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Human GDNF ELISA Kit for Sandwich ELISA - ABIN625302
Ye, Dang, Viet, Dolan, Schmidt: Analgesia targeting IB4-positive neurons in cancer-induced mechanical hypersensitivity. in The journal of pain : official journal of the American Pain Society 2012
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Human GDNF ELISA Kit for Sandwich ELISA - ABIN921058
Wang, Hou, Yuan, Hou, Liu, Li, Zhang: Association study between plasma GDNF and cognitive function in late-onset depression. in Journal of affective disorders 2011
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Mouse (Murine) GDNF ELISA Kit for Sandwich ELISA - ABIN1672848
Durbec, Marcos-Gutierrez, Kilkenny, Grigoriou, Wartiowaara, Suvanto, Smith, Ponder, Costantini, Saarma: GDNF signalling through the Ret receptor tyrosine kinase. in Nature 1996
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Rat (Rattus) GDNF ELISA Kit for Sandwich ELISA - ABIN411276
Bermingham, Hillermann, Gilmour, Martin, Fisher: Human glial cell line-derived neurotrophic factor (GDNF) maps to chromosome 5. in Human genetics 1996
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Rat (Rattus) GDNF ELISA Kit for Sandwich ELISA - ABIN2684356
Shinko, Agari, Kameda, Yasuhara, Kondo, Tayra, Sato, Sasaki, Sasada, Takeuchi, Wakamori, Borlongan, Date: Spinal cord stimulation exerts neuroprotective effects against experimental Parkinson's disease. in PLoS ONE 2014
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Rat (Rattus) GDNF ELISA Kit for Sandwich ELISA - ABIN416076
Yang, Liu, Chen, An, Zhang, Zhang, Zhao, He, Hao: Efficient Generation of Functionally Active Spinal Cord Neurons from Spermatogonial Stem Cells. in Molecular neurobiology 2016
Mouse (Murine) GDNF ELISA Kit for Sandwich ELISA - ABIN365485
Mäkelä, Toppari, Rivero-Müller, Ventelä: Reconstruction of mouse testicular cellular microenvironments in long-term seminiferous tubule culture. in PLoS ONE 2014
Rat (Rattus) GDNF ELISA Kit for Sandwich ELISA - ABIN366463
Afzalpour, Chadorneshin, Foadoddini, Eivari: Comparing interval and continuous exercise training regimens on neurotrophic factors in rat brain. in Physiology & behavior 2015
Human GDNF ELISA Kit for Sandwich ELISA - ABIN2684353
Lin, Doherty, Lile, Bektesh, Collins: GDNF: a glial cell line-derived neurotrophic factor for midbrain dopaminergic neurons. in Science (New York, N.Y.) 1993
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Results provide evidence that uPA (show PRAP1 ELISA Kits) and IGF1R (show IGF1R ELISA Kits) directly interact with uPAR (show PLAUR ELISA Kits) enhancing malignant potential of triple-negative breast cancer.
suggest that the low endogenous levels of uPA (show PRAP1 ELISA Kits) in blood are actively regulated, and that the regulatory mechanisms are disrupted in QPD in a megakaryocyte-specific manner
an intricate link between caveolin-1 (show CAV1 ELISA Kits) and Src kinase (show CSK ELISA Kits)-mediated cell signaling and alveolar epithelial cell apoptosis due to loss of SP-C (show SFTPC ELISA Kits) expression through p53 (show TP53 ELISA Kits) and uPA (show PRAP1 ELISA Kits) system-mediated cross-talk, is reported.
there is a decrease in epidermal GDNF and GFRalpha-1 protein expression in normal human skin with ageing
In SOD1 (show SOD1 ELISA Kits)(G93A) spinal cords, we verified a strict correlation in the expression of the TNFalpha (show TNF ELISA Kits), TNFR1 (show TNFRSF1A ELISA Kits) and GDNF triad at different stages of disease progression. Yet, ablation of TNFR1 (show TNFRSF1A ELISA Kits) completely abolished GDNF rises in both SOD1 (show SOD1 ELISA Kits)(G93A) astrocytes and spinal cords, a condition that accelerated motor neuron degeneration (show CLN8 ELISA Kits) and disease progression
results show that the uPA (show PRAP1 ELISA Kits)/uPAR (show PLAUR ELISA Kits)/LRP1 (show LRP1 ELISA Kits) system is a potential target for the development of therapeutic strategies to promote axonal recovery following a CNS injury
In functional dyspepsia patients, duodenal expression of GDNF protein was significantly increased compared with controls. GDNF was localized in enteric glial cells, eosinophils, and epithelial cells.
The present research concluded that aspirin suppressed prostate cancer cell invasion by reducing MMP-9 (show MMP9 ELISA Kits) activity and uPA (show PRAP1 ELISA Kits) expression through decreasing of IKK-beta (show IKBKB ELISA Kits)-mediated NF-kappaB (show NFKB1 ELISA Kits) activation, indicating that the ability of aspirin to inhibit cell invasion might be useful in the chemoprevention of metastatic prostate cancer.
Suppression of miR-383 may increase the therapeutic potential of human bone-marrow-derived MSCs in treating spinal cord injury via augmentation of GDNF protein levels.
These studies identify uPA (show PRAP1 ELISA Kits)-dependent de-repression of vegfr1 (show FLT1 ELISA Kits) and vegfr2 (show KDR ELISA Kits) gene transcription through binding to HHEX/PRH (show HHEX ELISA Kits) as a novel mechanism by which uPA (show PRAP1 ELISA Kits) mediates the pro-angiogenic effects of VEGF (show VEGFA ELISA Kits) and identifies a potential new target for control of pathologic angiogenesis.
results contradict previous studies suggesting that mammalian GFRalpha1 (show GFRA1 ELISA Kits) and GDNF cannot bind and activate non-mammalian RET (show RET ELISA Kits) and vice versa
the dynamics of glial cell line-derived neurotrophic factor (gdnf) and nitric oxide synthases (nos) mRNA expression in various regions of zebrafish brain
GDNF family ligands including tyrosine kinase receptor (show KDR ELISA Kits) RET (show RET ELISA Kits) are investigated within the adult zebrafish brain.
GDNF is a major determinant of directed neuritic growth , and GDNF acts by promoting local neurite outgrowth.
These results demonstrated the expression of the GDNF receptorial complex in adult zebrafish cerebellum and suggest an autocrine mode of action of GDNF in Purkinje cells.
These results showed that the expression of GDNF is not probably restricted during development but it might be involved in the physiology of adult zebrafish retina.
The expression of glial cell line-derived neurotrophic factor (GDNF) was not restricted to developmental periods but it seems that this factor might be involved in adult zebrafish brain physiology, as observed in mammals.
Analysis of striatal brain samples confirms increased GDNF expression in lentiviral vector-GDNF treated aged animals that correlates with functional improvements and preserved dopaminergic markers, dependent upon GDNF levels.
Ret (show RET ELISA Kits) is essential to mediate GDNF's neuroprotective and neuroregenerative effect in a Parkinson disease mouse model.
identified IFNg (show IFNG ELISA Kits), Neurturin (Nrtn (show NRTN ELISA Kits)), and glial-derived neurotrophic factor (GDNF) as ligands with unexpected roles in promoting neurogenic differentiation (show NEUROD1 ELISA Kits) of Neural Precursor Cells in vivo.
Using an organ culture system for prostate development and Ret mutant mice, we demonstrate that RET-mediated GDNF signaling in UGS increases proliferation of mesenchyme cells and suppresses androgen-induced proliferation and differentiation of prostate epithelial cells, inhibiting prostate development.
The GDNF-GFRalpha1 (show GFRA1 ELISA Kits) complex is essential for proper hippocampal circuit development.
GDNF signals were able to induce the stratified aggregate formation of GFRalpha1 (show GFRA1 ELISA Kits)-positive undifferentiated spermatogonia
Our results show the existence of two subpopulations of peptidergic nociceptors characterized by the presence of CGRP (show CALCA ELISA Kits), one expressing BDNF (show BDNF ELISA Kits) (plus SP), the other expressing GDNF (plus SST (show SST ELISA Kits)), suggesting a different role for these two neurotrophic factors in the discrimination of specific painful stimuli modalities.
The data of this study suggested that short-term exposure to hyperoxic conditions can affect the regulation and expression of BDNF (show BDNF ELISA Kits) potentially leading to alterations in neural development.
The Gdnf cKO males sired up to two litters but became infertile due to collapse of spermatogenesis and loss of undifferentiated spermatogonia
Our results reveal the role of GDNF in nigrostriatal dopamine system postnatal development and adult function, and highlight the importance of correct spatial expression of GDNF
Spatial expression analysis by whole-mount in situ hybridization showed that the GDNF mRNA was predominantly detected in somites, pronephros, pharyngeal arches, epibranchial placodes, digestive tract and some of the lateral line structure.
This gene encodes a highly conserved neurotrophic factor. The recombinant form of this protein was shown to promote the survival and differentiation of dopaminergic neurons in culture, and was able to prevent apoptosis of motor neurons induced by axotomy. The encoded protein is processed to a mature secreted form that exists as a homodimer. The mature form of the protein is a ligand for the product of the RET (rearranged during transfection) protooncogene. Multiple transcript variants encoding different isoforms have been found for this gene. Mutations in this gene may be associated with Hirschsprung disease.
, plasminogen activator, urinary
, urokinase-type plasminogen activator
, Glial cell line derived neutrophic factor
, astrocyte-derived trophic factor
, glial cell line derived neurotrophic factor
, glial cell line-derived neurotrophic factor
, glial cell line-derived neurotrophic factor long form
, glial cell derived neurotrophic factor
, glial cell-line derived neurotrophic factor
, neurotrophic factor
, glial cell line-derived neurotrophic factor a
, glia-derived neurotrophic growth factor