myocyte enhancer factor 2-mediated synapse remodeling
Our results identify novel target genes for MEF2 and define MEF2 as an important regulator of Leydig cell function and male reproduction.
this is a novel mechanism of H2S-mediated activation of MEF2C and induction of miR (show MLXIP Antibodies)-133a and inhibition of hypertrophy in HHcy cardiomyocytes.
The RBM4-MEF2C-miR-1 network constitutes a novel mechanism which programs the gene expression profile toward the development of brown adipocytes.
increased sclerostin (show SOST Antibodies) production achieved by HDAC5 (show HDAC5 Antibodies) shRNA is abrogated by simultaneous knockdown of MEF2C, indicating that MEF2C is a major target of HDAC5 (show HDAC5 Antibodies) in osteocytes
This locus encodes a member of the MADS box transcription enhancer factor 2 (MEF2) family of proteins, which play a role in myogenesis. The encoded protein, MEF2 polypeptide C, has both trans-activating and DNA binding activities. This protein may play a role in maintaining the differentiated state of muscle cells. Mutations and deletions at this locus have been associated with severe mental retardation, stereotypic movements, epilepsy, and cerebral malformation. Alternatively spliced transcript variants have been described.
myocyte-specific enhancer factor 2C
, myocyte enhancer factor 2C
, myocyte-specific enhancer factor 2C-like
, MADS box transcription enhancer factor 2, polypeptide C
, MADS box transcription enhancer factor 2, polypeptide C (myocyte enhancer factor 2C)
, Myocyte enhancer factor 2C protein
, myocyte enhancer factor 2c