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anti-Human TRIM29 Antibodies:
anti-Mouse (Murine) TRIM29 Antibodies:
anti-Rat (Rattus) TRIM29 Antibodies:
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Human Polyclonal TRIM29 Primary Antibody for IHC (fro), ELISA - ABIN537436
Katkoori, Shanmugam, Jia, Vitta, Sthanam, Callens, Messiaen, Chen, Zhang, Bumpers, Samuel, Manne: Prognostic significance and gene expression profiles of p53 mutations in microsatellite-stable stage III colorectal adenocarcinomas. in PLoS ONE 2012
Human Polyclonal TRIM29 Primary Antibody for ELISA, IHC - ABIN4362410
Kosaka, Inoue, Ohmachi, Yokoe, Matsumoto, Mimori, Tanaka, Watanabe, Mori: Tripartite motif-containing 29 (TRIM29) is a novel marker for lymph node metastasis in gastric cancer. in Annals of surgical oncology 2007
Human Polyclonal TRIM29 Primary Antibody for ELISA, IHC - ABIN4362411
Wang, Heidt, Lee, Yang, Logsdon, Zhang, Fearon, Ljungman, Simeone: Oncogenic function of ATDC in pancreatic cancer through Wnt pathway activation and beta-catenin stabilization. in Cancer cell 2009
NOTCH1 (show NOTCH1 Antibodies) inhibits activation of ATM (show ATM Antibodies) by impairing the formation of an ATM (show ATM Antibodies)-FOXO3a (show FOXO3 Antibodies)-KAT5 (show KAT5 Antibodies) complex.
No strong correlation was observed between ATM (show ATM Antibodies) mutation and function. Therefore, mutation status may not be taken as an indicator of ATM (show ATM Antibodies) function. Rather, a direct assay of the kinase activity should be used in the development of therapies.
Results show that ATM (show ATM Antibodies) protein expression is lost in 31% of patients with breast cancer. ATM (show ATM Antibodies) loss is frequently observed in a distinct group with more advanced-stage disease.
ATM (show ATM Antibodies) mutation impacts on the age-related telomere length shortening and is not related to cancer risk.
although recruitment of the MRE11 (show MRE11A Antibodies)-RAD50 (show RAD50 Antibodies)-NBS1 (show NBN Antibodies) (MRN) DSB-sensing complex to viral genomes and activation of the ATM (show ATM Antibodies) kinase can promote KSHV replication, proteins involved in nonhomologous end joining (NHEJ) repair restrict amplification of viral DNA.
A high frequency of chromothriptic events occurred in cases of acute lymphoblastic anemia arising in patients with ataxia telangectasia, specifically on acrocentric chromosomes, as compared with tumors from individuals with other types of DNA repair syndromes, due to the short telomeres and poor DNA repair caused by their two ATM (show ATM Antibodies) mutations. ATM (show ATM Antibodies) loss in other tumors also increases chromothripsis.
ASF1a (show ASF1A Antibodies) promotes non-homologous end joining repair by facilitating phosphorylation of MDC1 (show MDC1 Antibodies) by ATM (show ATM Antibodies) at double-strand breaks.
ectopic expression of Gene 33 triggers DNA damage response in an ATM serine/threonine kinase (ATM)-dependent fashion and through pathways dependent or not dependent on ABL proto-oncogene 1 non-receptor tyrosine kinase (c-Abl).
We demonstrate that, in breast cancer cells, ATM (show ATM Antibodies) and ATG4C (show ATG4C Antibodies) are essential drivers of mammosphere formation, suggesting that their targeting may improve current approaches to eradicate breast cancer cells with a stem-like phenotype.
ATM (show ATM Antibodies)-reactive oxygen species-iNOS (show NOS2 Antibodies) axis regulates nitric oxide mediated cellular senescence.
this study shows that deletion of TRIM29 enhanced macrophage production of type I interferons and protected mice from infection with influenza virus, while challenge of Trim29-/- mice with Haemophilus influenzae resulted in lethal lung inflammation due to massive production of proinflammatory cytokines by macrophages
Findings established a role for ATDC/TRIM29 as a robust pathogenic driver of bladder cancer development, identified downstream effector pathways, and implicated ATDC as a candidate biomarker and therapeutic target.
ATDC up-regulates CD44 (show CD44 Antibodies) in mouse and human PanIN lesions via activation of beta-catenin (show CTNNB1 Antibodies) signaling, leading to the induction of an epithelial-to-mesenchymal transition (EMT (show ITK Antibodies)) phenotype characterized by expression of Zeb1 (show ZEB1 Antibodies) and Snail1 (show SNAI1 Antibodies).
Histone deacetylase 9 (HDAC9 (show HDAC9 Antibodies)) regulates the functions of the ATDC (TRIM29) protein
ATDC increases cell proliferation via inhibition of p53 (show TP53 Antibodies) nuclear activities.
The protein encoded by this gene belongs to the TRIM protein family. It has multiple zinc finger motifs and a leucine zipper motif. It has been proposed to form homo- or heterodimers which are involved in nucleic acid binding. Thus, it may act as a transcriptional regulatory factor involved in carcinogenesis and/or differentiation. It may also function in the suppression of radiosensitivity since it is associated with ataxia telangiectasia phenotype.
ataxia telangiectasia group D-associated protein
, ataxia-telangiectasia group D-associated protein
, tripartite motif protein TRIM29
, tripartite motif-containing 29
, tripartite motif-containing protein 29
, tripartite motif protein 29
, tripartite motif-containing protein 29-like