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Increased CD11b+ and CD200+ monocytes in those with a strong presence of BVN indicate that BVN development may be associated with retinal injury and a VEGF-mediated process that is either reflected or propelled by systemic changes in monocytes.
CD200 (show CD200 Proteins) expression therefore may be used to help identify pulmonary small cell carcinoma in flow cytometry specimens.
PAK4 (show PAK4 Proteins) downregulation decreased PPARgamma (show PPARG Proteins)-mediated Nox1 expression and suppressed EMT (show ITK Proteins) in IR-treated glioma cells.
Nox1-SHH (show SHH Proteins)-Grem1 (show GREM1 Proteins) signaling axis in pulmonary vascular endothelium that is likely to contribute to Pulmonary hypertension.
Our data suggest a negative impact of CD200 (show CD200 Proteins) expression in cytogenetically-normal acute myeloid leukemia (show BCL11A Proteins)
CD200 (show CD200 Proteins) was expressed in 87% of the neuroendocrine neoplasms studied. CD200 (show CD200 Proteins) is a relatively sensitive marker of neuroendocrine neoplasms and represents a potential therapeutic target in these difficult-to-treat malignancies.
Up-regulation of CD200 (show CD200 Proteins) in MDS (show PAFAH1B1 Proteins) predicts poor prognosis.
this study shows that that CD200 (show CD200 Proteins) suppresses the immune system's response to vaccines, and that blocking CD200 (show CD200 Proteins) could improve the efficacy of cancer immunotherapy
5-HT1B receptor-dependent cellular Src (show SRC Proteins)-related kinase-Nox1-pathways contribute to vascular remodeling in pulmonary arterial hypertension.
NOX1 has a role in maintaining the proliferative phenotype of some colon cancers and has potential as a therapeutic target in this disease
The nox1 expression in zebrafish during early nervous system development from 12 to 48 hours post fertilization.
Over inhibition of the NADPH oxidase by the NADPH Inhibitor DPI may reduce the cell even the tissue in the progress of healing after the injury, in zebrafish liver cells.
NADPH oxidase plays an important role in proMMP-2 expression and activation and MMP-2 (show MMP2 Proteins) mediated SMC (show DYM Proteins) proliferation occurs through the involvement of Spm (show NPC1 Proteins)-Cer (show CBLN1 Proteins)-S1P (show MBTPS1 Proteins) signaling axis under ANG II (show AGT Proteins) stimulation of PASMCs
Differential Roles of Protein Complexes NOX1-NOXO1 and NOX2-p47phox in Mediating Endothelial Redox Responses to Oscillatory and Unidirectional Laminar Shear Stress.
The current study was designed to determine mechanisms underlying 20-hydroxyeicosatetraenoic acid -stimulated nitric oxide (NO) release, and particularly the role of NADPH oxidase, reactive oxygen species, and PI3-kinase (show PIK3CA Proteins) in stimulated NO release.
NEP (show MME Proteins) expression is down-regulated in vascular endothelial cells by physiological laminar shear, possibly via a mechanotransduction mechanism involving NADPH oxidase-induced reactive oxygen species production.
C-kit-positive hematopoietic stem/progenitor cells expressed significantly higher of Nox1 and catalase (show CAT Proteins), but less of lactoperoxidase (show LPO Proteins) than in matured mononuclear cells.
Nox1 deletion reduces oxidant load and restores microvascular health in obese mice.
Data suggests that ROS (show ROS1 Proteins) produced during primitive endoderm differentiation is dependent in part on increased NOX1 and NOX4 (show NOX4 Proteins) levels, which is under the control of GATA6 (show GATA6 Proteins). Furthermore, these results suggest that the combined activity of multiple NOX proteins is necessary for the differentiation of F9 cells to primitive endoderm.
Gp91phox (show CYBB Proteins) NADPH oxidase modulates litter size by up-regulating mucin1 (show MUC1 Proteins) expression in the uterus of mice.
NOX4 (show NOX4 Proteins)- and NOX1-derived ROS (show ROS1 Proteins) contribute to atherosclerosis in the aortic sinus of diabetic ApoE (show APOE Proteins) knockout mice.
Data show that pan-NOX-inhibitor APX-115 treatment decreased NADPH oxidase (Nox) Nox1, Nox2 (show CYBB Proteins), and Nox4 (show NOX4 Proteins) protein expression in the kidney.
NOX1 is selectively important in GPCR (show GPBAR1 Proteins)-dependent intracellular ROS (show ROS1 Proteins) generation but dispensable for GPVI (show GP6 Proteins)-ITAM-dependent ROS (show ROS1 Proteins) generation in blood platelets..
Plasmid driven elevation of NOX-1 resulted in elevated ROSreactive oxygen species, loss of glucose-stimulated-insulin (show INS Proteins)-secretion and increased apoptosis. These outcomes on beta cell function are analogous to cytokine treatment.
Nox1-knockout (Nox1-KO) mice were not more sensitive to S. Tm colonization and infection than WT B6 mice.
This gene encodes a member of the NADPH oxidase family of enzymes responsible for the catalytic one-electron transfer of oxygen to generate superoxide or hydrogen peroxide. Alternatively spliced transcript variants encoding multiple isoforms have been observed for this gene.
, MRC OX-2 antigen
, OX-2 membrane glycoprotein
, antigen identified by monoclonal antibody MRC OX-2
, NADPH oxidase-1
, predicted NADPH oxidase-1
, NADH/NADPH mitogenic oxidase subunit P65-MOX
, NADPH oxidase homolog-1
, mitogenic oxidase (pyridine nucleotide-dependent superoxide-generating)
, mitogenic oxidase 1
, GP91 phox homolog
, NADH/NADPH mitogenic oxidase subunit p65-mox
, NADPH oxidase 1 alpha