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Human KLF4 ELISA Kit for Sandwich ELISA - ABIN416925
Seipel, Marques, Bozzini, Meinken, Mueller, Pabst: Inactivation of the p53-KLF4-CEBPA Axis in Acute Myeloid Leukemia. in Clinical cancer research : an official journal of the American Association for Cancer Research 2016
zebrafih Klf4a is essential for the repression of intestinal cell proliferation
Proper heart valve formation in zebrafish critically depends on protein kinase D2 (show PKD2 ELISA Kits)-histone deacetylase 5 (show HDAC5 ELISA Kits)-Kruppel-like factor signaling.
we demonstrate that human papillomavirus proteins regulate the expression and activities of a critical cellular transcription factor, KLF4, through post-transcriptional and post-translational mechanisms
KLF4 expression mediated by lentiviral vector or induced by APTO-253 resulted in G1 phase arrest in ovarian cancer cells. Our results demonstrate that for the first time that inducing KLF4 expression with APTO-253 is a novel therapeutic strategy for treating ovarian cancer.
forced expression of KLF4 in plasmablasts enhances the yield of their differentiation into early plasma cell and long lived plasma cells, by inhibiting apoptosis and upregulating previously unknown plasma cell pathways.
BMP4 (show BMP4 ELISA Kits) promotes a phenotype change of an esophageal squamous epithelium via up-regulation of KLF4.
The results suggest that Kruppel-like factor 4 may act as a transcription factor mediating the activation of fibroblast-like synoviocytes in RA by inducing interleukin-6 (show IL6 ELISA Kits) expression in response to tumor necrosis factor-alpha (show TNF ELISA Kits).
Tumor suppressive role of KLF-4 in human endometrial carcinoma tissues.
Osteosarcoma spheres are slowly-proliferating populations, overexpress SOX2 (show SOX2 ELISA Kits), and KLF4 stemness-related genes and have enhanced tumorigenic potential.
KLF4 has been gaining a lot of attention, particularly after it was shown in 2006 as one of four factors involved in the induced pluripotent stem cells (iPSCs).
High KLF5 (show KLF5 ELISA Kits) expression is associated with glioma.
Klf4 directly regulates miR31 in hepatocellular carcinoma.
Klf4 is transcribed both maternally and zygotically and the transcript is ubiquitous in embryos during germ-layer formation
Induction of the transcription factor Klf4 in differentiated smooth muscle cells is essential for smooth muscle cell reprogramming.
KLF4 expression is negatively correlated to the Notch (show NOTCH1 ELISA Kits) pathway in hepatic stellate cells.
Data show that the reprogramming factors OCT4 (show POU5F1 ELISA Kits), SOX2 (show SOX2 ELISA Kits), KLF4, and MYC (show MYC ELISA Kits) (OSKM) drive cells along two distinct and parallel pathways, one pluripotent and one endodermal.
Study demonstrates that KLF4 plays a significant role in the pathogenesis of colorectal neoplasia.
our study provides valuable information on the molecular interactions between OSKM(Oct4 (show POU5F1 ELISA Kits), Sox2 (show SOX2 ELISA Kits), Klf4, and Myc (show MYC ELISA Kits)) and cofactors and molecular mechanisms for the functional importance of Set1a (show SETD1A ELISA Kits) in ESCs (show NR2E3 ELISA Kits) and early development
Data show that R-Smad Proteins SMAD1 and SMAD5, which transduce bone morphogenetic protein (BMP) signals, recognize enhancer regions together with Kruppel-like factors KLF4 and KLF5 in naive embryonic stem cell (mESCs).
Results suggest that Kruppel-like factor 4 is not required for retinal cell differentiation or survival. Data support a hypothesis that KLF4 suppresses axon growth during development.
KLF4 is a master regulator of EndMT in CCM pathology; thus, the inhibition of Klf4 expression may have a therapeutic role in limiting CCM appearance and progression.
Authors show that absence of Klf4 accelerates carcinogenesis and correlates with the absence of cytokeratin 1 (show KRT1 ELISA Kits) expression. Results suggest a potential role for KLF4 as a tumor suppressor gene for the tongue epithelium.
KLF-4 was downregulated in the TGF-beta (show TGFB1 ELISA Kits)-induced EMT (show ITK ELISA Kits) model in LA-4 cells. KLF-4 overexpression markedly reduced the PAI-1 (show SERPINE1 ELISA Kits) expression and inhibited the TGF-beta (show TGFB1 ELISA Kits)-induced EMT (show ITK ELISA Kits) .
miR (show MYLIP ELISA Kits)-92a coregulates KLF4 and KLF2 (show KLF2 ELISA Kits) expression in arterial endothelium and contributes to phenotype heterogeneity associated with regional atherosusceptibility and protection in vivo.
a transcription factor that works with Sp1 to activate the Laminin gamma1 chain gene
Krueppel-like factor 4
, Kruppel-like transcription factor 4a
, Kruppel-like factor 4 (gut)
, Kruppel-like factor 4
, endothelial Kruppel-like zinc finger protein
, epithelial zinc finger protein EZF
, gut-enriched krueppel-like factor
, Kruppel-like transcription factor
, blood island enriched kruppel like factor
, Kruppel-like factor 4 (Gut enriched kruppel-like factor) (Epithelial zinc-finger protein EZF)