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anti-Mouse (Murine) CD28 Antibodies:
anti-Human CD28 Antibodies:
anti-Rat (Rattus) CD28 Antibodies:
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Mouse (Murine) Monoclonal CD28 Primary Antibody for BR, CyTox - ABIN1176978
Gelfanov, Lai, Gelfanova, Dong, Su, Liao: Differential requirement of CD28 costimulation for activation of murine CD8+ intestinal intraepithelial lymphocyte subsets and lymph node cells. in Journal of immunology (Baltimore, Md. : 1950) 1995
Show all 14 Pubmed References
Mouse (Murine) Monoclonal CD28 Primary Antibody for BR, CyTox - ABIN2689144
Bluestone: New perspectives of CD28-B7-mediated T cell costimulation. in Immunity 1995
Show all 13 Pubmed References
Mouse (Murine) Monoclonal CD28 Primary Antibody for FACS - ABIN2689149
Wells, Gudmundsdottir, Turka et al.: Following the fate of individual T cells throughout activation and clonal expansion. Signals from T cell receptor and CD28 differentially regulate the induction and duration of a proliferative ... in The Journal of clinical investigation 1998
Show all 13 Pubmed References
Mouse (Murine) Monoclonal CD28 Primary Antibody for Func, ICC - ABIN457400
Albert, Yu, Martin, Anasetti: Prevention of lethal acute GVHD with an agonistic CD28 antibody and rapamycin. in Blood 2005
Show all 10 Pubmed References
Mouse (Murine) Monoclonal CD28 Primary Antibody for Func, FACS - ABIN370901
Gross, Callas, Allison: Identification and distribution of the costimulatory receptor CD28 in the mouse. in Journal of immunology (Baltimore, Md. : 1950) 1992
Show all 16 Pubmed References
Rat (Rattus) Monoclonal CD28 Primary Antibody for Func, FACS - ABIN2749055
Guillonneau, Séveno, Dugast, Li, Renaudin, Haspot, Usal, Veziers, Anegon, Vanhove: Anti-CD28 antibodies modify regulatory mechanisms and reinforce tolerance in CD40Ig-treated heart allograft recipients. in Journal of immunology (Baltimore, Md. : 1950) 2007
Show all 9 Pubmed References
Rat (Rattus) Monoclonal CD28 Primary Antibody for Func, FACS - ABIN2749056
Kerstan, Armbruster, Leverkus, Hünig: Cyclosporin A abolishes CD28-mediated resistance to CD95-induced apoptosis via superinduction of caspase-3. in Journal of immunology (Baltimore, Md. : 1950) 2006
Show all 8 Pubmed References
Rat (Rattus) Monoclonal CD28 Primary Antibody for Func, FACS - ABIN611634
van den Brandt, Wang, Reichardt: Resistance of single-positive thymocytes to glucocorticoid-induced apoptosis is mediated by CD28 signaling. in Molecular endocrinology (Baltimore, Md.) 2004
Show all 9 Pubmed References
Human Monoclonal CD28 Primary Antibody for FACS, Func - ABIN638435
Nunès, Klasen, Ragueneau, Pavon, Couez, Mawas, Bagnasco, Olive: CD28 mAbs with distinct binding properties differ in their ability to induce T cell activation: analysis of early and late activation events. in International immunology 1993
Show all 7 Pubmed References
Human Monoclonal CD28 Primary Antibody for FACS, Func - ABIN349705
Galea-Lauri, Darling, Gan, Krivochtchapov, Kuiper, Gäken, Souberbielle, Farzaneh: Expression of a variant of CD28 on a subpopulation of human NK cells: implications for B7-mediated stimulation of NK cells. in Journal of immunology (Baltimore, Md. : 1950) 1999
Show all 6 Pubmed References
findings revealed a dual mechanism of monocyte and neutrophil recruitment by T cells relying on overlapping and nonoverlapping roles for the noninducible costimulatory receptor CD28 and the inflammatory cytokine TNF (show TNF Antibodies)
results are consistent with a complex pathway in which CD28 is the primary driver of Treg proliferation and CTLA-4 (show CTLA4 Antibodies) functions as the main brake but is also dependent on TCR signals and interactions with CD80 (show CD80 Antibodies)/CD86 (show CD86 Antibodies).
data suggest that mPEG PV1 (show PLVAP Antibodies)-Fab (show FANCB Antibodies)' acts mainly on IFN-gamma (show IFNG Antibodies)-producing CD4 (show CD4 Antibodies)+ T cells and emphasize that this specific CD28 blockade strategy is a potential specific and alternative tool for the treatment of autoimmune disorders in the eye.
the scaffolding role of RLTPR predominates during CD28 co-stimulation and underpins the similar function of RLTPR in human and mouse T cells.
BAFF (show TNFSF13B Antibodies) upregulates CD28/B7 and CD40 (show CD40 Antibodies)/CD154 (show CD40LG Antibodies) expression, and promotes the interactions between T and B cells in a BAFF-R (show TNFRSF13C Antibodies)-dependent manner
we report that cell-intrinsic deletion of CD28 after the peak of the primary response does not affect the establishment, maintenance, or recall of long-term memory. Thus, if given sufficient time, the progeny of primed CD8 (show CD8A Antibodies)(+) T cells adapt to the absence of this costimulator.
Deletion of CD28 co-stimulatory signals exacerbates left ventricular remodeling and increases cardiac rupture after MI through prolongation of the inflammatory period and reduction of collagen fiber in the infarct scars.
identified a new plasmacytoid dendritic cells regulatory mechanism by which the same CD28 molecule that promotes stimulation in most cells
Ndrg1 (show NDRG1 Antibodies) is phosphorylated and degraded by CD28 signalling in a proteasome-dependent manner.
The CD4/CD8 positive lymphocyte count and the expression of CD28 and CD38 antigens in the murine schistosomiasis japonica model are reported.
The mutant CD28 isoforms could accelerate tumor cell growth.
CD3 (show CD3 Antibodies)/28-activated T cells expanded in IL-7 (show IL7 Antibodies) and IL-15 (show IL15 Antibodies) produced greater expansion of memory stem T cells and central memory T cell-derived T cells compared with IL-2 (show IL2 Antibodies). Our strategy provides a powerful tool to elucidate the characteristics of CAR-modified T cells, regardless of the protocol used for expansion, reveals the functional properties of each expanded T cell subset.
identified recurrent mutations in CD28 in peripheral T-cell lymphomas. Molecular modeling studies on each of these mutations suggested how these mutants result in increased affinities.
Our data show that mast cells can costimulate human CD4 (show CD4 Antibodies)(+) T cells to induce strong T-cell proliferation, but that therapies aiming at disrupting the interaction of CD28 and B7 molecules do not inhibit mast cell mediated T-cell activation.
High CD28 Circulating Levels are associated with Breast Cancer.
The CTLA4 (show CTLA4 Antibodies)-CD28 gene fusion is likely a major contributor to the pathogenesis of T-cell lymphomas and represents a potential target for anti-CTLA4 (show CTLA4 Antibodies) cancer immunotherapy.
Following phosphorylation of the tyrosine, the proteins growth factor receptor-bound protein 2 (Grb2 (show GRB2 Antibodies)), Grb2 (show GRB2 Antibodies)-related adaptor downstream of Shc (show SHC1 Antibodies) (Gads (show GRAP2 Antibodies)), and p85 subunit of phosphoinositide 3-kinase may bind to pYMNM (where pY is phosphotyrosine) via their Src (show SRC Antibodies) homology 2 (SH2) domains, leading to downstream signaling to distinct immune pathways. These three adaptor proteins bind to the same site on CD28 with variable affinity
CD28 family receptors are potential clinical indicators for the rapid monitoring of changes in T cell function during CHB treatment.
The eQTL mapping analysis revealed that the variations in CD28 and NFKB1 gene content might affect the abundance of transcripts of CD28 and Family with sequence similarity 177 member A1 (FAM177A1) genes, respectively. These results suggest that CD28 and NFKB1 gene variants may be associated with increased risks to IRM.
The protein encoded by this gene is essential for T-cell proliferation and survival, cytokine production, and T-helper type-2 development. Several alternatively spliced transcript variants encoding different isoforms have been found for this gene.
, T-cell-specific surface glycoprotein CD28
, CD28 antigen (Tp44)
, T-cell-specific surface glycoprotein CD28 homolog
, T-cell costimulatory molecule CD28
, cell surface protein
, costimulatory molecule B7 receptor CD28
, antigen CD28
, T-cell specific surface glycoprotein CD28