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Human CTNNB1 ELISA Kit for Sandwich ELISA - ABIN832441
Gaudio, Privitera, Battaglia, Torrisi, Sidoti, Pulvirenti, Canzonieri, Tringali, Fiore: Sclerostin levels associated with inhibition of the Wnt/?-catenin signaling and reduced bone turnover in type 2 diabetes mellitus. in The Journal of clinical endocrinology and metabolism 2012
Rat (Rattus) CTNNB1 ELISA Kit for Sandwich ELISA - ABIN431880
Shao, Cai, Sheng, Yin: Role of SDF-1 and Wnt signaling pathway in the myocardial fibrosis of hypertensive rats. in American journal of translational research 2015
Mouse (Murine) CTNNB1 ELISA Kit for Sandwich ELISA - ABIN424163
Chen, Feng, Bao, Li, Zhang, Shen, Zhao, Guo, Jing, Lin, Zong: Adverse Effects of Osteocytic Constitutive Activation of ß-Catenin on Bone Strength and Bone Growth. in Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research 2015
temporal expression of CTNNB1 mRNA during dermal wound repair in the horse
These findings indicated that mechanical strain promoted osteoblastic differentiation through integrinbeta1mediated beta-catenin signaling.
CTNNB1 ablation suppresses melanoma growth by targeted deactivation of cancer-associated fibroblasts.
Wnt10a (show WNT10A ELISA Kits)/beta-catenin signaling pathway is able to exacerbate keloid cell proliferation and inhibit the apoptosis of keloid cells through its interaction with TERT (show TERT ELISA Kits).
a precise level of beta-catenin activity is essential for regulating the amplification and differentiation of muscle stem cells descendants during adult myogenesis.
Wnt (show WNT2 ELISA Kits)/beta-catenin signaling pathway abnormalities possibly play an important role in the development of cognitive deficits among mice exposed to chronic intermittent hypoxia.
Our findings highlight the critical roles of SIK1 (show SIK1 ELISA Kits) and its targets in the regulation of HCC (show FAM126A ELISA Kits) development and provides potential new candidates for HCC (show FAM126A ELISA Kits) therapy.
PAX5 (show PAX5 ELISA Kits) was found to be an epigenetically inactivated tumor suppressor that inhibited non-small-cell lung proliferation and metastasis, through down-regulating the beta-catenin pathway and up-regulating GADD45G (show GADD45G ELISA Kits) expression.
Data show athat beta-catenin can be activated by bone morphogenetic protein 9 (BMP9 (show GDF2 ELISA Kits)) and the activation of beta-catenin plays an important role in the differentiation of C3H10T1/2 cells into cardiomyocyte-like cells induced by BMP9 (show GDF2 ELISA Kits).
STAT3 (show STAT3 ELISA Kits)/Arid1b (show ARID1B ELISA Kits)/beta-catenin pathway is driving neurofibroma initiation.
stabilization of b-catenin in cholangiocyte precursors perturbed duct development and cholangiocyte differentiation. We conclude that b-catenin is dispensable for biliary development but that its activity must be kept within tight limits.
Mechanistically, Hif-3alpha2 binds to beta-catenin and destabilizes the nuclear beta-catenin complex.
Nuclear beta-catenin is an indication of an activated Wnt (show WNT2 ELISA Kits) pathway, therefore suggesting a possible role for Wnt (show WNT2 ELISA Kits) signalling during zebrafish tooth development and replacement.
Findings suggest that microRNA 19b (miR (show MYLIP ELISA Kits)-19b) regulates laterality development and heart looping in embryos by targeting beta-catenin ctnnb1.
Custos binds to beta-catenin in a Wnt (show WNT2 ELISA Kits) responsive manner without affecting its stability, but rather modulates the cytoplasmic to nuclear translocation of beta-catenin.
The role of the beta-catenin mechanosensitive pathway in mesoderm identity has been conserved over the large evolutionary distance separating zebrafish and Drosophila.
The Wnt (show WNT2 ELISA Kits)/beta-catenin signaling pathway establishes neuroanatomical asymmetries and their laterality. (Review)
This study demonstrated that beta-catenin/Wnt (show WNT2 ELISA Kits) signaling is initially required to activate cell-cycle re-entry in Muller glia following injury and that Wnt (show WNT2 ELISA Kits) signaling subsequently controls the fate of the progeny of those cell divisions.
A novel role for Eaf1 and Eaf2 (show EAF2 ELISA Kits) in inhibiting canonical Wnt (show WNT2 ELISA Kits)/beta-catenin signaling, which might form the mechanistic basis for Eaf1 and Eaf2 (show EAF2 ELISA Kits) tumor suppressor activity.
Ccr7 (show CCR7 ELISA Kits) functions during axis formation as a GPCR (show GPRC6A ELISA Kits) to inhibit beta-catenin, likely by promoting Ca(2 (show CA2 ELISA Kits)+) transients throughout the blastula.
ctnnb1 and ctnnb2 regulate multiple processes of laterality development in zebrafish embryos through similar and distinct mechanisms.
maternal Wnt (show WNT2 ELISA Kits)/STOP signaling, but not beta-catenin signaling, has a role in cleavage after fertilization and cell cycle progression
Wnt (show WNT2 ELISA Kits) signalling can benefit from nucleo-cytoplasmic shuttling of APC (show APC ELISA Kits), Axin (show AXIN1 ELISA Kits) and GSK3, although they are in general beta-catenin antagonising proteins.
Phosphoinositide 3-kinase and Rac (show AKT1 ELISA Kits) polarization depend specifically on the N-cadherin (show CDH2 ELISA Kits)-p120 catenin (show CTNND1 ELISA Kits) complex, whereas myosin II light chain and actin filament polarization depend on the N-cadherin (show CDH2 ELISA Kits)-beta-catenin complex.
HERG (show KCNH2 ELISA Kits) channel activity is stimulated by beta-catenin
Zic3 (show ZIC3 ELISA Kits) can suppress Wnt (show WNT2 ELISA Kits)/beta-catenin signaling and control development of the notochord and Spemann's organizer.
Notch initially destabilises beta-catenin in a process that does not depend on its phosphorylation by GSK3
The tyrosine kinase receptor (show KDR ELISA Kits), PTK7 (show PTK7 ELISA Kits), is implicated in beta-catenin-dependent developmental processes.
Kazrin (show KAZ ELISA Kits) interacts with ARVCF (show ARVCF ELISA Kits)-catenin, spectrin and p190B (show ARHGAP5 ELISA Kits) RhoGAP (show ARHGAP1 ELISA Kits), and modulates RhoA (show RHOA ELISA Kits) activity.
Activated Xenopus CTNNB1 regulates embryonic limb development via FGF signaling
Data suggest that a combined inhibition of mechanistic target of rapamycin (show FRAP1 ELISA Kits) protein (mTOR (show FRAP1 ELISA Kits)) and beta-catenin signaling transduction may be a therapeutic strategy for patients with chronic kidney.
p44 (show GTF2H2 ELISA Kits)/42 MAPK (show MAPK1 ELISA Kits) phosphorylation, the signaling pathway which is often regulated by beta-arrestins is not influenced by KLHL12 (show KLHL12 ELISA Kits), but seems to be exclusively mediated by Galphai protein upon dopamine D4 receptor (show DRD4 ELISA Kits) stimulation.
These results suggested that miR (show MLXIP ELISA Kits)-98 functioned as a potential tumor suppressor by regulating Wnt (show WNT2 ELISA Kits)/beta-catenin signal pathway through direct suppression of EZH2 (show EZH2 ELISA Kits) expression and might sever as a potential therapeutic target for HCC (show FAM126A ELISA Kits) patients.
Data indicate that clinical specimens showed a significant inverse correlation between zinc and ring finger 3 (ZNRF3) and beta-catenin mRNA levels.
Data show that overexpression of 4.1N (show EPB41L1 ELISA Kits) protein decreased expression of flotillin-1 (show FLOT1 ELISA Kits), decreased activation of beta-catenin/Wnt (show WNT2 ELISA Kits) pathway in non-small-cell lung cancer (NSCLC) cells.
Data show that evodiamine suppresses beta-catenin and vascular dndothelial growth factor A (show GTF3A ELISA Kits) (VEGFa (show VEGFA ELISA Kits)) in a hepatocellular carcinoma (HCC (show FAM126A ELISA Kits))cell line SMMC-7721 xenograft model.
miR (show MLXIP ELISA Kits)-93/ZNRF3/Wnt (show WNT2 ELISA Kits)/beta-catenin regulatory network contributes to the growth of lung carcinoma.
NLRC5 (show NLRC5 ELISA Kits) not only positively correlates with the increase of beta-catenin but also coordinates the activation of downstream Wnt (show WNT2 ELISA Kits)/beta-catenin signaling pathway, regulating cell proliferation, migration and invasion in hepatocellular carcinoma.
our findings first indicate that the interaction of HDGF (show HDGF ELISA Kits) and beta-catenin may play a crucial role in tumorigenesis of synovial sarcoma.
overexpression of beta-catenin promoted cisplatin resistance in OSCC in vitro and in vivo.
Flutamide exposure led to decreased beta-catenin in corpus luteum of mid/late pregnancy.
beta-catenin signaling is involved in formation of contractile membranes by dedifferentiated retinal pigment epithelium cells.
Wnt3a (show WNT3A ELISA Kits) produced a significant increase in heart valve interstitial cell number at day 4 and in the percentage of BrdU-positive nuclei at 24 h. The increase in proliferation was abolished by beta-catenin siRNA.
beta-catenin controls myocardin (show MYOCD ELISA Kits)-related transcription factor-dependent transcription and emerges as a critical regulator of an array of cytoskeletal genes
beta-Catenin plays a critical role in mediating TGF-beta1 (show TGFB1 ELISA Kits)-induced myofibroblast differentiation in aortic valve interstitial cells.
scratching-induced injury and repair of bronchial epithelial cells may involve inhibition of Glycogen synthase kinase 3beta activity which can lead to activation of the downstream signaling through beta-catenin
Data show that Wnt3a (show WNT3A ELISA Kits) can inhibit the adipogenic differentiation of porcine AMSCs, and suggest that Wnt (show WNT2 ELISA Kits)/beta-catenin signaling inhibits adipogenic differentiation potential and alters the cell fate from adipocytes to osteoblasts.
TNF-alpha (show TNF ELISA Kits) inhibits adipogenesis through stabilization of beta-catenin protein in porcine preadipocytes.
These results demonstrate that activation of AKT (show AKT1 ELISA Kits) is required for gonadotropin regulation of CTNNB1 accumulation and subsequent ovarian E2 production.
beta-catenin, a transcription factor activated by the canonical Wnt signaling pathway, was frequently detected in bovine herpesvirus 1 ORF2-positive trigeminal ganglionic neurons of latently infected, but not mock-infected, calves.
These data demonstrate for the first time that FSH (show BRD2 ELISA Kits) regulates CTNNB1 protein and WNT2 (show WNT2 ELISA Kits) mRNA expressions in bovine granulosa cells, suggesting a potential role of canonical WNT (show WNT2 ELISA Kits) signaling in ovarian steroidogenesis and follicular growth of cattle.
These data provide evidence that testosterone increases cellular beta-catenin content which promotes the expression of beta-catenin-targeted genes and myogenesis in the muscle-derived stem cells of cattle.
that beta-catenin is a plasma membrane-associated protein (show PDZK1IP1 ELISA Kits) in airway smooth muscle that regulates active tension development, presumably by stabilizing cell-cell contacts and thereby supporting force transmission between neighboring cells.
This study demonstrated a relationship between the timing of the development of in vitro-produced bovine embryos and the distribution and localization of the junction protein beta-catenin.
TGF-beta3 (show TGFB3 ELISA Kits) induces the chondrogenic differentiation of pericytes by inducing Wnt (show WNT2 ELISA Kits)/beta-catenin signaling and T-cell factor-induced gene transcription.
cAMP/PKA regulation of GSK3beta/beta-catenin signaling contributes to the increase in progesterone production in corpus luteum.
Palmatin effect on osteoarthritis is likely mediated via the Wnt (show WNT2 ELISA Kits)/beta-catenin and Hedgehog (show SHH ELISA Kits) signaling
These results collectively showed that 2-Deoxy-D-glucose regulates dedifferentiation via beta-catenin pathway in rabbit articular chondrocytes.
The protein encoded by this gene is part of a complex of proteins that constitute adherens junctions (AJs). AJs are necessary for the creation and maintenance of epithelial cell layers by regulating cell growth and adhesion between cells. The encoded protein also anchors the actin cytoskeleton and may be responsible for transmitting the contact inhibition signal that causes cells to stop dividing once the epithelial sheet is complete. Finally, this protein binds to the product of the APC gene, which is mutated in adenomatous polyposis of the colon. Mutations in this gene are a cause of colorectal cancer (CRC), pilomatrixoma (PTR), medulloblastoma (MDB), and ovarian cancer. Three transcript variants encoding the same protein have been found for this gene.
catenin beta 1 subunit
, catenin beta-1
, beta catenin 1
, catenin (cadherin-associated protein), beta 1, 88kDa
, catenin beta-1-like
, catenin (cadherin associated protein), beta 1, 88kDa
, catenin beta
, beta catenin