Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Actins are highly conserved proteins that are involved in various types of cell motility, and maintenance of the cytoskeleton. Additionally we are shipping Actin, gamma 1 Kits (34) and Actin, gamma 1 Proteins (19) and many more products for this protein.
Showing 10 out of 83 products:
Human Polyclonal ACTG1 Primary Antibody for EIA, WB - ABIN452715
de Heer, Huygen, Collin, Oostrik, Kremer, Cremers: Audiometric and vestibular features in a second Dutch DFNA20/26 family with a novel mutation in ACTG1. in The Annals of otology, rhinology, and laryngology 2009
Show all 3 references for ABIN452715
Human Polyclonal ACTG1 Primary Antibody for ICC, IF - ABIN153419
Watters, Kraja, Meucci, Province, McLeod: Genome-wide discovery of loci influencing chemotherapy cytotoxicity. in Proceedings of the National Academy of Sciences of the United States of America 2004
Show all 3 references for ABIN153419
Chicken Monoclonal ACTG1 Primary Antibody for ICC, IF - ABIN4278101
Tomasek, Haaksma, Schwartz, Howard: Whole animal knockout of smooth muscle alpha-actin does not alter excisional wound healing or the fibroblast-to-myofibroblast transition. in Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society 2013
Human Monoclonal ACTG1 Primary Antibody for PLA, ELISA - ABIN513091
Yang, Kwon, Choi, Park, Yang, Park, Ren, Chung, Kim, Park, Jeong, Lee: In-depth analysis of cysteine oxidation by the RBC proteome: advantage of peroxiredoxin II knockout mice. in Proteomics 2012
Human Polyclonal ACTG1 Primary Antibody for FACS, IF - ABIN390439
Liu, Li, Ren, Mao, Zhu, Zhu, Yang, Yuan, Liu, Wang, Liu: Novel ACTG1 mutation causing autosomal dominant non-syndromic hearing impairment in a Chinese family. in Journal of genetics and genomics = Yi chuan xue bao 2008
a novel mutation in ACTG1 was found to be co-segregated with hearing loss and the genetic cause of autosomal dominant nonsyndromic hearing impairment in a Chinese family
Three unrelated cases of rare ACTG1 variants in fetal microlissencephaly have been described.
In this study we sought to clarify the detailed mutational spectrum, clinical features, and genotype-phenotype correlations for ACTG1 gene associated autosomal dominant sensorineural hearing loss
Mutations in ACTB (show ACTB Antibodies) cause a distinctly more severe phenotype than ACTG1 mutations in Baraitser-Winter syndrome.
The data, for the first time, link ASAP3 (show ASAP3 Antibodies) with ACTG1 in the regulation of cytoskeletal maintenance and cell motility
The actin/MKL1 (show MKL1 Antibodies) signalling pathway influences cell growth and gene expression through large-scale chromatin reorganization and histone post-translational modifications.
the novel variant p.M305T in ACTG1 (DFNA20/26) was selected as a disease-causing variant.
These results showed the biphasic F-actin dynamics in herpes simplex virus 1 neuronal infection and confirmed the association of F-actin with the changes in the expression and activity of cofilin 1 (show CFL1 Antibodies).
identified de novo missense changes in the cytoplasmic actin-encoding genes ACTB (show ACTB Antibodies) and ACTG1 in one and two probands, respectively; suggest that Baraitser-Winter syndrome is the predominant phenotype associated with mutation of these two genes
knockdown of gamma-actin significantly reduced speed of motility and severely affected the cell's ability to explore, which was, in part, due to a loss of cell polarity
Data indicate that the complex formation of actin by thymosin beta4 is more likely to be flexible than rigid and is localized along the subdomains 1 to 3 of actin.
Splicing to include exon 3a is concomitant with previously described down-regulation of Actg1 in differentiating C2C12 cells
These data support an essential role for beta-actin (show ACTB Antibodies) in regulating cell migration and gene expression through control of the cellular G-actin (show ACTB Antibodies) pool
Results demonstrate overlapping developmental roles but unique post-developmental functions for beta-actin (show ACTB Antibodies) and gamma-actin in maintaining hair cell stereocilia.
Since the total amount of actin protein was maintained in Actg1(-/-) cells, our data suggests a distinct requirement for gamma(cyto)-actin in cell growth and survival.
arginylated gamma-actin, unlike beta-, was highly unstable and selectively ubiquitinated and degraded in vivo; instability was regulated by differences in nucleotide coding sequence between the actin isoforms, which conferred different translation rates
Eplin-alpha transcription is regulated by actin-MAL-SRF signalling.
Organisation of non-muscle gamma-actin and co-localisation with sarcomeric alpha-actinin (show ACTN1 Antibodies), a marker of sarcomere assembly and a major component of Z lines, was noted. Gamma-Actin was also found in young myotubes with developing sarcomeric myofibrils.
Our data demonstrate an important role for minimally abundant but strategically localized gamma(cyto)-actin in adult skeletal muscle and describe a new mouse model to study the in vivo relevance of subcellular actin isoform sorting.
G-actin regulates rapid induction of actin nucleation by mDia1 to restore cellular actin polymers.
Study uncovers a strong reciprocal interdependence between Nck and PI(4,5)P(2) in promoting localized N-WASp-mediated actin polymerization in cells.
Actins are highly conserved proteins that are involved in various types of cell motility, and maintenance of the cytoskeleton. In vertebrates, three main groups of actin isoforms, alpha, beta and gamma have been identified. The alpha actins are found in muscle tissues and are a major constituent of the contractile apparatus. The beta and gamma actins co-exist in most cell types as components of the cytoskeleton, and as mediators of internal cell motility. Actin, gamma 1, encoded by this gene, is a cytoplasmic actin found in non-muscle cells. Mutations in this gene are associated with DFNA20/26, a subtype of autosomal dominant non-syndromic sensorineural progressive hearing loss. Alternative splicing results in multiple transcript variants.
actin, cytoplasmic 2
, actin, gamma 1
, actin, cytoplasmic type 5
, actin, gamma 1 b
, actin, cytoplasmic 2-like
, cytoskeletal gamma-actin
, actin, gamma, cytoplasmic 1
, actin, cytoplasmic, type 5
, actin, gamma 1 propeptide
, cytoplasmic 2
, cytoplasmic type 5
, gamma 1 propeptide
, type 5
, cytoplasmic actin
, actin, cytoplasmic 2 a
, beta-actin FE-3