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Rat (Rattus) p65 ELISA Kit for Sandwich ELISA - ABIN368280
Ahmed, El Morsy, Ahmed: Pomegranate extract protects against cerebral ischemia/reperfusion injury and preserves brain DNA integrity in rats. in Life sciences 2014
Human p65 ELISA Kit for Sandwich ELISA - ABIN366831
Wu, Yao, Wang, Qiu, Sai, Yang, Yao, Li, Bian, Wang, Yao: Suppression of human hepatoma (HepG2) cell growth by nuclear factor-kappaB/p65 specific siRNA. in Tumour biology 2010
In the premature rupture of membranes group,the NF-kappaB (show NFKB1 ELISA Kits) p65 (show GORASP1 ELISA Kits) and sTREM-1 levels in maternal blood were significantly higher in women with chorioamnionitis than women without chorioamnionitis (P<.05).
IL-10 (show IL10 ELISA Kits) stimulation activated the JAK (show JAK3 ELISA Kits)/Stat-3 (show STAT3 ELISA Kits) and PI3K (show PIK3CA ELISA Kits)/Akt (show AKT1 ELISA Kits) signaling pathways. Moreover, IL-10 (show IL10 ELISA Kits) treatment increased translocation of p65 NF-kappaB into the nuclear compartment, and up-regulated expression of the pro-survival proteins Bcl-2 (show BCL2 ELISA Kits) and Bcl-xL (show BCL2L1 ELISA Kits).
In glioma, elevated miR (show MLXIP ELISA Kits)-221 expression is a biomarker for glioma. DNM3 (show DNM3 ELISA Kits) is a target of miR (show MLXIP ELISA Kits)-221 and RELA regulates miR (show MLXIP ELISA Kits)-221 expression.
Stat5 (show STAT5A ELISA Kits) activation increased the DNA binding activity of NF-kappaB (show NFKB1 ELISA Kits) though binding of p-Stat5 (show STAT5A ELISA Kits) and p-RelA in nucleus.
These findings suggested that USP14 induces NF-kappaB (show NFKB1 ELISA Kits) activity and ERK1/2 (show MAPK1/3 ELISA Kits) phosphorylation triggered by microbial infection.
These results suggest that SUMO1 (show SUMO1 ELISA Kits) contributes to hepatocellular carcinoma progression by promoting p65 (show GORASP1 ELISA Kits) nuclear translocation
knockdown of NAP1L1 (show NAP1L1 ELISA Kits) suppresses IkappaBalpha (show NFKBIA ELISA Kits) degradation and nuclear transport of p65 (show GORASP1 ELISA Kits) subunit after treatment with TNF-a (show TNF ELISA Kits) stimulation, leading to attenuation of the NF-kappaB (show NFKB1 ELISA Kits) transcriptional activity, whereas NAP1L4 (show NAP1L4 ELISA Kits) knockdown remains silent.results of this study suggest that NAP1L1 (show NAP1L1 ELISA Kits) downregulation renders the cell vulnerable to apoptotic cell death through attenuation of NF-kappaB (show NFKB1 ELISA Kits) transcriptional activity on the anti-apop...
Data show that miR (show MLXIP ELISA Kits)-155-5p downregulation triggers the transition depending on NF-kappaB (show NFKB1 ELISA Kits) p65 (show GORASP1 ELISA Kits) activation.
This study investigated the effects of DYN (show SPTBN4 ELISA Kits) 3-14 on lipopolysaccharide (LPS (show IRF6 ELISA Kits))-induced nuclear factor-kappaB/p65 (NF-kappaB/p65) nuclear translocation.These findings indicate that DYN (show SPTBN4 ELISA Kits) 3-14 is a potential TLR4 (show TLR4 ELISA Kits) antagonist that alters cellular signaling in response to LPS (show IRF6 ELISA Kits) and cytokine release, implicating a role for biotransformed endogenous opioid peptides in immunomodulation.
p65 (show GORASP1 ELISA Kits) expression correlates with elevated total cellular levels of STAT3 (show STAT3 ELISA Kits) and STAT1 (show STAT1 ELISA Kits) and supports activation of these transcription factors.
TMZ pretreatment effectively reduced the myocardial damage caused by CME via inhibiting the PDCD4 (show PDCD4 ELISA Kits)/NF-kappaB (show NFKB1 ELISA Kits)/ TNF-alpha (show TNF ELISA Kits) pathway in cardiomyocytes.
Transmissible gastroenteritis virus infection activates NF-kappaB (show NFKB1 ELISA Kits).
Zinc finger nuclease (show DCLRE1C ELISA Kits) in-embryo editing of the RELA locus generated live born domestic pigs with the warthog RELA orthologue, associated with resilience to African Swine Fever.
SIRT1 (show SIRT1 ELISA Kits), p53 (show TP53 ELISA Kits) and NF-kappaB (show NFKB1 ELISA Kits) are involved in the control of both the proliferation and the apoptosis of ovarian cells.
Porcine Coro1A (show CORO1A ELISA Kits) is an important immunity related gene that helps to inhibit NF-kB activation during H. parasuis infection.
We propose that the variation in RELA identified between the warthog and domestic pig has the potential to underlie the difference between tolerance and rapid death upon African swine fever virus infection.
The characterization of porcine NF-kappaB (show NFKB1 ELISA Kits) p65 (show SYT1 ELISA Kits) subunit (pp65 (show LCP1 ELISA Kits)), is reported.
Viral non-structural protein 1 suppressed host TNF-alpha (show TNF ELISA Kits) promoter by inhibiting NF-kappaB (show NFKB1 ELISA Kits) and Sp1 (show SP1 ELISA Kits) promoter binding activity.
Hepatic SREBP-1c (show SREBF1 ELISA Kits)-mediated lipid synthesis and the NF-kappaB (show NFKB1 ELISA Kits) inflammatory pathway were both overinduced in cows with fatty liver.
Cytopathic bovine viral diarrhea virus strain induces immune marker production in bovine cells through the NF-kappaB (show NFKB1 ELISA Kits) signaling pathway.
The role of NF-kappaB (show NFKB1 ELISA Kits) and C/EBP (show CEBPA ELISA Kits) factors in regulating basal and pathogen-induced expression of both genes from cattle, is investigated.
Altering the extracellular matrix to promote p38 (show MAPK14 ELISA Kits) activation in cells on fibronectin (show FN1 ELISA Kits) suppresses NF-kappaB (show NFKB1 ELISA Kits) activation, suggesting a novel therapeutic strategy for treating
full length coding sequence of the cattle transcription factor p65 was isolated and cloned
the pathogen causing subclinical mastitis impairs NF-kappaB (show NFKB1 ELISA Kits) activation in MEC (show CCL28 ELISA Kits) thereby severely weakening the immune response,induction of IL-8 (show IL8 ELISA Kits) and TNFalpha (show TNF ELISA Kits), in the udder
These results confirm that VEGI (show TNFSF15 ELISA Kits) utilizes NF-kappaB (show NFKB1 ELISA Kits) as a pro-survival role factor in endothelial cells.
TNF-alpha (show TNF ELISA Kits) and the transcription factor nuclear factor kappa B (NF-kappaB (show NFKB1 ELISA Kits)) p65 subunit were both upregulated in bleomycin-induced fibrotic lung tissue.
this study demonstrates that phosphorylation of p65 serine 467 augment NF-kappaB (show NFKB1 ELISA Kits) activity and exacerbates various deleterious effects of overnutrition in mice
RORalpha/gamma are important therapeutic targets for cutaneous inflammation; RORa (show RORA ELISA Kits) and RORg (show RORC ELISA Kits) have roles in inflammation in mouse models of atopic dermatitis and acute irritant dermititis
These findings identify differential requirements for the canonical NF-kappaB (show NFKB1 ELISA Kits) subunits c-REL and RELA at distinct stages of mature B-cell development.
Expansion of myeloid-derived suppressor cells with aging in the bone marrow of mice through a Rela-dependent mechanism.
p65 expression correlates with elevated total cellular levels of STAT3 (show STAT3 ELISA Kits) and STAT1 (show STAT1 ELISA Kits) and supports activation of these transcription factors.
Acanthopanax senticosus polysaccharide pretreatment may be associated with inhibition of the NF-kappaB (show NFKB1 ELISA Kits)/MLCK (show MYLK ELISA Kits) pathway and concomitant amelioration of LPS (show TLR4 ELISA Kits)-induced tight junction dysfunction of intestinal epithelium in endotoxemia
Data suggest that activation of NFkappaB/RelA can be moderated by dietary factors; here, naringenin (an anti-inflammatory, antioxidant flavanone dietary supplement) reduces lipopolysaccharide-induced inflammatory pain, mechanical hyperalgesia, and thermal hyperalgesia; naringenin reduces mononuclear-leukocyte/neutrophil/macrophage recruitment by inhibiting NFkappaB/RelA activation.
hyperresponsive promoter engaged the RelA:p52 dimer generated during costimulation of macrophages through TLR4 (show TLR4 ELISA Kits) and LTbetaR to trigger synthesis of IkappaBalpha (show NFKBIA ELISA Kits) at late time points, which prevented the late-acting RelA cross-talk response.
NF-kappa-B is a ubiquitous transcription factor involved in several biological processes. It is held in the cytoplasm in an inactive state by specific inhibitors. Upon degradation of the inhibitor, NF-kappa-B moves to the nucleus and activates transcription of specific genes. NF-kappa-B is composed of NFKB1 or NFKB2 bound to either REL, RELA, or RELB. The most abundant form of NF-kappa-B is NFKB1 complexed with the product of this gene, RELA. Four transcript variants encoding different isoforms have been found for this gene.
, nuclear factor NF-kappa-B p65 subunit
, nuclear factor of kappa light polypeptide gene enhancer in B-cells 3
, transcription factor p65
, v-rel reticuloendotheliosis viral oncogene homolog A
, NF-kappaB transcription factor p65 subunit
, v-rel reticuloendotheliosis viral oncogene homolog A, nuclear factor of kappa light polypeptide gene enhancer in B-cells 3, p65
, nuclear factor kappa B subunit p65
, avian reticuloendotheliosis viral (v-rel) oncogene homolog A
, p65 NF kappaB
, p65 NF-kappa B
, p65 NFkB
, NF-kB p65 subunit