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Transcriptional activator essential for osteoblast differentiation.
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OSX served a key role in the development and progression of ALD (show ABCD1 ELISA Kits)-induced VSMC calcification. This observation may aid in the explanation of the role of OSX in the pathogenesis of vascular calcification
results suggest that expression of Sp7 during the early stage of Satb2 (show SATB2 ELISA Kits)-induced osteogenic differentiation of BMSCs is regulated by miR (show MLXIP ELISA Kits)-27a.
Fibrillin-2 (show FBN2 ELISA Kits) and periostin (show POSTN ELISA Kits) are target genes in Osterix-mediated osteoblast differentiation.
osterix is a downstream target of IGF1R (show IGF1R ELISA Kits) in chondrocytes.
cells expressing osterix are mesenchymal progenitors contributing to all relevant cell types during morphogenesis.
Results propose that Utx (show KDM6A ELISA Kits) plays important roles in osteoblast differentiation by controlling the expressions of Runx2 (show RUNX2 ELISA Kits) and Osterix.
Decreased expression of Osterix, as well as impaired TGFbeta and BMP2 signaling, contribute to the observed osteopenic bone phenotype of TIEG1 KO mice.
Intermittent stretching promotes osteogenic differentiation of bone marrow derived mesenchymal stem cells; the p38MAPK (show MAPK14 ELISA Kits)-osterix pathway has an important role in the control of osteogenesis related gene expression.
c-Src (show SRC ELISA Kits) signaling modulates osteoblast differentiation at least in part through phosphorylation of Osterix.
CHIP targets Osx for ubiquitination and degradation in osteoblasts after chronic exposure to TNF-alpha (show TNF ELISA Kits).
Transcriptional activator essential for osteoblast differentiation. Binds to SP1 and EKLF consensus sequences and to other G/C-rich sequences.
, trans-acting transcription factor 7
, transcription factor Sp7
, zinc finger protein osterix