Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species
Human FLT4 ELISA Kit for Sandwich ELISA - ABIN625371
Navid, Baker, McCarville, Stewart, Billups, Wu, Davidoff, Spunt, Furman, McGregor, Hu, Panetta, Turner, Fofana, Reddick, Leung, Santana: Phase I and clinical pharmacology study of bevacizumab, sorafenib, and low-dose cyclophosphamide in children and young adults with refractory/recurrent solid tumors. in Clinical cancer research : an official journal of the American Association for Cancer Research 2013
Show all 5 Pubmed References
Mouse (Murine) FLT4 ELISA Kit for Sandwich ELISA - ABIN2859280
Li, Fan, Song, Zhang, Chen, Li, Mi, Ma, Song, Tao, Li: Expression of angiopoietin-2 and vascular endothelial growth factor receptor-3 correlates with lymphangiogenesis and angiogenesis and affects survival of oral squamous cell carcinoma. in PLoS ONE 2013
Human FLT4 ELISA Kit for Sandwich ELISA - ABIN414865
Zhao, Geng, Hua, Cun, Chen, Xi, Yang, Li: Fenofibrate inhibits the expression of VEGFC and VEGFR-3 in retinal pigmental epithelial cells exposed to hypoxia. in Experimental and therapeutic medicine 2015
Lymphangiogenic growth in the diaphragm was highly dependent on vascular endothelial growth factor receptor (show RYK ELISA Kits) (VEGFR)-3 signaling. During diaphragm development, macrophages appeared first in a linearly arranged pattern, followed by ingrowth of lymphatic vessels along these patterned lines.
found that WT1 (show WT1 ELISA Kits) and KDR (show KDR ELISA Kits) are co-expressed in Sertoli cells of the testes and somatic cells of embryonic ovaries. Furthermore, WT1 (show WT1 ELISA Kits) bound to the Kdr (show KDR ELISA Kits) promoter in the chromatin of embryonic testes and ovaries. KDR (show KDR ELISA Kits) signaling represses the testis-promoting gene Sox9 (show SOX9 ELISA Kits) in embryonic XX gonads
This is the first report demonstrating the spatiotemporal expression patterns of Flk1 (show KDR ELISA Kits) and Flt1 (show FLT1 ELISA Kits) in the coronary vascular system during development and after MI; thus, this study suggests that these factors have distinct and important functions in coronary angiogenesis.
VEGFR2 (show KDR ELISA Kits)-associated alpha(2,6)-linked sialic acid plays an important role in modulating VEGF (show VEGFA ELISA Kits)/VEGFR2 (show KDR ELISA Kits) interaction, EC pro-angiogenic activation and neovessel formation.
These results revealed that vascular sprouting and permeability are both controlled through the VEGFR2-TSAd-c-Src signaling pathway in a subset of tissues, which may be useful in developing strategies to control tissue-specific pathological angiogenesis.
Data show that editing of genomic VEGFR2 (show KDR ELISA Kits) locus using rAAV1-mediated CRISPR/Cas9 abrogates angiogenesis in the mouse models of oxygen-induced retinopathy and laser-induced choroid neovascularization.
Our study demonstrates that Prox1-GFP/Flk1 (show KDR ELISA Kits)::myr-mCherry mice are a useful model for studying coordinated hemangiogenic and lymphangiogenic responses
Endoglin (show ENG ELISA Kits) prevents vascular malformation by regulating flow-induced cell migration and specification through VEGFR2 (show KDR ELISA Kits) signalling
CLEC14A (show CLEC14A ELISA Kits) acts in vascular homeostasis by fine-tuning VEGFR-2 (show KDR ELISA Kits) and VEGFR-3 signaling in endothelial cells
The elevated soluble VEGFR-2 (show KDR ELISA Kits) that was found in the aortas of apoE (show APOE ELISA Kits)(-/-) mice with atherosclerosis binds to and diminishes the activity of VEGF-C (show VEGFC ELISA Kits).
Rare inherited and de novo variants in 2,871 congenital heart disease probands identified GDF1 (show GDF1 ELISA Kits), MYH6 (show MYH6 ELISA Kits), and FLT4 as causative genes.
Data show that VEGF-C (show VEGFC ELISA Kits), VEGF-D (show Figf ELISA Kits), and VEGFR-3 were expressed in a substantial percentage of breast carcinomas.
There was a significant decrease in VEGFR3 expression in pulmonary arterial endothelial cells from pulmonary arterial hypertension patients.
By treating LECs with VEGF (show VEGFA ELISA Kits)-C156S and analyzing subsequent changes in gene expression, we identified several 'immediate early (show JUN ELISA Kits)' transcription factors that showed a rapid transient upregulation VEGFR-3 stimulation. these results reveal an important and unanticipated role of HOXD10 (show HOXD10 ELISA Kits) in the regulation of VEGFR-3 signaling in lymphatic endothelial cells, and in the control of lymphangiogenesis and permeability.
The normalized methylation values for the VEGFR1 (show FLT1 ELISA Kits), VEGFR2 (show KDR ELISA Kits) and VEGFR3 promoters tended to be higher in the tumour cell lines than in normal tonsil samples, whereas amounts of VEGFR1 (show FLT1 ELISA Kits), VEGFR2 (show KDR ELISA Kits) and VEGFR3 messenger RNA were significantly higher
These results indicate that VEGF-C (show VEGFC ELISA Kits)-induced MSC (show MSC ELISA Kits) osteogenesis is mediated through VEGFR2 (show KDR ELISA Kits) and VEGFR3, and followed the activation of the ERK (show EPHB2 ELISA Kits)/RUNX2 (show RUNX2 ELISA Kits) signaling pathway.
Assessment of VEGFR-2/VEGFR (show KDR ELISA Kits)-3 on tumor samples might serve as a putative prognostic factor in renal cell carcinoma cases, identifying a subset of patients that may benefit from antiangiogenic treatments targeting VEGFR (show KDR ELISA Kits) receptors.
This study suggests that NRP1 (show NELL1 ELISA Kits) expression and LVD are independent factors that are likely to predict the risk of LN metastasis in squamous cell carcinoma (SCC (show CYP11A1 ELISA Kits))of the tongue, whereas the expression of VEGFC (show VEGFC ELISA Kits), VEGFR3, CCR7 (show CCR7 ELISA Kits), and SEMA3E (show SEMA3E ELISA Kits) are nonindependent predictive factors
Data indicate that vascular endothelial growth factor D (VEGF-D (show Figf ELISA Kits)) was the best indicator of metastasis and vascular endothelial growth factors and receptor-3 (VEGFR-3) may help to determine the prognosis and management of colorectal cancer (CRC (show CALR ELISA Kits)).
The summarizes the structure and function features of pathway-related molecules of VEGFC (show VEGFC ELISA Kits)/D-VEGFR3/NRP2 (show NELL2 ELISA Kits) axis, stages of various tumors and their molecular mechanisms and significances in tuthe expression changes of these molecules in different anatomic organs or histopathologic types or development lymphatic metastasis.
miR (show MYLIP ELISA Kits)-126a directs lymphatic endothelial cell sprouting and extension by interacting with Cxcl12a-mediated chemokine (show CCL1 ELISA Kits) signaling and Vegfc (show VEGFC ELISA Kits)-Flt4 signal axis.
Ca(2 (show CA2 ELISA Kits)+) oscillations depended upon VEGF receptor-2 (Vegfr2) and Vegfr3 in endothelial cells budding from the dorsal aorta (DA) and posterior cardinal (show CARD8 ELISA Kits) vein, respectively.
Experiments in mice and zebrafish demonstrate that changing levels of VEGFR3/Flt4 modulates aortic lumen diameter consistent with flow-dependent remodeling
In the embryo, phenotypes driven by increased Vegfc (show VEGFC ELISA Kits) are suppressed in the absence of Ccbe1 (show CCBE1 ELISA Kits), and Vegfc (show VEGFC ELISA Kits)-driven sprouting is enhanced by local Ccbe1 (show CCBE1 ELISA Kits) overexpression. Moreover, Vegfc (show VEGFC ELISA Kits)- and Vegfr3-dependent Erk (show MAPK1 ELISA Kits) signaling is impaired in the absence of Ccbe1 (show CCBE1 ELISA Kits).
Flt4 plays an essential role in lymphangiogenesis [review]
The parallel growth of motoneuron axons with the dorsal aorta depends on Vegfc (show VEGFC ELISA Kits)/Vegfr3 signaling in zebrafish.
Rspo1-Wnt-VegfC-Vegfr3 signaling plays a crucial role as an endothelial-autonomous permissive cue for developmental angiogenesis.
flt4 signalling is suppressed by Dll4 (show DLL4 ELISA Kits) in developing zebrafish intersegmental arteries.
This gene encodes a tyrosine kinase receptor for vascular endothelial growth factors C and D. The protein is thought to be involved in lymphangiogenesis and maintenance of the lymphatic endothelium. Mutations in this gene cause hereditary lymphedema type IA.
, fms-like tyrosine kinase 4
, soluble VEGFR3 variant 1
, soluble VEGFR3 variant 2
, soluble VEGFR3 variant 3
, tyrosine-protein kinase receptor FLT4
, vascular endothelial growth factor receptor 3
, chylous ascites
, receptor protein tyrosine kinase
, vascular endothelial growth factor receptor-3
, FMS-like tyrosine kinase 4
, tyrosine kinase VEGFR-3
, receptor tyrosine kinase Flt4