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neuropeptide arginine vasopressin receptor\; may have a role in the central nervous system [RGD, Feb 2006].. Additionally we are shipping Cullin 5 Kits (10) and Cullin 5 Proteins (5) and many more products for this protein.
Showing 10 out of 62 products:
Human Polyclonal Cullin 5 Primary Antibody for EIA, IHC (p) - ABIN117951
Ng, Yu, Lai, Lau: Biliary Complications Associated with Selective Internal Radiation (SIR) Therapy for Unresectable Liver Malignancies. in Digestive diseases and sciences 2008
Show all 9 references for ABIN117951
Human Polyclonal Cullin 5 Primary Antibody for EIA, WB - ABIN359745
Kamura, Burian, Yan, Schmidt, Lane, Querido, Branton, Shilatifard, Conaway, Conaway: Muf1, a novel Elongin BC-interacting leucine-rich repeat protein that can assemble with Cul5 and Rbx1 to reconstitute a ubiquitin ligase. in The Journal of biological chemistry 2001
Show all 3 references for ABIN359745
Dog (Canine) Polyclonal Cullin 5 Primary Antibody for WB - ABIN2776157
Burnatowska-Hledin, Kossoris, Van Dort, Shearer, Zhao, Murrey, Abbott, Kan, Barney: T47D breast cancer cell growth is inhibited by expression of VACM-1, a cul-5 gene. in Biochemical and biophysical research communications 2004
cullin-5 and cullin-2 (show CUL2 Antibodies) play a role in the development of neuromuscular junction and the female germ line of Drosophila
Data show that the follicular epithelium that normally encapsulates single germ line cysts develops aberrantly in cul-5 mutant.
cul-5 plays multiple roles in cell fate specification and synapse formation during Drosophila development.
These results provide important information on the assembly of the Vif (show BTG1 Antibodies)-CUL5-E3 ubiquitin ligase (show MUL1 Antibodies) and identify a new viV binding interface with CBF-beta (show CBFB Antibodies) at the C-terminus of HIV-1 Vif (show BTG1 Antibodies).
Data indicate that cell surface adhesion molecules VCAM-1 (show VCAM1 Antibodies) and P-selectin (show SELP Antibodies) play some roles in mechanical stretch-induced HL-60 cell adhesion to mouse common carotid arteries.
N-terminal mutants of HIV-1 vif (show BTG1 Antibodies) that demonstrated reduced Cul5 binding were also unable to degrade APOBEC3G (show APOBEC3G Antibodies) as well as APOBEC3F (show APOBEC3F Antibodies).
A tumor suppressor gene, CUL5, is identified as a direct target of miR-7 (show LILRB1 Antibodies) in hepatocellular carcinoma.
Silencing CUL5 reduced cellular sensitivity to three distinct HSP90 (show HSP90 Antibodies) inhibitors, across four cancer types driven by different protein kinases.
In the absence of CBFbeta (show CBFB Antibodies), Vif (show BTG1 Antibodies) does not bind Cul5, thus preventing the assembly of the E3 ligase complex.
CBF-beta (show CBFB Antibodies) is critical for the formation of the Vif (show BTG1 Antibodies)-ElonginB/ElonginC-Cul5 core E3 ubiquitin ligase (show MUL1 Antibodies) complex.
data reveal the structural basis for Vif (show BTG1 Antibodies) hijacking of the CBF-beta (show CBFB Antibodies) and CUL5 E3 ligase complex, laying a foundation for rational design of novel anti-HIV drugs
ASB9 (show ASB9 Antibodies) is unstable alone but forms a stable ternary complex with EloBC that binds with high affinity to the Cullin 5 N-terminal domain.
crystal structure shows interaction between SOCS2 (show SOCS2 Antibodies)-elongin BC and Cullin-5
Our results show that endogenous Cul5 suppresses epithelial cell transformation by several pathways, including inhibition of Src (show SRC Antibodies)-Cas (show CTNND1 Antibodies)-induced ruffling through SOCS6 (show SOCS6 Antibodies)
The expression of cullin5 on retinal cells and reduction in the percentage of dead retinal cells induced by L-glutamate (show GRIN1 Antibodies) suggest that cullin5 has neuroprotective properties in retinal cells.
SOCS (show CISH Antibodies) box acts as an independent binding domain capable of recruiting elonginBC and cullin5 to promote E3 ligase formation.
The role of Cul5 is to degrade disabled homolog 1 (Dab1 (show DAB1 Antibodies)) and thus control neuron migration speed and neuron insertion at the top of the cortical plate.
These results suggest that, when Src (show SRC Antibodies) is activated by mutation or physiological mechanisms, its effects are limited by Culllin 5, which downregulates active Src (show SRC Antibodies) and its phosphorylated substrates.
data imply the existence of two distinct subclasses of SOCS (show CISH Antibodies) proteins with a high affinity for Cullin5, the E3 ligase scaffold, possibly reflecting complete dependence upon ubiquitination for suppression of cytokine signalling
The study results suggest that VACM-1 may regulate endothelial AQP1 concentration both in vivo and in vitro.
oocyte maturation from pachytene exit and MPK-1 (show MAPK1 Antibodies) activation are redundantly controlled by the RBX-2-CUL-5- and RBX-1 (show RBX1 Antibodies)-CUL-2 (show CUL2 Antibodies)-based complexes.
neuropeptide arginine vasopressin receptor\; may have a role in the central nervous system
Vasopressin-activated calcium-mobilizing receptor (VACM-1)
, cullin 5
, Vasopressin-activated calcium-mobilizing receptor-1
, Cullin-5 (vasopressin-activated calcium-mobilizing receptor-1)
, vasopressin-activated calcium-mobilizing receptor 1
, vasopressin-activated calcium-mobilizing receptor protein
, vasopressin-activated calcium-mobilizing protein