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changes in mRNA expression of selected toll (show TLR4 Proteins)-like receptors (TLR2, TLR4 (show TLR4 Proteins), TLR7), stress cytokine prolactin (PRL (show PRL Proteins)), and proand anti-inflammatory cytokines (TNF-alpha (show TNF Proteins), IL-6 (show IL6 Proteins), IL-12 (show IL12A Proteins), IL-10 (show IL10 Proteins)) in peripheral blood monocytes of celiac disease patients, were investiagted.
TLR8 (show TLR8 Proteins) polymorphisms were associated with an increased risk and TLR7 polymorphisms with a decreased risk of recurrent rhinovirus infections.
We found lower expression levels of TLR1 (show TLR1 Proteins), TLR3 (show TLR3 Proteins), TLR4 (show TLR4 Proteins), TLR7 and TLR9 (show TLR9 Proteins) in PBMCs from patients with ALL compared with those from control patients. We also observed that the PBMCs from patients with Pre-B and B ALL had lower TLR4 (show TLR4 Proteins) expression than controls
interferon alpha (IFN-alpha (show IFNA Proteins)) secretion induced by Toll-like receptor 7 and Toll-like receptor 8 (show TLR8 Proteins) (TLR7/TLR8 (show TLR8 Proteins)) activation was observed in common variable immunodeficiency (CVID (show TNFRSF13B Proteins)), which was recovered with Toll-like receptor 9 (TLR9 (show TLR9 Proteins)) signaling.
this study shows that TLR7 gene polymorphism is associated with susceptibility to HIV and HCV co-infection
TLR7 gene rs2897827 may influence TLR7 mRNA expression and the plasma ApoA1 (show APOA1 Proteins) level in male ischemic stroke patients.
Considering that chronic hepatitis B infection is not yet curable, it could be possible to activate TLR7-related immunological pathways as a therapy directed towards persistent HBV infection
Both TLR 5 (show TLR5 Proteins) and 7 are expressed in salivary adenoid cystic carcinoma on the cell membranes as well as in cytoplasm.
human Toll-like receptor 7 ligand binding domain
TLR7 mRNA expression was significantly elevated in patients with relapsing remitting multiple sclerosis.
this study demonstrates the critical role of Gfi1 (show ZNF163 Proteins) in the regulation of myeloid cells, and prevention of spontaneous lupus autoimmunity by negatively controlling TLR7 signaling
this study shows that beta,beta-dimethylacryloyl alkannin could inhibit psoriasis-activated dendritic cells via the TLR7/8 pathway
synergy of TLR3 (show TLR3 Proteins) and 7 ligands could significantly enhance the function of DCs to present inactivated PRRSV antigen through TRIF (show RNF138 Proteins)/MyD88 (show MYD88 Proteins)-NF-kappaB (show NFKB1 Proteins) signaling pathway and be used as adjuvant candidate for the development of novel PRRS inactivated vaccine
Orally administered R848 triggers TLR-7 on CD11c (show ITGAX Proteins)(+) dendritic cells, inducing interleukin-23 (IL-23 (show IL23A Proteins)) expression followed by a burst of IL-22 (show IL22 Proteins) secretion by innate lymphoid cells, leading to Reg3gamma expression and restoration of colonization resistance against vancomycin-resistant enterococcus.
The results imply that recognition of resident viruses by TLR3 (show TLR3 Proteins) and TLR7 is required for protective immunity during gut (show GUSB Proteins) inflammation.
pulmonary interstitial macrophages may not be a source of IL-33 (show IL33 Proteins) during Respiratory syncytial virus infection
Aging Impairs the Ability of Conventional Dendritic Cells to Cross-Prime CD8 (show CD8A Proteins)+ T Cells upon Stimulation with a TLR7 Ligand.
Type I Interferons maintain expression of TLR7 in B cells and conventional dendritic cells in different ways; total amount of TLR7 is kept in B cells and TLR7(+) population is retained among conventional dendritic cells.
TLR7/9-MyD88 (show MYD88 Proteins) might be important in the induction of neutrophils and the relevant inflammatory mediators in Bjerkandera adusta-Induced Lung Inflammation
These data provide direct evidence that B cells require TLR7-dependent priming through an autophagy-dependent mechanism before autoimmunity is induced, thereafter involving many cell types.
The results from this study demonstrate that expression of at least TLR3 (show TLR3 Proteins), TLR7 and TLR8 (show TLR8 Proteins) is stimulated upon bovine alpha-herpesvirus infection of the brain.
expression of TLR3 (show TLR3 Proteins), TLR7 and TLR9 (show TLR9 Proteins) in alveolar macrophages infected with different genotype 1 PRRSV strains
Porcine TLR7 and TLR8 (show TLR8 Proteins) genes from pig lymph node tissue, were cloned and characterized.
The messenger RNA sequences and exon/intron structures of Toll (show TLR4 Proteins)-like receptors 3, 7, and 8 were determined.
A total of 22 polymorphic sites were observed in TLR7 gene of 24 goats representing 12 different breeds, out of which 19 were present within the coding region and three in 3'UTR.
Activation of Toll-like receptor 7 might prevent development of airway hyperresponsiveness by acting on the airway smooth muscle.
The protein encoded by this gene is a member of the Toll-like receptor (TLR) family which plays a fundamental role in pathogen recognition and activation of innate immunity. TLRs are highly conserved from Drosophila to humans and share structural and functional similarities. They recognize pathogen-associated molecular patterns (PAMPs) that are expressed on infectious agents, and mediate the production of cytokines necessary for the development of effective immunity. The various TLRs exhibit different patterns of expression. This gene is predominantly expressed in lung, placenta, and spleen, and lies in close proximity to another family member, TLR8, on chromosome X.
toll-like receptor 7
, toll-like receptor 7-like
, toll like receptor 7
, toll-like receptor 7-long