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Chemotactic factor. Additionally we are shipping CX3CL1 Kits (79) and CX3CL1 Proteins (74) and many more products for this protein.
Showing 10 out of 227 products:
Human Monoclonal CX3CL1 Primary Antibody for ELISA (Capture), FACS - ABIN4899898
Hundhausen, Schulte, Schulz, Andrzejewski, Schwarz, von Hundelshausen, Winter, Paliga, Reiss, Saftig, Weber, Ludwig: Regulated shedding of transmembrane chemokines by the disintegrin and metalloproteinase 10 facilitates detachment of adherent leukocytes. in Journal of immunology (Baltimore, Md. : 1950) 2007
Show all 6 references for ABIN4899898
Human Monoclonal CX3CL1 Primary Antibody for FACS - ABIN4896869
Truman, Ford, Pasikowska, Pound, Wilkinson, Dumitriu, Melville, Melrose, Ogden, Nibbs, Graham, Combadiere, Gregory: CX3CL1/fractalkine is released from apoptotic lymphocytes to stimulate macrophage chemotaxis. in Blood 2008
Show all 6 references for ABIN4896869
Human Polyclonal CX3CL1 Primary Antibody for EIA, WB - ABIN499690
Mizuno, Kawanokuchi, Numata, Suzumura: Production and neuroprotective functions of fractalkine in the central nervous system. in Brain research 2003
Show all 4 references for ABIN499690
Human Polyclonal CX3CL1 Primary Antibody for IP, IHC - ABIN223526
Yeo, Kim, Ryu, Seo, Lee, Choi, Kim, Kang: The roles of fractalkine/CX3CR1 system in neuronal death following pilocarpine-induced status epilepticus. in Journal of neuroimmunology 2011
Show all 2 references for ABIN223526
Human Polyclonal CX3CL1 Primary Antibody for ICC, IF - ABIN4300994
Schenkel, Fraser, Vezys, Masopust: Sensing and alarm function of resident memory CD8⁺ T cells. in Nature immunology 2013
Human Polyclonal CX3CL1 Primary Antibody for FACS, IF (p) - ABIN728488
Xie, Shi, Zhang, Zhang, Gong, Guo, Wang, Xu, Wang, Chen, Liu, Dong: Abnormal activation of microglia accompanied with disrupted CX3CR1/CX3CL1 pathway in the brains of the hamsters infected with scrapie agent 263K. in Journal of molecular neuroscience : MN 2013
Authors provide evidence that interactions between CX3CL1 and CX3CR1 (show CX3CR1 Antibodies) play crucial roles in determining the number of M1 macrophages within the skin of mice, which in turn can have dramatic effects on psoriasis-like inflammation.
In the absence of the rd8 allele, deficiency of CCR2 (show CCR2 Antibodies) and CX3CL1 in mice leads to a mild form of retinal degeneration which is associated with the recruitment of macrophages, particularly to the subretinal space. This model enables to assess consequences of perturbed chemokine (show CCL1 Antibodies) signaling, but it does not recapitulate cardinal (show CARD8 Antibodies) age-related macular degeneration features.
Also icariin reduced CX3CR1 (show CX3CR1 Antibodies) and CX3CL1 protein levels in the artery wall. In conclusion, icariin could be a potential anti-atherosclerosis agent by downregulating the expression of CX3CR1 (show CX3CR1 Antibodies).
The biological activity of CX3CL1 is regulated by conversion of a membrane integrated to a soluble form during neurogenesis and in response to pathologic changes in the adult retinal milieu.
CX3CL1/CX3CR1 (show CX3CR1 Antibodies) signaling is involved in LTP (show SCP2 Antibodies) of C-fiber-evoked field potentials in the rodent spinal dorsal horn
CX3CL1/CX3CR1-mediated microglial activation plays a detrimental role in ischemic brain via p38MAPK/PKC signaling
Cx3cl1 overexpression suppresses alpha-synuclein (show SNCA Antibodies)-mediated neurodegeneration.
IFN-gamma (show IFNG Antibodies) induces aberrant CD49b (show ITGA2 Antibodies)+ NK cell recruitment and pregnancy failure through regulating CX3CL1.
tumours are characterized by expression of inflammatory chemokines (CCL2 (show CCL2 Antibodies), CCL5 (show CCL5 Antibodies), CCL7 (show CCL7 Antibodies), CCL8 (show CCL8 Antibodies), CCL12 (show Ccl12 Antibodies), CXCL9 (show CXCL9 Antibodies), CXCL10 (show CXCL10 Antibodies) and CX3CL1), reflected by an enrichment of activated Foxp3 (show FOXP3 Antibodies)(-) and Foxp3 (show FOXP3 Antibodies)(+) T cells
Insulin (show INS Antibodies) resistance increases plaque vulnerability by augmenting the CX3CL1/CX3CR1 (show CX3CR1 Antibodies) axis, which is mechanistically linked to reduced vascular smooth muscle cell survival
CX3CL1 exerts numerous effects on pathophysiological conditions that have both negative and positive consequences on pathogenesis and outcome [review]
XCL2 (show XCL2 Antibodies) and CX3CL1 expression in lung cancers and adjacent non-cancerous tissues was detected by quantitative PCR and ELISA. The expression of XCL2 (show XCL2 Antibodies) and CX3CL1 increases with increasing degree of malignancy, indicating that both chemokines might be important targets in gene therapy for lung cancer.
Recent works show that, in allergic diseases, there is an increased expression of fractalkine/CX3CL1 and its unique receptor CX3CR1 (show CX3CR1 Antibodies) and that this chemokine (show CCL1 Antibodies) does not act as chemoattractant. In allergic asthma, CX3CR1 (show CX3CR1 Antibodies) expression regulates Th2 and Th1 (show TH1L Antibodies) cell survival in the inflammatory lung, while, in atopic dermatitis, it regulate Th2 and Th1 (show TH1L Antibodies) cell retention into the inflammatory site. [review]
Post-transcriptional suppression of KSRP (show KHSRP Antibodies) destabilizes CX3CL1 mRNA in liver epithelial cells.
CX3CL1 increased the migration and invasiveness of DU145 and PC-3 (show PCSK1 Antibodies), causing epithelial-to-mesenchymal transition. TACE (show ADAM17 Antibodies)/TGF-alpha (show TGFA Antibodies)/EGFR (show EGFR Antibodies) pathway activation and Slug (show SNAI2 Antibodies) upregulation were responsible for this.
Cell proliferation enhancing and anti-apoptosis activity requires the intracellular domain and apparently the dimerization of the transmembrane chemokine (show CCL1 Antibodies) ligand.
CX3CL1 was expressed only by the normal and cancer adjacent normal fallopian tube epithelium; its expression was largely lost in the malignant fallopian epithelium.
Skin and serum CX3CL1 elevated expression was associated with psoriasis severity.
CX3CL1(+) apo (show C9orf3 Antibodies)-MPs released by apoptotic cells mediate the chemotactic transmigration of alveolar macrophages.
Suggest that CX3CL1 participates in cross-talk mechanisms between endothelium and other muscle tissue cells and may promote a shift in the microenvironment toward a more regenerative milieu after exercise.
Chemotactic factor. Binds to CX3CR1.
, chemokine (C-X3-C motif) ligand 1
, chemokine (C-X3-C motif) ligand 1, related sequence 1
, small inducible cytokine subfamily D, 1-related sequence 1
, CX3CL1 chemokine
, C-X3-C motif chemokine 1
, CX3C membrane-anchored chemokine
, small inducible cytokine subfamily D, 1
, small-inducible cytokine D1
, small inducible cytokine subfamily D (Cys-X3-Cys), member 1 (fractalkine, neurotactin)
, small inducible cytokine subfamily D (Cys-X3-Cys), member-1