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Human Polyclonal LHCGR Primary Antibody for ELISA, WB - ABIN4229674
Yung, Maman, Ophir, Rubinstein, Barzilay, Yerushalmi, Hourvitz: Progesterone antagonist, RU486, represses LHCGR expression and LH/hCG signaling in cultured luteinized human mural granulosa cells. in Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology 2013
Show all 4 Pubmed References
Human Polyclonal LHCGR Primary Antibody for IHC (p) - ABIN270629
Yung, Aviel-Ronen, Maman, Rubinstein, Avivi, Orvieto, Hourvitz: Localization of luteinizing hormone receptor protein in the human ovary. in Molecular human reproduction 2014
Show all 3 Pubmed References
Rat (Rattus) Polyclonal LHCGR Primary Antibody for WB - ABIN336206
Berndt, Perrier dHauterive, Blacher, Péqueux, Lorquet, Munaut, Applanat, Hervé, Lamandé, Corvol, van den Brûle, Frankenne, Poutanen, Huhtaniemi, Geenen, Noël, Foidart: Angiogenic activity of human chorionic gonadotropin through LH receptor activation on endothelial and epithelial cells of the endometrium. in FASEB journal : official publication of the Federation of American Societies for Experimental Biology 2006
Human Polyclonal LHCGR Primary Antibody for IF (p), IHC (p) - ABIN873328
Yang, Dou, Zhang, Gu, Lv, DU, Ba, Mu, Lu: Increased 3β-hydroxysteroid dehydrogenase 2 and 17α-hydroxylase activities in a virilized adolescent female with adrenal adenoma: A case report. in Experimental and therapeutic medicine 2016
Human Monoclonal LHCGR Primary Antibody for FACS, IF - ABIN5582545
Pidoux, Gerbaud, Tsatsaris, Marpeau, Ferreira, Meduri, Guibourdenche, Badet, Evain-Brion, Frendo: Biochemical characterization and modulation of LH/CG-receptor during human trophoblast differentiation. in Journal of cellular physiology 2007
Data show that double mutation of follicle-stimulating hormone receptor (fshr (show FSHR Antibodies)) and luteinizing hormone receptor (lhcgr) resulted in infertile males
Data show for the first time in a vertebrate species that Leydig cells as well as Sertoli cells express the mRNAs for both fshr (show FSHR Antibodies) and lhcgr.
suggest that upregulation of NG2/CSPG4 (show MCSP Antibodies) rather than changes in CD44 (show CD44 Antibodies) or Ki-67 (show MKI67 Antibodies) expression is associated with low overall survival in glioblastoma multiforme patients, supporting NG2/CSPG4 (show MCSP Antibodies) as a potential prognostic marker in glioblastoma
Suggest that CD44 (show CD44 Antibodies)-positivity might be a candidate tumor stem cell marker in intrahepatic cholangiocarcinoma and a prognostic indicator.
Mechanistically, CD44v interacts with and stabilizes xCT (show SLC7A11 Antibodies) and thereby promotes the uptake of cysteine for glutathione synthesis and stimulates side-population cell enrichment.
These tumor samples express CD44 (show CD44 Antibodies) protein at low rather than high levels. There is no correlation between CLDN3 (show CLDN3 Antibodies) gene expression and protein expression in these CPTAC samples; hence, the claudin-low subtype defined by gene expression is not the same group of tumors as that defined by low expression of CLDN3 (show CLDN3 Antibodies) protein.
this study shows that genkwadaphnin promotes leukocyte migration by increasing CD44 (show CD44 Antibodies) expression via PKD1 (show PKD1 Antibodies)/NF-kappaB (show NFKB1 Antibodies) signaling pathway
High CD44 (show CD44 Antibodies) expression is associated with high grade glioma.
CNS disease was associated with enhanced expression of cytoplasmic and membranous ITGA10 and nuclear PTEN (show PTEN Antibodies) (P < 0.0005, P = 0.002, P = 0.024, respectively). sCNSL presented decreased membranous CD44 (show CD44 Antibodies) and nuclear and cytoplasmic cadherin-11 expressions (P = 0.001, P = 0.006, P = 0.048, respectively). In PCNSL lactoferrin (show LTF Antibodies) expression was upregulated (P < 0.0005).
In clinical specimens of breast cancer, authors established that the expression of CD44s and HAS2 (show HAS2 Antibodies) was positively correlated. Results establish a positive feedback mechanism that sustains PI3K (show PIK3CA Antibodies)/Akt (show AKT1 Antibodies) signaling in tumor cells, further illuminating the nearly universal role of this pathway in cancer cell survival.
In regulation of tumour cells with high CD44 (show CD44 Antibodies) expression.
ZEB1 (show ZEB1 Antibodies)-induced epithelial-to-mesenchymal transition and associated molecular changes in ESRP1 (show ESRP1 Antibodies) and CD44 (show CD44 Antibodies) contribute to early pathogenesis and metastatic potential in established lung cancer
Study tested for the first time a role of ZFP36L2 (show ZFP36L2 Antibodies) in the decay of LHR mRNA, when transcription was inhibited; results of our cell-based assay support the conclusion that LHR mRNA expression is controlled post-transcriptionally by ZFP36L2 (show ZFP36L2 Antibodies).
FSHR (show FSHR Antibodies) and LHR proteins are significantly upregulated in CCs (show CCS Antibodies) surrounding oocytes arrested at the 2-cell stage, reflecting their developmental incompetence.
Triptorelin and cetrorelix induce immune responses and affect uterine development and expressions of genes and proteins of ESR1 (show ESR1 Antibodies), LHR, and FSHR (show FSHR Antibodies)
Data suggest that persistent cAMP signals from internalized luteinizing hormone receptor (LH receptors) contribute to transmitting LH effects inside follicle cells and ultimately to the oocyte.
Demonstrate the presence of LH receptors. Activation resulted in a dose-dependent increase in glucose-induced release of insulin (show INS Antibodies).
LHCGR signaling in regulating the Ahr (show AHR Antibodies) message involves protein kinase A pathway and is attributable to decreased transcription rate.
Data from mutant mouse strain (gain-of-function mutation in LHR, D578G; most common mutation found in familial male-limited precocious puberty) confirm that LHR is critical for male steroidogenesis, gametogenesis, and Leydig cell development.
LH/hCG (show CGA Antibodies) tightly up-regulates MKP-3 (show DUSP6 Antibodies) which in turn, dephosphorylates ERK1/2 (show MAPK1/3 Antibodies) and drives p21 (show D4S234E Antibodies) expression.
Data suggest that Lhcgr in endometrium and luteinizing hormone in blastocyst are involved in embryo/blastocyst implantation; expression of Lhcgr is up-regulated in endometrial epithelium in estrus cycle at time of implantation readiness (estrus).
Through the LHR, LH/hCG (show CGA Antibodies) tightly regulates MKP-2 (show DUSP4 Antibodies) expression, which modulates the induction of CYP11A1 (show CYP11A1 Antibodies) by 8Br-cAMP.
The outcomes of the present study support a dynamic multi-facetted regulation of LHR during pre-translation.
expression of LHR mRNA in bovine granulosa cells is established after follicle deviation, and the lower abundance of LRBP (show MVK Antibodies) mRNA after the expected time of deviation may contribute to greater expression of LHR in the bovine dominant follicle
These results suggested an acute regulation of INSL3 (show INSL3 Antibodies) by luteinizing hormone (LH) because INSL3 (show INSL3 Antibodies) concentrations increased immediately after endogenous and exogenous LH stimulation.
INVESTIGATION OF STAT5A (show STAT5A Antibodies), FSHR (show FSHR Antibodies) AND LHR GENE POLYMORPHISMS IN TURKISH INDIGENOUS CATTLE BREEDS
These findings strongly support the concept that IGF-1 (show IGF1 Antibodies) upregulates LHR expression in granulosa cells and that IGF-1 (show IGF1 Antibodies) is required for determining which follicle becomes dominant and acquires ovulatory capacity.
LHCGR mRNA expression in granulosa cells was significantly higher in large antral follicles than in cysts, and not detected in granulosa cells of small and medium antral follicles.
The luteinizing hormone receptor [LHR] splicing pattern is complex in bovine Leydig cells, and expression of full-length LHR and isoforms A and B changes when induced with LH.
The LHCGR gene is a potential marker for superovulation response and can be used to predict the most appropriate dose of FSH (show BRD2 Antibodies) for superovulation in Chinese Holstein cows.
Dominant follicles experience a reduction in FSH (show BRD2 Antibodies) dependence (diminished expression of FSHR (show FSHR Antibodies)), but acquire increased LH dependence (enhanced expression of LHCGR) as they grow during the low FSH (show BRD2 Antibodies) milieu of follicular waves.
Three single nucleotide polymorphisms in LHCGR were significantly associated with variations in cattle fertility and production traits.
In porcine ovaries, MAb found 6 distinct LHR bands migrating at approximately 92, 80, 68, 59, 52 & 48 kDa. There is a possible role for LHR in the development of abnormal pregnancy, pelvic floor disorders & Alzheimer's disease.
This gene encodes the receptor for both luteinizing hormone and choriogonadotropin. This receptor belongs to the G-protein coupled receptor 1 family, and its activity is mediated by G proteins which activate adenylate cyclase. Mutations in this gene result in disorders of male secondary sexual character development, including familial male precocious puberty, also known as testotoxicosis, hypogonadotropic hypogonadism, Leydig cell adenoma with precocious puberty, and male pseudohermaphtoditism with Leydig cell hypoplasia.
luteinizing hormone receptor
, lutropin-choriogonadotropic hormone receptor
, luteinizing hormone/choriogonadotropin receptor
, lutropin-choriogonadotropic hormone receptor-like
, Lutropin-choriogonadotropic hormone receptor
, CD44 antigen
, GP90 lymphocyte homing/adhesion receptor
, Hermes antigen
, cell surface glycoprotein CD44
, chondroitin sulfate proteoglycan 8
, extracellular matrix receptor III
, hematopoietic cell E- and L-selectin ligand
, heparan sulfate proteoglycan
, homing function and Indian blood group system
, hyaluronate receptor
, phagocytic glycoprotein 1
, hypergonadotropic hypogonadism
, lutropin/choriogonadotropin receptor
, LH receptor
, luteinizing hormone receptor 2 protein
, luteinizing hormone receptor precursor variant 1
, luteinizing hormone receptor precursor variant 2
, lutropin-choriogonadotropin receptor