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anti-Mouse (Murine) Antibodies:
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Low aortic mitochondria-encoded COX1 (show COX1 Antibodies) in obese diabetic double knock-out mice was associated with a larger plaque area and higher M1 macrophages and oxidized LDL. Caloric restriction increased COX1 (show COX1 Antibodies) and reduced plaque area and oxidized LDL. Low COX1 (show COX1 Antibodies) related to low expression of PPAR alpha (show PPARA Antibodies), delta, and gamma and of peroxisome proliferative activated receptor, gamma, co-activator 1 alpha reflecting mitochondrial dysfunction.
a dependence of COX1 (show COX1 Antibodies) AND capital ES, CyrillicOX2 expression on the presence of MMP-9 (show MMP9 Antibodies) was shown in neutrophil infiltration during inflammation.
CCOI expression is inhibited by osteopontin (show SPP1 Antibodies) as the result of a novel CD44 (show CD44 Antibodies)-dependent transcriptional regulatory mechanism of the mitochondrial H strand
The gene expression of Cox1 (show COX1 Antibodies) in adipocytes was studied.
CcOX (show COX5A Antibodies) I protein levels significantly decreased following CO exposure while enzyme turnover number and CcOX (show COX5A Antibodies) I mRNA levels remained unchanged.
IOP elevation may directly damage mitochondria in the ONH axons by promoting reduction of COX (show CPOX Antibodies), mitochondrial fission and cristae depletion, alterations of OPA1 (show MED12 Antibodies) and Dnm1 (show DNM1 Antibodies) expression, and induction of OPA1 (show MED12 Antibodies) release.
Tumor necrosis factor alpha (show TNF Antibodies) inhibits oxidative phosphorylation through tyrosine phosphorylation at subunit I of cytochrome c (show CYCS Antibodies) oxidase
Catalyzes the ATP-dependent amination of UTP to CTP with either L-glutamine or ammonia as the source of nitrogen (By similarity).
, CTP synthase 1-like
, CTP synthetase
, CTP synthase 1-B
, CTP synthetase 1-B
, UTP--ammonia ligase 1-B
, cytidine 5'-triphosphate synthase