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Rat (Rattus) Tumor Necrosis Factor ELISA Kit for Sandwich ELISA - ABIN625216
Mohamed, Elmelegy, El-Aziz, Abdel Kawy, AbdEl-Samad, El-Kharashi: Hepatoprotective effects of early pentoxifylline administration on hepatic injury induced by concanavalin A in rat. in Canadian journal of physiology and pharmacology 2014
Show all 83 Pubmed References
Mouse (Murine) Tumor Necrosis Factor ELISA Kit for Sandwich ELISA - ABIN625182
Bil-Lula, Sochocka, Zatońska, Szuba, Sawicki, Woźniak: Adenovirus type 9 enhances differentiation and decreases cytokine release from preadipocytes. in Journal of medical virology 2014
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Mouse (Murine) Tumor Necrosis Factor ELISA Kit for Sandwich ELISA - ABIN415537
Li, Zhu, Shen, Wan, Arnold, Peng: Deficiency of rac1 blocks NADPH oxidase activation, inhibits endoplasmic reticulum stress, and reduces myocardial remodeling in a mouse model of type 1 diabetes. in Diabetes 2010
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Rat (Rattus) Tumor Necrosis Factor ELISA Kit for Sandwich ELISA - ABIN416259
Zhang, Huang, Qin, Ren: Anti-inflammatory effect of Guan-Xin-Er-Hao via the nuclear factor-kappa B signaling pathway in rats with acute myocardial infarction. in Experimental animals / Japanese Association for Laboratory Animal Science 2010
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Rabbit Tumor Necrosis Factor ELISA Kit for Sandwich ELISA - ABIN415985
Hoekstra, van Lienden, Verheij, van der Loos, Heger, van Gulik: Enhanced tumor growth after portal vein embolization in a rabbit tumor model. in The Journal of surgical research 2013
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Human Tumor Necrosis Factor ELISA Kit for Sandwich ELISA - ABIN414928
Deng, Zhang, Wang, Xu, Zhang, Yang, Mao, Gao, Song, Kong, Qu: Soluble BAFF-R produced by decidual stromal cells plays an inhibitory role in monocytes and macrophages. in Reproductive biomedicine online 2012
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Rat (Rattus) Tumor Necrosis Factor ELISA Kit for Sandwich ELISA - ABIN1672752
Fan, Wang, Shi, Die, Wang, Dang: Tetramethylpyrazine protects spinal cord and reduces inflammation in a rat model of spinal cord ischemia-reperfusion injury. in Journal of vascular surgery 2011
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Human Tumor Necrosis Factor ELISA Kit for Sandwich ELISA - ABIN625102
Goktas, Coskun, Bicik, Horuz, Unsal, Serteser, Albayrak, Sarıca: Evaluating ESWL-induced renal injury based on urinary TNF-α, IL-1α, and IL-6 levels. in Urological research 2012
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Mouse (Murine) Tumor Necrosis Factor ELISA Kit for Sandwich ELISA - ABIN365457
Ren, Luo, Wu, Liu, Yu, Fang, Li, Yin, Wu: Dietary L-glutamine supplementation improves pregnancy outcome in mice infected with type-2 porcine circovirus. in Amino acids 2013
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Human Tumor Necrosis Factor ELISA Kit for Sandwich ELISA - ABIN411361
Luo, Liu, Zhang, Huang, Yang, Zhang, Cui, Ma, Wang, Sun, Xiong, Zhu, Li, Wu, Jin, Hu, Gao: Protective effects of a novel trimerized sTNFRII on acute liver injury. in International immunopharmacology 2012
Show all 105 Pubmed References
Our findings demonstrate that TNF-alpha plays a pivotal role in HF-mediated neuroinflammation and associated alterations of cortical dendritic spine density and has the potential to reveal novel treatment strategies for HF-associated memory deficits.
the present study assessed the combined impacts of estrogens and bacterial infection on the insulin (show INS ELISA Kits)-like growth factors (IGF) and tumor-necrosis factor (TNF)-alpha.
The expression of miR (show MYLIP ELISA Kits)-125b-1 regulated by NF-kappaB (show NFKB1 ELISA Kits) has been reported in diverse cell types under various stimuli, this study found that its expression was also significantly regulated by NF-kappaB (show NFKB1 ELISA Kits) in TNFalpha-stimulated HeLa and HepG2 cells
In the Han Chinese population, the allele 'A' of TNF-alpha -308 may increase the risk for osteoarthritis (OA), whereas TNF-alpha -238 polymorphisms do not play a role in OA patients.
Letter: serum TNF-alpha was not elevated in chronic spontaneous urticaria.
Metastatic cells can escape the proapoptotic effects of TNF-alpha through increased autocrine IL-6 (show IL6 ELISA Kits)/STAT3 (show STAT3 ELISA Kits) signaling
The predicted 3D structures of IL-1beta (show IL1B ELISA Kits), TNF-alpha, and IL-8 (show IL8 ELISA Kits) from Oncorhynchus mykiss, Dicentrarchus labrax, and Chionodraco hamatus are reported and the differences between them and their mammalian counterparts investigated.
The mitochondrial dysfunction may be caused by increased brain levels of TNFalpha without physiological stress but will be exacerbated after middle cerebral artery occlusion.
TNF-alpha is released rapidly in trout macrophages after stimulation with lipopolysaccharide and zymosan, and its secretion is dependent on TACE (show ADAM17 ELISA Kits)-like activity that is regulated by ERK (show MAPK1 ELISA Kits), JNK (show MAPK8 ELISA Kits) and p38MAPK (show MAPK14 ELISA Kits) signaling pathways.
Recombinant, as well as native trout TNF-alpha, directly stimulates glucose uptake in trout muscle cells and provide evidence, for the first time in nonmammalian vertebrates, for a potential regulatory role of TNF-alpha in skeletal muscle metabolism.
In intestinal cells, TNF activates NF-kappaB (show NFKB1 ELISA Kits), which reduces expression of the Cl(-) / HCO3(-) exchanger SLC26A3 (show SLC26A3 ELISA Kits), via direct binding to the promoter region.
The levels of IL-1 (show IL1A ELISA Kits), IL-6 (show IL6 ELISA Kits) and TNF-alpha in GCF (show GUCY2F ELISA Kits) were higher in patients with ischemic stroke but the difference was not significant. In conclusion, ischemic stroke was found to be associated with higher levels of IL-1beta (show IL1B ELISA Kits) and IL-6 (show IL6 ELISA Kits) in serum
A hypo-fucosylated form of anti-TNF binds FcgammaRIIIa with higher affinity and induces development o (show MRC1 ELISA Kits)f CD206(+) macrophages in human PBMCs, especially PBMCs that express low-affinity FcgammaRIIIa.
High TNF-alpha levels are associated with B12 (show NDUFB3 ELISA Kits) deficiency and anemia.
A higher intake of fruits was related to lower insulin (show INS ELISA Kits), HOMA-IR, and TNF-alpha promoter methylation. A healthy dietary pattern and higher fruit intake (particularly apples and citrus fruits) were related to better glucose tolerance in healthy subjects, which could be mediated by lower TNF-alpha methylation.
The TNF-alpha-308A polymorphism is unlikely to play a major role in the risk for ocular C. Trachomatis in the Chinese population.
Calcipotriol increases the immunosuppressive power of betamethasone by suppressing the inflammatory TNF-alpha - IL-23 (show IL23A ELISA Kits) - IL-17 (show IL17A ELISA Kits) axis in psoriasis.
study reports that plasmacytoid dendritic cells (pDCs) incubated with IFNlambda3 plus IL-3 (show IL-3 ELISA Kits) synergize in triggering the production of large amounts of both IFNalpha and TNFalphaTNFalpha; production of IFNalpha by IFNlambda3 plus IL-3 (show IL-3 ELISA Kits)-treated pDCs is mostly dependent on endogenously produced TNFalpha
Our findings suggest that tumor necrosis factor-alpha promoter polymorphism may be an important candidate for body mass index, fasting glucose and postprandial triacylglycerol response.
High TNF-alpha expression is associated with breast cancer.
FcgammaR engagement by anti-TNF is required for reduction of colitis in mice and development of CD206 (show MRC1 ELISA Kits)(+) macrophages.
long-term PM2.5 exposure caused hepatic steatosis and dyslipidemia through triggering inflammation, which was, at least partly, dependent on iRhom2 (show RHBDF2 ELISA Kits)/TNF-alpha pathway in liver-resident macrophages
Pretreating mice with carrageenan once a day before injecting LPS (show TLR4 ELISA Kits) increased the levels of IL-10 (show IL10 ELISA Kits) by 2.5-fold and reduced TNF-alpha production by 2-fold compared with control. So, kappa/beta-carrageenan alone and in combination with LPS (show TLR4 ELISA Kits) enhanced the cellular activity and mobility of peritoneal macrophages by increasing cell adhesion and migration compared with control.
TNF/Lymphotoxin alpha/beta deficiency influences PM2.5 exposure-induced response of energy metabolism through alterations in both food intake and energy expenditure.
TNF has an essential role in TLR ligand-mediated but not protease-mediated allergic airway inflammation
TonEBP (show NFAT5 ELISA Kits) expression correlated with canonical osmoregulatory targets TauT/SLC6A6 (show TAUT ELISA Kits), SMIT/SLC5A3 (show SLC5A3 ELISA Kits), and AR/AKR1B1 (show AKR1B1 ELISA Kits), supporting in vitro findings that the inflammatory milieu during IDD (show DGS2 ELISA Kits) does not interfere with TonEBP (show NFAT5 ELISA Kits) osmoregulation. In summary, whereas TonEBP (show NFAT5 ELISA Kits) participates in the proinflammatory response to TNF-alpha
Increased basal transmission and plasticity of glutamate (show GRIN1 ELISA Kits) synapses that was related to increased levels of tumor necrosis factor alpha (TNFalpha).
Results showed that lncRNA SeT and miR (show MLXIP ELISA Kits)-155 mediate Tnfalpha gene dosage control by regulating the homologous pairing of the two gene alleles and ultimately regulating its biallelic pattern.
Internalized CpG oligodeoxynucleotide (ODN)-conjugated PPMs (PPM-CpG) greatly enhance the induction of selected cytokines (TNF-alpha and IL-6 (show IL6 ELISA Kits)) in RAW 264.7 cells compared to that by the soluble CpG ODN and ionic complexes.
TMZ pretreatment effectively reduced the myocardial damage caused by CME via inhibiting the PDCD4 (show PDCD4 ELISA Kits)/NF-kappaB (show NFKB1 ELISA Kits)/ TNF-alpha pathway in cardiomyocytes.
TNF-alpha, secreted from activated Monocytes, mediates the downregulation of OTX2 (show OTX2 ELISA Kits) and essential retinal pigment epithelium genes.
TNF-alpha induced MMP-13 (show MMP13 ELISA Kits) expression by condylar cells might be involved in the degradation of the juvenile condyle.
TNF-alpha was able to promote theca interna cell proliferation. Our results suggest that TNF-alpha might play a role in hyperandrogenism, cortex thickness, and the increased ovary volume observed in polycystic ovaries.
This study showed that the -791(C-->T) mutation of the TNF-alpha gene could be considered an important potential genetic marker of enterotoxigenic Escherichia coli F18 (show MAMLD1 ELISA Kits) resistance.
Both Nsp1beta and Nsp11 of porcine reproductive and respiratory syndrome virus were demonstrated to be responsible for the inhibitory effect on TNF-alpha production in pulmonary alveolar macrophages.
Study presents evidence demonstrating a single species of exotoxin ApxI, derived from A. pleuropneumoniae serotype 10, induces the expression and production of proinflammatory cytokines IL-1beta (show IL1B ELISA Kits), IL-8 (show IL8 ELISA Kits) and TNF-alpha in porcine alveolar macrophages.
the role of miR (show MYLIP ELISA Kits)-181a in adipocyte differentiation by regulation of TNF-alpha
The NOS (show NOS ELISA Kits) inhibitor, L-NMMA, significantly suppressed the combined effects of HT and CORM-2 on TNFalpha-triggered NFkappaBp65 phosphorylation as well as decreased cell viability.
High-volume hemofiltration improves hemodynamics and heart dysfunction in septic shock pigs, which may be attributed to reduction of TNF-alpha in myocardium but not in circulation.
Data suggest that luteolytic factors (such as TNFa, interferon gamma (show IFNG ELISA Kits), and PGF2a) control expression of MMP1 (show MMP1 ELISA Kits), other matrix metalloproteinases, and tissue inhibitors of metalloproteinase in cultured luteal cells.
These results suggest that polymorphism of the TNFalpha-824 A>G gene and mTNFalpha protein expression play an important role in the pathogenesis of enzootic bovine leukosis.
These results suggest that the endometrium might lower the TNF concentration in the blastocyst by (1) regulating TNF secretion into the uterine fluid and (2) inducing decreased TNF and TNFR2 (show TNFRSF1B ELISA Kits) mRNA transcription in the embryo.
There was a significant association between the proviral load and a low frequency of the G/G genotype of TNF-alpha at position -824.
SNP in the TNF-alpha gene affects immune function and reproductive performance in dairy cows.
Studied genotypic and expression profiling of partial TNF-alpha gene and its association with mastitis susceptibility in 129 crossbred cattle.
Messenger RNA and protein levels of prostaglandin (PG) E synthase (PGES (show PTGES ELISA Kits)), PGF2alpha receptor (PGFR), tumor necrosis factor-alpha (TNF) and Fas (show FAS ELISA Kits) were found to be higher in the corpus luteum of pregnancy than in corpus luteum of the cycle.
TNF-alpha up-regulates NaV1.7 (show SCN9A ELISA Kits) mRNA in both adrenal chromaffin cells and dorsal root ganglia (DRG) neurons, highlighting the peripheral nociceptive mechanism of TNF-alpha
These results provide evidence for a high prevalence of subclinical endometritis in repeat breeding cows as well as the involvement of TNFalpha and iNOS (show NOS2 ELISA Kits) pathways in the regulation of this pathological condition.
Exposure to follicular fluid transiently increased the transcript levels of IL8 (show IL8 ELISA Kits) and PTGS2 (show PTGS2 ELISA Kits), and decreased the expression of SOD2 (show SOD2 ELISA Kits), GPX3 (show GPX3 ELISA Kits), DAB2 (show DAB2 ELISA Kits), and NR3C1 (show NR3C1 ELISA Kits). TNF and IL6 (show IL6 ELISA Kits) levels were also decreased while those of NAMPT (show NAMPT ELISA Kits) were unaffected.
The results obtained showed that IL-6 (show IL6 ELISA Kits) expression in adipose tissue biopsies derived from animals with equine metabolic syndrome was enhanced while TNF-alpha levels of both groups were comparable.
Data indicate that tumor necrosis factor-alpha production is closely related to ovarian steroid actions.
These results indicate that in equine corpus luteum, cytokines TNF, IFNG (show IFNG ELISA Kits) and FASL (show FASL ELISA Kits) regulate nitric oxide activity, via eNOS (show NOS3 ELISA Kits) expression modulation.
These data show the presence of cytokines TNF and IFNG (show IFNG ELISA Kits), and their receptors, in the equine corpus luteum and indicate their potential involvement in regulation of luteal function.
The contribution of bronchial epithelium to airway inflammation, with focus on mRNA and protein expression of IL-6 (show IL6 ELISA Kits), IL-10 (show IL10 ELISA Kits) and TNF-alpha, in horses with recurrent airway obstruction during exacerbation and in remission is reported.
TUNEL staining was positively correlated with TNF-a protein expression. Our findings suggest that apoptosis can be induced in the vocal fold epithelium after 120min of modal intensity phonation. In contrast, shorter durations of vibration exposure do not result in apoptosis signaling.
Inflammatory factors such as TNF-alpha can stimulate MMP-2 (show MMP2 ELISA Kits)/9 activity in corneal epithelium cells. This may be a potential manipulating mechanism of MMP expression in the pathogenesis of corneal diseases.
The JNK (show MAPK8 ELISA Kits) pathway plays an important role in mechanical ventilation-stimulated TNF-alpha expression in alveolar macrophages, but the injury-stimulated IL-8 (show IL8 ELISA Kits) expression may be regulated by other signaling pathways.
The TNFalpha-evoked Cl- current.
IL-1beta (show IL1B ELISA Kits) and TNF-alpha expression increases significantly during acute lung injury. Ambroxol combined with low-dose heparin inhibits teh release of IL-1beta (show IL1B ELISA Kits) and TNF-alpha.
Hypercapnia increased expression of TNFa and decreased expression of NFKB in acute lung injury models.
In the early stages of myocardial ischemia, bone marrow stem cells are mobilized and home to ischemic myocardium with a concomitant increase in expression of cytokines VEGF (show VEGFA ELISA Kits) and TNFalpha.
Ammonium perchlorate can increase gene expressions of types I, III collagens, TGF-beta(1 (show TGFB1 ELISA Kits)) and TNF-alpha in lung of rabbits.
TNFalpha may either be directly or indirectly involved in vascular damage following an embolic stroke. Moreover, TNFalpha may mediate some of the detrimental effects of tPA (show PLAT ELISA Kits) on the vascular compartment
These interactions result in the induction of the TNF signaling pathway, activation of apoptosis, and DNA-damage stress response.
Findings showed that both mucosal compartments harbor similar percentages of memory CD4 (show CD4 ELISA Kits)(+) T cells and displayed comparable cytokine TNF-alpha responses to mitogenic stimulations prior to infection.
Primary role for IL-1beta (show IL1B ELISA Kits) and TNF-alpha in the triggering of preterm labor associated with inflammation or infection.
Data indicate that three single-nucleotide polymorphisms were identified in tumor necrosis factor-alpha (TNF-alpha) gene.
The results reveal a crucial role for TNFalpha/TNFR2 (show TNFRSF1B ELISA Kits) axis in the protection of the skin against DUOX1 (show DUOX1 ELISA Kits)-mediated oxidative stress.
TNF alpha is the first molecule identified that is produced by dying retinal neurons and is necessary to induce Muller glia to proliferate in the zebrafish retinal regeneration response.
The data suggest that Mycobacterium marinum SecA2 modulates adaptive immunity to promote granuloma stability, perhaps through induction of tumor necrosis factor alpha.
TNF is not required for tuberculous granuloma formation, but maintains granuloma integrity indirectly by restricting mycobacterial growth within macrophages and preventing their necrosis.
The main proinflammatory effects of fish TNF-alpha are mediated through the activation of endothelial cells and point to the complexity of the evolution that has taken place in the regulation of innate immunity by cytokines.
This gene encodes a multifunctional proinflammatory cytokine that belongs to the tumor necrosis factor (TNF) superfamily. This cytokine is mainly secreted by macrophages. It can bind to, and thus functions through its receptors TNFRSF1A/TNFR1 and TNFRSF1B/TNFBR. This cytokine is involved in the regulation of a wide spectrum of biological processes including cell proliferation, differentiation, apoptosis, lipid metabolism, and coagulation. This cytokine has been implicated in a variety of diseases, including autoimmune diseases, insulin resistance, and cancer. Knockout studies in mice also suggested the neuroprotective function of this cytokine.
, TNF, macrophage-derived
, TNF, monocyte-derived
, tumor necrosis factor ligand superfamily member 2
, tumor necrosis factor-alpha
, tumor necrosis factor (TNF superfamily, member 2)
, tumor necrosis factor alpha
, tumor necrosis factor
, tumor-necrosis factor
, tumour necrosis factor
, TNF alpha
, ATP-binding cassette, sub-family F (GCN20), member 1
, tumour necrosis factor alpha
, putative tumour necrosis factor alpha
, LPS-induced TNF-alpha factor homolog
, TNF-alpha factor
, lipid-induced TNF-alpha factor
, lipopolysaccharide-induced tumor necrosis factor-alpha factor homolog
, small integral membrane protein of lysosome/late endosome
, tissue necrosis factor-alpha
, tumor necrosis factor, alpha
, Tumor necrosis factor ligand superfamily member 2
, TNF-alpha 1