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observations suggest that Rtn4A counteracts the Nrdp1-mediated degradation of ErbB3 by sequestering the ubiquitin ligase into ER tubules.
NOGO-A/B may be a negative prognostic factor of malignant melanoma.
LILRA3 (show LILRA3 ELISA Kits) significantly reversed Nogo-66-mediated inhibition of neurite outgrowth and promoted synapse formation in primary cortical neurons through regulation of the ERK (show EPHB2 ELISA Kits)/MEK (show MAP2K1 ELISA Kits) pathway.
Nogo-B expression is down-regulated in intrahepatic cholangiocarcinoma, the implication of which, however, remains to be investigated.
Data show that the mean peak serum neuroglobin and Nogo-A concentrations were both significantly higher in patients with an unfavorable outcome at 6 months after traumatic brain injury (TBI).
RTN4-C knockdown blocks cell cycle progression and cell growth in colorectal cancer cell lines.
Epithelial RTN-4B/NOGO-B was downregulated in human and experimental inflammatory bowel disease
The Nogo-B-PirB (show LILRB3 ELISA Kits) axis controls macrophage-mediated vascular remodeling.
a novel mechanism that functionally couples cAMP signaling with the proteolytic turnover of NOGO-A, positively impacting on neurite outgrowth in mammalian brain.
The RTN4 del allele could significantly increase NSCLC risk.
The steady-state level of reticulon 4-B mRNA was shown to be up-regulated by pressure, but not by mechanical stretch; close association with endoplasmic reticulum
deregulation of Nogo-C by miRNA may be a potential therapeutic target for ischemia-related heart diseases.
stimulated angiogenesis upon Nogo-A gene deletion results in the insertion of complete capillary micro-networks and not just single vessels into existing vascular networks.
The increase of Nogo-A expression can selectively influence the distribution of inwardly rectifying potassium channel (show KCNAB2 ELISA Kits) 4.1 in glia but is not essential for inwardly rectifying potassium channel (show KCNAB2 ELISA Kits) 4.1-mediated potassium conductance at the plasma membrane in physiological conditions.
Nogo-A restricts visual experience-driven plasticity of the optokinetic response and plays a role in the segregation and maintenance of retinal projections to the brain.
The Ad-Nogo-B-treated mice also had less severe lung injury, less alveolar protein exudation, and a higher number of macrophages but less neutrophil infiltration compared with Ad-RFP (show RFP ELISA Kits)-treated mice.
Nogo knockdown (KD) in non-transformed and Ras-transformed cells both enhanced virus-induced IFN response, suggesting that both cleaved and uncleaved Nogo can suppress IFN response.
the Nogo/NgR (show RTN4R ELISA Kits) signal might be involved in multiple processes in various inflammation-associated CNS diseases.
identifies Nogo-B as a key inhibitor of local sphingolipid synthesis
The findings indicate a critical role of Nogo-B and GRAMD4 (show GRAMD4 ELISA Kits) in trafficking of TLR9 (show TLR9 ELISA Kits).
rtn4a is essential for embryonic development and patterning of the nervous system.
data demonstrates that zebrafish Rtn4/Nogo transcripts might be generated by coupling mechanisms of alternative first exons and alternative promoter usage.
The present results have uncovered new factors underlying successful axon regeneration in the fish CNS: Binding of Nogo-66 of fish RTN-4 to NgR (show RTN4R ELISA Kits) is growth promoting on fish retinal ganglion cell axon growth
Data suggest that localized Nogo/Nogo66 expression defines inhibitory territories that through repulsion restrict axon growth to permissive regions.
This gene belongs to the family of reticulon encoding genes. Reticulons are associated with the endoplasmic reticulum, and are involved in neuroendocrine secretion or in membrane trafficking in neuroendocrine cells. The product of this gene is a potent neurite outgrowth inhibitor which may also help block the regeneration of the central nervous system in higher vertebrates. Alternatively spliced transcript variants derived both from differential splicing and differential promoter usage and encoding different isoforms have been identified.
, My043 protein
, neurite growth inhibitor 220
, neurite outgrowth inhibitor
, neuroendocrine-specific protein C homolog
, reticulon 5
, reticulon 4
, GLUT4 vesicle 20kDa protein
, glut4 vesicle 20 kDa protein
, nogo protein