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Human BAX Protein expressed in Wheat germ - ABIN1346448
Bras, Yuste, Roué, Barbier, Sancho, Virely, Rubio, Baudet, Esquerda, Merle-Béral, Sarfati, Susin: Drp1 mediates caspase-independent type III cell death in normal and leukemic cells. in Molecular and cellular biology 2007
These results suggest a role for mitochondrial p53 (show TP53 Proteins) activity in promoting hair cell death due to aminoglycosides, likely upstream of Bax and Bcl2 (show BCL2 Proteins).
xlbax is a regulator of muscle fiber death in the regressing tail during metamorphosis.
BoHV-5 replication apparently modulates BCL-2 (show BCL2 Proteins) expression and gene transcription, enhancing production of virus progeny, but does not increase Bax expression.
It was concluded that the Fas (show FAS Proteins)-FasL (show FASL Proteins) signaling pathway was involved in regulation of bovine oocyte apoptosis, perhaps related to B (show TDO2 Proteins) cell lymphoma/leukemia-2 associated X upregulation.
apoptosis-inducing factor (show AIFM1 Proteins) was expressed in luminal alveolar cells and, in concert with a change in bax protein to bcl-2 (show BCL2 Proteins) protein ratio, might contribute to signalling of a change in the dynamic balance of the cell population as lactation progresses
mRNA expression of Bax, Bcl-2 (show BCL2 Proteins), caspase-3 (show CASP3 Proteins) and-7 cannot be used as a reliable apoptosis detection method.
The effect of culture conditions on expression of heat shock protein 70 (show HSP70 Proteins) and Bax protein in bovine blastocysts is reported.
Findings reveal a novel mechanism by which p53 (show TP53 Proteins) utilizes TFIIS.h (show TCEA3 Proteins) to selectively promote the transcriptional elongation of the bax gene, upsurging cell death in response to severe DNA damage.
elevated MMP-2 (show MMP2 Proteins) expression and disturbance balance of Bcl-2 (show BCL2 Proteins)/BAX expressions may be associated with the development and maintenance of atrial fibrillation.
TG2 (show TGM2 Proteins) can inhibit tumor cell apoptosis through down-regulation of Bax and prevention of release Cyt C (show CYCS Proteins) from mitochondria into cytoplasm
Bcl-2 (show BCL2 Proteins) and Bax expression was significantly associated with histologic grade and clinical stage, which are classic factors of poor prognosis. We suggest the use of these proteins as potential prognostic markers in STS (show STS Proteins) of extremities
The aim of this study was to determine the expression of apoptotic factors Bax, Bcl-2 (show BCL2 Proteins), and Caspase-3 (show CASP3 Proteins) in lens epithelial cells (LECs) from cataracts secondary to pars (show EPRS Proteins) plana vitrectomy with silicone oil (SO) tamponade.
The expression of PHF20 was associated with Bax expression.
Deletion of chromosomal region 19q13.1-13.4 is common in hereditary non-polyposis colorectal cancer (CRC (show CALR Proteins)). This deletion could be the cause of the reduction in the expression of the BAX gene observed in CRC (show CALR Proteins).
miR (show MLXIP Proteins)-449 regulates the SIRT1 (show SIRT1 Proteins)/p53 (show TP53 Proteins)/BAX pathway, which may be its possible mechanism in modulating cell apoptosis of cisplatin-induced Acute kidney injury.
increased platelet apoptosis and activation as well as reduced expression of Bcl-xL (show BCL2L1 Proteins), increased expression of Bax and caspase-3 (show CASP3 Proteins) activity were found in platelets after treated with ITP (show ITPA Proteins) plasma in comparison with control plasma.
Polymorphism of BAX and TP53 (show TP53 Proteins) genes may be potential genetic modifiers for developing ovarian cancer.
These results suggest that Bax mediates beta-cell apoptosis in Pdx1 (show PDX1 Proteins)-deficient diabetes.
Bim (show BCL2L11 Proteins) and Puma (show BBC3 Proteins) overlapped apoptosis only partially during physiological apoptotic stage and they were present in non-apoptotic parts of the follicles.
This study demonstated that Bax KO mice show Hyperactivity and depression.
The influence of chronic ethanol consumption on the expression of the Bdnf (show BDNF Proteins), Bax, Bcl-xL (show BCL2L1 Proteins), and CASP3 (show CASP3 Proteins) genes was studied in the brain structures of B6-1473C (C/C) and B6-1473G (G/G) mice that had been obtained on the base of the C57BL/6 strain.
These data indicate that the rapid and robust microglial activation and PIF (show DCD Proteins) expression observed in situ following acute EtOH exposure is largely driven indirectly by BAX-dependent apoptotic cell death, rather than directly by the EtOH.
lipid profile in the absence of the proapoptotic proteins BAX and BAK (show BAK1 Proteins) in mouse embryonic fibroblasts
despite its role in Bax inhibition, Bcl-xL (show BCL2L1 Proteins) also primes mitochondria to permeabilization and cytochrome c (show CYCS Proteins) release.
BAX and BAK (show BAK1 Proteins) have a nonapoptotic role in eicosanoid metabolism in inflammation.
Puma (show BBC3 Proteins) is the major mediator of virus-induced Bax/Bak (show BAK1 Proteins) activation and mitochondrial membrane permeabilization induction.
The Ca(2 (show CA2 Proteins)+) channel inhibitor 2-APB (show RNPEP Proteins) reverses beta-amyloid-induced long term potentiation deficit in hippocampus by blocking BAX and caspase-3 (show CASP3 Proteins) hyperactivation.
Bid (show BID Proteins) and Bax are signal transduction factors in granulosa cells and play proapoptotic roles.
The data demonstrate that severe hypocapnia results in increased Bax expression, DNA fragmentation, and membrane lipid peroxidation in mitochondria of cerebral cortical neurons of newborn piglets, and may result in apoptotic cell death.
bcl-2 (show BCL2 Proteins) and bax were expressed strongly in denervated guinea-pig facial muscle. [bcl-2 (show BCL2 Proteins); bax]
goat oocytes based on G6PDH (show G6PD Proteins)-activity through the BCB test improves their developmental competence, increases intracellular GSH content, and affects the expression of the apoptosis-related genes9 Bax and Bcl-2 (show BCL2 Proteins) ).
These results indicate that RI-PostC can ameliorate myocardial ischemia-reperfusion injury and increase the Bcl-2/Bax ratio through a mechanism involving protein kinase C.
Cinobufagin can remarkably inhibit the proliferation and induce the apoptosis of lens epithelial cells by increasing expression of bax and decreasing expression of bcl2 (show BCL2 Proteins).
Elevated local temperature of the testis buried in the inguinal pocket increases the apoptosis of spermatogenic cells, and the spermatogenic cell apoptosis is highly correlated with the decreased expression of Bcl-2 (show BCL2 Proteins) and increased expression of Bax.
In this study evidence is provided that exogenous PGF2alpha differentially modulates luteal expression of BCL2-associated X protein (BAX) transcripts and protein depending on luteal stage.
The protein encoded by this gene belongs to the BCL2 protein family. BCL2 family members form hetero- or homodimers and act as anti- or pro-apoptotic regulators that are involved in a wide variety of cellular activities. This protein forms a heterodimer with BCL2, and functions as an apoptotic activator. This protein is reported to interact with, and increase the opening of, the mitochondrial voltage-dependent anion channel (VDAC), which leads to the loss in membrane potential and the release of cytochrome c. The expression of this gene is regulated by the tumor suppressor P53 and has been shown to be involved in P53-mediated apoptosis. Multiple alternatively spliced transcript variants, which encode different isoforms, have been reported for this gene.
, bax zeta
, apoptosis regulator BAX
, BCL2-associated X protein
, BCL2-associated X protein omega
, BCL2-associated X protein transcript variant delta2
, bcl-2-like protein 4
, BCL2-associated protein