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anti-Human Thymidine Phosphorylase Antibodies:
anti-Mouse (Murine) Thymidine Phosphorylase Antibodies:
anti-Rat (Rattus) Thymidine Phosphorylase Antibodies:
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Human Monoclonal Thymidine Phosphorylase Primary Antibody for ICC, IF - ABIN251182
Nie, Bhat, Al-Saleem, Vonderheid, Hou: Expression of thymidine phosphorylase in lymph nodes involved with mycosis fungoides and sézary syndrome. in Advances in hematology 2011
Human Monoclonal Thymidine Phosphorylase Primary Antibody for IHC (p), IP - ABIN5590451
Fox, Westwood, Moghaddam, Comley, Turley, Whitehouse, Bicknell, Gatter, Harris: The angiogenic factor platelet-derived endothelial cell growth factor/thymidine phosphorylase is up-regulated in breast cancer epithelium and endothelium. in British journal of cancer 1996
Data show that thymidine phosphorylase (TYMP) positive patients had higher systemic immune-inflammation index (SII (show TCEA1 Antibodies)) score.
Investigated the contribution of myeloma-expressed thymidine phosphorylase (TP) to bone lesions. In osteoblast progenitors, TP up-regulated the methylation of RUNX2 (show RUNX2 Antibodies) and osterix (show SP7 Antibodies), leading to decreased bone formation. In osteoclast progenitors, TP up-regulated the methylation of IRF8 (show IRF8 Antibodies), leading to increased bone resorption.
TP(thymidine phosphorylase ) curbed the expression of three proteins-IRF8 (show IRF8 Antibodies), RUNX2 (show RUNX2 Antibodies), and osterix (show SP7 Antibodies). This downregulation was epigenetically driven: High levels of 2DDR, a product of TP secreted by myeloma cells, activated PI3K (show PIK3CA Antibodies)/AKT (show AKT1 Antibodies) signaling and increased the methyltransferase DNMT3A's expression
TP expression is associated with tumor stage, histological grade and thrombocytosis in patients with renal cell carcinoma (show MOK Antibodies)
Studied the role of thymidine phosphorylase (TP) in promoting the invasion and metastasis of hepatocellular carcinoma (HCC (show FAM126A Antibodies)). It was shown that TP correlated with MMP2 (show MMP2 Antibodies) and MMP9 (show MMP9 Antibodies) expression, and that TP mediated the migration of tumor cells through activity of MMPs in HCC (show FAM126A Antibodies) cell lines.
Increases in gene expression levels of TYMP, DPYD (show DPYD Antibodies), and HIF1A (show HIF1A Antibodies) in tumor tissues at 7 days after the start of CRT (show SLC6A8 Antibodies) may be useful for predicting the efficacy of CRT (show SLC6A8 Antibodies) including S-1 or UFT
the highest expression of GGH (show GGH Antibodies) and EGFR (show EGFR Antibodies) was noted in the left-sided colon; the highest expression of DHFR (show DHFR Antibodies), FPGS (show FPGS Antibodies), TOP1 (show TOP1 Antibodies) and ERCC1 (show ERCC1 Antibodies) was noted in the rectosigmoid, whereas TYMP expression was approximately equivalent in the right-sided colon and rectum
High VEGF (show VEGFA Antibodies) expression was subsequently correlated with a short overall survival rate for patients exhibiting lymph node metastasis (P=0.0128); however, there was no significant difference in overall survival rate regarding the expression levels of TP and CD34 (show CD34 Antibodies)
Pretreatment expression of thymidine phosphorylase/HIF-1alpha (show HIF1A Antibodies) were found to predict pathologic response and outcomes in clinical stage II/III rectal cancer receiving neoadjuvant chemoradiotherapy.
Gastrointestinal cancer patient carriers of the thymidine phosphorylase rs11479 T allele, with a low platelet count, could be at risk of a poor outcome.
data identify TYMP as an astrocyte-derived permeability factor, and suggest TYMP and VEGFA (show VEGFA Antibodies) together promote blood-brain barrier breakdown.
TYMP participates in multiple platelet signaling pathways and regulates platelet activation and thrombosis. Targeting TYMP might be a novel antiplatelet and antithrombosis therapy.
Thymidine phosphorylase and UPP1 (show UPP1 Antibodies) double knockout mice showed increased thymidine and deoxyuridine in tissues and developed encephalopathy.
This gene encodes an angiogenic factor which promotes angiogenesis in vivo and stimulates the in vitro growth of a variety of endothelial cells. It has a highly restricted target cell specificity acting only on endothelial cells. Mutations in this gene have been associated with mitochondrial neurogastrointestinal encephalomyopathy. Multiple alternatively spliced transcript variants have been identified.
, endothelial cell growth factor 1 (platelet-derived)
, platelet derived growth factor, endothelial cell
, thymidine phosphorylase
, Thymidine phosphorylase