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Allelic variation of the rs701428 SNP of RTN4R (show RTN4R ELISA Kits) was significantly associated with volumetric differences in gray matter of the lingual gyrus and cuneus of the occipital lobe. Moreover, occipital gray matter volumes were robustly associated with ultra high risk symptoms of psychosis in the presence of the G allele of rs701428.
Panax notoginseng saponins provide neuroprotective effects in a rat model of cerebral ischemia and SH-SY5Y cells exposed to oxygen/glucose deprivation injury by inhibiting the overexpression of NgR1 (show NEUROG1 ELISA Kits), RhoA (show RHOA ELISA Kits), and ROCK2 (show ROCK2 ELISA Kits).
(188)Re-NGR (show RTN4R ELISA Kits)-VEGI (show TNFSF15 ELISA Kits) has the potential as a theranostic agent.
NOGO-A/B may be a negative prognostic factor of malignant melanoma.
LILRA3 (show LILRA3 ELISA Kits) significantly reversed Nogo-66-mediated inhibition of neurite outgrowth and promoted synapse formation in primary cortical neurons through regulation of the ERK (show EPHB2 ELISA Kits)/MEK (show MAP2K1 ELISA Kits) pathway.
Nogo-B expression is down-regulated in intrahepatic cholangiocarcinoma, the implication of which, however, remains to be investigated.
Data show that the mean peak serum neuroglobin and Nogo-A concentrations were both significantly higher in patients with an unfavorable outcome at 6 months after traumatic brain injury (TBI).
Messenger RNA expression from RTN4R in human cortical brain tissue correlated significantly with the genotypes of rs701427. Observations suggest that a functional RTN4R gene variant is associated with sporadic ALS.
RTN4-C knockdown blocks cell cycle progression and cell growth in colorectal cancer cell lines.
Epithelial RTN-4B/NOGO-B was downregulated in human and experimental inflammatory bowel disease
The steady-state level of reticulon 4-B mRNA was shown to be up-regulated by pressure, but not by mechanical stretch; close association with endoplasmic reticulum
deregulation of Nogo-C by miRNA may be a potential therapeutic target for ischemia-related heart diseases.
stimulated angiogenesis upon Nogo-A gene deletion results in the insertion of complete capillary micro-networks and not just single vessels into existing vascular networks.
Here we identify that the nogo-66 receptor (show RTN4R ELISA Kits) 1 gene restricts an early and essential step in OD plasticity to the critical period. These findings link the emerging circuit-level description of OD plasticity to the genetic regulation of the critical period. Understanding how plasticity is confined to critical periods may provide clues how to better treat amblyopia.
The increase of Nogo-A expression can selectively influence the distribution of inwardly rectifying potassium channel (show KCNAB2 ELISA Kits) 4.1 in glia but is not essential for inwardly rectifying potassium channel (show KCNAB2 ELISA Kits) 4.1-mediated potassium conductance at the plasma membrane in physiological conditions.
Nogo-A restricts visual experience-driven plasticity of the optokinetic response and plays a role in the segregation and maintenance of retinal projections to the brain.
Rtn4r expression was reduced in spinal motor neurons from mice with a transgenic model of ALS.
This study detected the expression of NgR1 (show NEUROG1 ELISA Kits) in microglia during development and found that NgR1 (show NEUROG1 ELISA Kits) protein expression increased significantly in microglia with aging.
The Ad-Nogo-B-treated mice also had less severe lung injury, less alveolar protein exudation, and a higher number of macrophages but less neutrophil infiltration compared with Ad-RFP (show RFP ELISA Kits)-treated mice.
Nogo knockdown (KD) in non-transformed and Ras-transformed cells both enhanced virus-induced IFN response, suggesting that both cleaved and uncleaved Nogo can suppress IFN response.
Ngr1 (show NEUROG1 ELISA Kits) regulates tactile and motor task performance, it does not limit the rate of tactile or motor learning nor determine the low set point for synaptic turnover in adult sensory cortex.
This gene encodes the receptor for reticulon 4, oligodendrocyte myelin glycoprotein and myelin-associated glycoprotein. This receptor mediates axonal growth inhibition and may play a role in regulating axonal regeneration and plasticity in the adult central nervous system.
, reticulon 4
, neurite outgrowth inhibitor
, Human NogoA
, My043 protein
, neurite growth inhibitor 220
, neuroendocrine-specific protein C homolog
, reticulon 5
, GLUT4 vesicle 20kDa protein
, glut4 vesicle 20 kDa protein
, nogo protein
, Nogo-66 receptor
, nogo receptor
, reticulon-4 receptor
, nogo receptor; Nogo-66 receptor
, Nogo66 receptor
, nogo-66 receptor