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Human TNFRSF1A ELISA Kit for Sandwich ELISA - ABIN625089
Sennikov, Alshevskaya, Shkaruba, Chumasova, Sizikov, Lopatnikova: Expression of TNF? membrane-bound receptors in the peripheral blood mononuclear cells (PMBC) in rheumatoid arthritis patients. in Cytokine 2015
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Human TNFRSF1A ELISA Kit for Sandwich ELISA - ABIN417656
Mern, Fontana, Beierfuß, Thomé, Hegewald et al.: A combinatorial relative mass value evaluation of endogenous bioactive proteins in three-dimensional cultured nucleus pulposus cells of herniated intervertebral discs: identification of potential ... in PLoS ONE 2013
Human TNFRSF1A ELISA Kit for Sandwich ELISA - ABIN456707
Hofmann-Kiefer, Knabl, Martinoff, Schiessl, Conzen, Rehm, Becker, Chappell: Increased serum concentrations of circulating glycocalyx components in HELLP syndrome compared to healthy pregnancy: an observational study. in Reproductive sciences (Thousand Oaks, Calif.) 2013
The highest levels of TNFR1 are independently associated with progression of renal disease and death in type 2 diabetic nephropathy.
High plasma levels of TNFR1 and TNFR2 (show TNFRSF1B ELISA Kits) were associated with incident intracerebral hemorrhage.
Renal clear cell carcinoma cells cells express increased amounts of RIPK1 (show RIPK1 ELISA Kits) and RIPK3 (show RIPK3 ELISA Kits) and are poised to undergo necroptosis in response to TNFR1 signaling.
TRIM28 (show TRIM28 ELISA Kits) acts as a central factor in controlling endothelial inflammatory responses and angiogenic activities by retaining expression of TNFR-1 and -2 and VEGF receptor 2 in endothelial cells
Since mumps virus SH coimmunoprecipitated with tumor necrosis factor receptor 1 (TNFR1), RIP1, and IRAK1 (show IRAK1 ELISA Kits), we hypothesize that SH exerts its NF-kappaB (show NFKB1 ELISA Kits) activation inhibitory function by interacting with TNFR1, interleukin-1 receptor type 1 (show IL-1R1 ELISA Kits) (IL-1R1), and TLR3 (show TLR3 ELISA Kits) complexes in the plasma membrane of infected cells.
a specific link between the penetrance of the TNFRSF1A mutation and the observed T cell phenotype, is reported.
Collectively, this study provides more insights into RELT (show RELT ELISA Kits) expression, RELT (show RELT ELISA Kits) family member function, and the mechanism of RELT (show RELT ELISA Kits)-induced death.
Burkholderia cenocepacia BcaA (show ARID4B ELISA Kits) binds to tumor necrosis factor receptor 1.
In SOD1 (show SOD1 ELISA Kits)(G93A) spinal cords, we verified a strict correlation in the expression of the TNFalpha (show TNF ELISA Kits), TNFR1 and GDNF triad at different stages of disease progression. Yet, ablation of TNFR1 completely abolished GDNF rises in both SOD1 (show SOD1 ELISA Kits)(G93A) astrocytes and spinal cords, a condition that accelerated motor neuron degeneration (show CLN8 ELISA Kits) and disease progression
TNFRSF1A variants were identified in 10 tumor necrosis factor (show TNF ELISA Kits) receptor-associated periodic syndrome patients from 10 independent families. The T61I variant was found in patients, while the V136M and S321I variants were found in 1 patient each. All the patients were heterozygous for the variants. Among the healthy controls, 7 of 363 individuals were heterozygous for the T61I variant.
The deficiency of TNFRp55 promoted the development of endometriosis.
TNFalpha (show TNF ELISA Kits) enhanced murine NK cell IFNgamma production via TNFR2 (show TNFRSF1B ELISA Kits) in vitro
TNFR2 (show TNFRSF1B ELISA Kits) sensitizes macrophages for endogenous TNF (show TNF ELISA Kits)-induced TNFR1-mediated necroptosis
this study shows that epithelial TNFR1 signaling promotes mucosal repair in inflammatory bowel disease
HACE1 (show HACE1 ELISA Kits) controls TNF (show TNF ELISA Kits)-elicited cell fate decisions and exerts tumor suppressor and anti-inflammatory activities via a TNFR1-RIP3 (show MPRIP ELISA Kits) kinase-necroptosis pathway.
impaired TNF (show TNF ELISA Kits)/TNFR2 (show TNFRSF1B ELISA Kits) signaling enhances Th2 and Th17 polarization and aggravates allergic airway inflammation.
TNFR2 (show TNFRSF1B ELISA Kits) activation exerted beneficial effects on OPA1 (show MED12 ELISA Kits) expression in an aortic constriction model.
Following activation by transmembrane TNF (show TNF ELISA Kits), TNFR2 (show TNFRSF1B ELISA Kits) initiates pathways that drive oligodendrocytes into a reparative mode contributing to remyelination following disease
The apoptotic signals through TNFR1 appear critical for the pathogenesis of pulmonary emphysema.
Retinal ischemia results in increased expression of TNF-alpha (show TNF ELISA Kits) and its receptors (TNF-R1 and TNF-R2 (show TNFRSF1B ELISA Kits)).
These results suggest that TNF-alpha (show TNF ELISA Kits) sources include immune cells, as well as large and small luteal cells, and that TNF-RI and TNF-RII (show TNFRSF1B ELISA Kits) are present in the luteal cells of the bovine corpus luteum.
The expression and cellular localization of tumor necrosis factor-alpha (TNF (show TNF ELISA Kits)) and its receptors (TNFRI and TNFRII (show TNFRSF1B ELISA Kits)) mRNAs and proteins, were determined.
The upregulation of TNFRI mRNA expression by IFNG (show IFNG ELISA Kits) suggests that TNF (show TNF ELISA Kits) and IFNG (show IFNG ELISA Kits) synergistically affect the death of luteal endothelial cells resulting in acute luteolysis
TNF (show TNF ELISA Kits) binding induces release of AIP1 (DAB2IP (show DAB2IP ELISA Kits)) from TNFR1, resulting in cytoplasmic translocation and concomitant formation of an intracellular signaling complex comprised of TRADD (show TRADD ELISA Kits), RIP1 (show RALBP1 ELISA Kits), TRAF2 (show TRAF2 ELISA Kits), and AIPl.
Targeted gene knockdown of TNFRSF1B (show TNFRSF1B ELISA Kits) in zebrafish embryos results in the induction of a caspase-8 (show CASP8 ELISA Kits), caspase-2 (show CASP2 ELISA Kits) and P53 (show TP53 ELISA Kits)-dependent apoptotic program in endothelial cells that bypasses caspase-3 (show CASP3 ELISA Kits).
The protein encoded by this gene is a member of the TNF-receptor superfamily. This protein is one of the major receptors for the tumor necrosis factor-alpha. This receptor can activate NF-kappaB, mediate apoptosis, and function as a regulator of inflammation. Antiapoptotic protein BCL2-associated athanogene 4 (BAG4/SODD) and adaptor proteins TRADD and TRAF2 have been shown to interact with this receptor, and thus play regulatory roles in the signal transduction mediated by the receptor. Germline mutations of the extracellular domains of this receptor were found to be associated with the autosomal dominant periodic fever syndrome. The impaired receptor clearance is thought to be a mechanism of the disease.
, tumor necrosis factor binding protein 1
, tumor necrosis factor receptor 1A isoform beta
, tumor necrosis factor receptor superfamily member 1A
, tumor necrosis factor receptor type 1
, tumor necrosis factor-alpha receptor
, TNF receptor alpha chain
, tumor necrosis factor receptor 1
, tumor necrosis factor receptor type I
, p55 TNF receptor
, tumor necrosis factor receptor p60
, TNF Receptor 1 (TR1)
, tumor necrosis factor type I
, tumor necrosis factor receptor superfamily, member 1A
, tumor necrosis factor receptor superfamily member 1A-like