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Human CTNNB1 ELISA Kit for Sandwich ELISA - ABIN832441
Gaudio, Privitera, Battaglia, Torrisi, Sidoti, Pulvirenti, Canzonieri, Tringali, Fiore: Sclerostin levels associated with inhibition of the Wnt/?-catenin signaling and reduced bone turnover in type 2 diabetes mellitus. in The Journal of clinical endocrinology and metabolism 2012
Rat (Rattus) CTNNB1 ELISA Kit for Sandwich ELISA - ABIN431880
Shao, Cai, Sheng, Yin: Role of SDF-1 and Wnt signaling pathway in the myocardial fibrosis of hypertensive rats. in American journal of translational research 2015
Mouse (Murine) CTNNB1 ELISA Kit for Sandwich ELISA - ABIN424163
Chen, Feng, Bao, Li, Zhang, Shen, Zhao, Guo, Jing, Lin, Zong: Adverse Effects of Osteocytic Constitutive Activation of ß-Catenin on Bone Strength and Bone Growth. in Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research 2015
temporal expression of CTNNB1 mRNA during dermal wound repair in the horse
lung myeloid cells are responsive to Lrp5 (show LRP5 ELISA Kits)/beta-catenin signaling, leading to differentiation of an alveolar macrophage subtype that antagonizes the resolution of lung fibrosis.
Vascular smooth muscle beta-catenin is required for formation of neointima after vascular injury.
Fzd7 expressed by endothelial cells drives postnatal angiogenesis via activation of Dvl (show DVL1 ELISA Kits)/beta-catenin signaling and can control the integrative interaction of Wnt (show WNT2 ELISA Kits) and Notch (show NOTCH1 ELISA Kits) signaling during postnatal angiogenesis.
The data suggest that vitamin D and calcium signaling are necessary components of the epidermal response to wounding, likely by regulating stem cell activation through increased beta-catenin transcriptional activity.
Data show that methyl gallate (MG) prevents beta-catenin degradation during 3T3-L1 adipocytes differentiation.
the persistence of p-beta-catenin(Y489) is a durable marker of fibroblast activation in Bronchopulmonary dysplasia and may play an important role in BPD disease pathobiology.
Activation of the EP3 (show PTGER3 ELISA Kits) receptor facilitates sprouting angiogenesis through protein kinase A/beta (show SUCLA2 ELISA Kits)-catenin/notch (show NOTCH1 ELISA Kits) signaling.
AT2R (show AGTR2 ELISA Kits) inhibits adipogenic differentiation in mesenchymal stem cells. Moreover, this inhibitory effect is associated with Wnt10b (show WNT10B ELISA Kits)/beta-catenin signaling.
Beta-catenin signaling controls C4ST-1 (show CHST11 ELISA Kits) gene expression through histone deacetylase (show HDAC1 ELISA Kits).
Data show that beta-catenin was increased in expression when Tbx1 (show TBX1 ELISA Kits) was inactivated, suggesting that there may be a negative feedback loop between canonical Wnt (show WNT2 ELISA Kits) and Tbx1 (show TBX1 ELISA Kits).
Mechanistically, Hif-3alpha2 binds to beta-catenin and destabilizes the nuclear beta-catenin complex.
Nuclear beta-catenin is an indication of an activated Wnt (show WNT2 ELISA Kits) pathway, therefore suggesting a possible role for Wnt (show WNT2 ELISA Kits) signalling during zebrafish tooth development and replacement.
Findings suggest that microRNA 19b (miR (show MYLIP ELISA Kits)-19b) regulates laterality development and heart looping in embryos by targeting beta-catenin ctnnb1.
Custos binds to beta-catenin in a Wnt (show WNT2 ELISA Kits) responsive manner without affecting its stability, but rather modulates the cytoplasmic to nuclear translocation of beta-catenin.
The role of the beta-catenin mechanosensitive pathway in mesoderm identity has been conserved over the large evolutionary distance separating zebrafish and Drosophila.
The Wnt (show WNT2 ELISA Kits)/beta-catenin signaling pathway establishes neuroanatomical asymmetries and their laterality. (Review)
This study demonstrated that beta-catenin/Wnt (show WNT2 ELISA Kits) signaling is initially required to activate cell-cycle re-entry in Muller glia following injury and that Wnt (show WNT2 ELISA Kits) signaling subsequently controls the fate of the progeny of those cell divisions.
A novel role for Eaf1 and Eaf2 (show EAF2 ELISA Kits) in inhibiting canonical Wnt (show WNT2 ELISA Kits)/beta-catenin signaling, which might form the mechanistic basis for Eaf1 and Eaf2 (show EAF2 ELISA Kits) tumor suppressor activity.
Ccr7 (show CCR7 ELISA Kits) functions during axis formation as a GPCR (show GPRC6A ELISA Kits) to inhibit beta-catenin, likely by promoting Ca(2 (show CA2 ELISA Kits)+) transients throughout the blastula.
ctnnb1 and ctnnb2 regulate multiple processes of laterality development in zebrafish embryos through similar and distinct mechanisms.
Together, these results indicate that capsaicin inhibits the patterning of the dorso-ventral and anterior-posterior body axes of embryo by repressing PP2A and thereby down-regulating the Wnt (show WNT2 ELISA Kits)/beta-catenin signaling.
maternal Wnt (show WNT2 ELISA Kits)/STOP signaling, but not beta-catenin signaling, has a role in cleavage after fertilization and cell cycle progression
Wnt (show WNT2 ELISA Kits) signalling can benefit from nucleo-cytoplasmic shuttling of APC (show APC ELISA Kits), Axin (show AXIN1 ELISA Kits) and GSK3, although they are in general beta-catenin antagonising proteins.
Phosphoinositide 3-kinase and Rac (show AKT1 ELISA Kits) polarization depend specifically on the N-cadherin (show CDH2 ELISA Kits)-p120 catenin (show CTNND1 ELISA Kits) complex, whereas myosin II light chain and actin filament polarization depend on the N-cadherin (show CDH2 ELISA Kits)-beta-catenin complex.
HERG (show KCNH2 ELISA Kits) channel activity is stimulated by beta-catenin
Zic3 (show ZIC3 ELISA Kits) can suppress Wnt (show WNT2 ELISA Kits)/beta-catenin signaling and control development of the notochord and Spemann's organizer.
Notch initially destabilises beta-catenin in a process that does not depend on its phosphorylation by GSK3
The tyrosine kinase receptor (show KDR ELISA Kits), PTK7 (show PTK7 ELISA Kits), is implicated in beta-catenin-dependent developmental processes.
Kazrin (show KAZ ELISA Kits) interacts with ARVCF (show ARVCF ELISA Kits)-catenin, spectrin and p190B (show ARHGAP5 ELISA Kits) RhoGAP (show ARHGAP1 ELISA Kits), and modulates RhoA (show RHOA ELISA Kits) activity.
The BC cells showed the coexpression of E- and P-cadherins, as well as release of the molecules b- and p120-catenins into the cytoplasm of tumor cells, which leads to the activation of intracellular mechanisms for changing the structure of the cytoskeleton and the level of proliferation
Our findings indicate that MSK1 (show RPS6KA5 ELISA Kits)/beta-catenin signaling serves as an escape survival signal upon PI3K (show PIK3CA ELISA Kits) inhibition and provides a strong rationale for the combined use of PI3K (show PIK3CA ELISA Kits) and MSK1 (show RPS6KA5 ELISA Kits)/beta-catenin inhibition to induce lethal growth inhibition in human GBM cells.
CTNNB1 mutation is associated with Adrenocortical Adenomas.
Our data suggests that total cellular b-catenin levels decrease in the presence of secreted frizzled-related protein 1 (show SFRP1 ELISA Kits) and Wnt inhibitory factor 1 (show WIF1 ELISA Kits), and a significant increase in cell death after tyrosine kinase (show TXK ELISA Kits) inhibitor treatment is observed. On the contrary, when secreted frizzled-related protein 1 (show SFRP1 ELISA Kits) is suppressed, total b-catenin levels increase in the cell and the cells become resistant to tyrosine kinase (show TXK ELISA Kits) inhibitors.
Results indicate a mechanism of beta-arrestin1 in modulating epithelial-mesenchymal transition (EMT) and glycogen synthase kinase 3 beta (GSK-3beta)/beta-catenin signaling in prostate cancer, and suggest that assessment of beta-arrestin1 may provide a potential therapeutic target for prostate cancer.
Cytoplasmic beta-catenin expression is an independent prognostic factor for conventional renal cell carcinoma
our surprise, NRAGE (show MAGED1 ELISA Kits) induces nuclear localization of beta-catenin and increases its DNA binding ability. Further studies reveal that NRAGE (show MAGED1 ELISA Kits) leads to the modification of beta-catenin/Arm with O-linked beta-N-acetylglucosamine (O-GlcNAc (show OGT ELISA Kits)), and failure of the association between beta-catenin/Arm and pygopus(pygo) protein, which is required for transcriptional activation of Wnt (show WNT2 ELISA Kits) target genes. Therefore, our findings suggest a ...
These findings indicate a novel role for zinc regulation in the PKCalpha (show PKCa ELISA Kits)/beta-catenin pathway and explain an important mechanism for controlling of stem cell program in lung cancer cells.
A long noncoding RNA, lnc-beta-Catm, elicits EZH2 (show EZH2 ELISA Kits)-dependent beta-catenin stabilization and sustains liver cancer stem cells self-renewal.
EVI1 (show MECOM ELISA Kits) transcription is directly regulated by LEF1 (show LEF1 ELISA Kits)/beta-catenin complex in myeloid blast crisis of chronic myeloid leukemia (show BCL11A ELISA Kits). Loss of p53 (show TP53 ELISA Kits) function as a key regulator for beta-catenin-EVI1 (show MECOM ELISA Kits) in myeloid blast crisis of chronic myeloid leukemia (show BCL11A ELISA Kits).
Flutamide exposure led to decreased beta-catenin in corpus luteum of mid/late pregnancy.
beta-catenin signaling is involved in formation of contractile membranes by dedifferentiated retinal pigment epithelium cells.
Wnt3a (show WNT3A ELISA Kits) produced a significant increase in heart valve interstitial cell number at day 4 and in the percentage of BrdU-positive nuclei at 24 h. The increase in proliferation was abolished by beta-catenin siRNA.
beta-catenin controls myocardin (show MYOCD ELISA Kits)-related transcription factor-dependent transcription and emerges as a critical regulator of an array of cytoskeletal genes
beta-Catenin plays a critical role in mediating TGF-beta1 (show TGFB1 ELISA Kits)-induced myofibroblast differentiation in aortic valve interstitial cells.
scratching-induced injury and repair of bronchial epithelial cells may involve inhibition of Glycogen synthase kinase 3beta activity which can lead to activation of the downstream signaling through beta-catenin
Data show that Wnt3a (show WNT3A ELISA Kits) can inhibit the adipogenic differentiation of porcine AMSCs, and suggest that Wnt (show WNT2 ELISA Kits)/beta-catenin signaling inhibits adipogenic differentiation potential and alters the cell fate from adipocytes to osteoblasts.
TNF-alpha (show TNF ELISA Kits) inhibits adipogenesis through stabilization of beta-catenin protein in porcine preadipocytes.
Bovine herpesvirus 1 transiently increased beta-catenin protein levels in bovine kidney cells, but not in rabbit skin cells.
These results demonstrate that activation of AKT (show AKT1 ELISA Kits) is required for gonadotropin regulation of CTNNB1 accumulation and subsequent ovarian E2 production.
beta-catenin, a transcription factor activated by the canonical Wnt signaling pathway, was frequently detected in bovine herpesvirus 1 ORF2-positive trigeminal ganglionic neurons of latently infected, but not mock-infected, calves.
These data demonstrate for the first time that FSH (show BRD2 ELISA Kits) regulates CTNNB1 protein and WNT2 (show WNT2 ELISA Kits) mRNA expressions in bovine granulosa cells, suggesting a potential role of canonical WNT (show WNT2 ELISA Kits) signaling in ovarian steroidogenesis and follicular growth of cattle.
These data provide evidence that testosterone increases cellular beta-catenin content which promotes the expression of beta-catenin-targeted genes and myogenesis in the muscle-derived stem cells of cattle.
that beta-catenin is a plasma membrane-associated protein (show PDZK1IP1 ELISA Kits) in airway smooth muscle that regulates active tension development, presumably by stabilizing cell-cell contacts and thereby supporting force transmission between neighboring cells.
This study demonstrated a relationship between the timing of the development of in vitro-produced bovine embryos and the distribution and localization of the junction protein beta-catenin.
TGF-beta3 (show TGFB3 ELISA Kits) induces the chondrogenic differentiation of pericytes by inducing Wnt (show WNT2 ELISA Kits)/beta-catenin signaling and T-cell factor-induced gene transcription.
cAMP/PKA regulation of GSK3beta/beta-catenin signaling contributes to the increase in progesterone production in corpus luteum.
Palmatin effect on osteoarthritis is likely mediated via the Wnt (show WNT2 ELISA Kits)/beta-catenin and Hedgehog (show SHH ELISA Kits) signaling
These results collectively showed that 2-Deoxy-D-glucose regulates dedifferentiation via beta-catenin pathway in rabbit articular chondrocytes.
The protein encoded by this gene is part of a complex of proteins that constitute adherens junctions (AJs). AJs are necessary for the creation and maintenance of epithelial cell layers by regulating cell growth and adhesion between cells. The encoded protein also anchors the actin cytoskeleton and may be responsible for transmitting the contact inhibition signal that causes cells to stop dividing once the epithelial sheet is complete. Finally, this protein binds to the product of the APC gene, which is mutated in adenomatous polyposis of the colon. Mutations in this gene are a cause of colorectal cancer (CRC), pilomatrixoma (PTR), medulloblastoma (MDB), and ovarian cancer. Three transcript variants encoding the same protein have been found for this gene.
catenin beta 1 subunit
, catenin beta-1
, beta catenin 1
, catenin (cadherin-associated protein), beta 1, 88kDa
, catenin beta-1-like
, catenin (cadherin associated protein), beta 1, 88kDa
, catenin beta
, beta catenin