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Browse our anti-ENOS (NOS3) Antibodies

Full name:
anti-Nitric Oxide Synthase 3 (Endothelial Cell) Antibodies (NOS3)
On www.antibodies-online.com are 460 Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Antibodies from 30 different suppliers available. Additionally we are shipping ENOS Kits (81) and ENOS Proteins (19) and many more products for this protein. A total of 574 ENOS products are currently listed.
Synonyms:
2310065A03Rik, cNOS, EC-NOS, ecNOS, ENOS, NOS, Nos-3, NOS3, NOSIII

All available anti-ENOS Antibodies

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Top referenced anti-ENOS Antibodies

  1. Amphibian Polyclonal ENOS Primary Antibody for IHC (fro), IHC (p) - ABIN152659 : Guo, Comhair, Zheng, Dweik, Eissa, Thomassen, Calhoun, Erzurum: Molecular mechanisms of increased nitric oxide (NO) in asthma: evidence for transcriptional and post-translational regulation of NO synthesis. in Journal of immunology (Baltimore, Md. : 1950) 2000 (PubMed)
    Show all 7 references for ABIN152659

  2. Human Polyclonal ENOS Primary Antibody for EIA, FACS - ABIN953739 : Yanamandra, Napper, Pramanik, Bocchini, Dhanireddy: Endothelial nitric oxide synthase genotypes in the etiology of retinopathy of prematurity in premature infants. in Ophthalmic genetics 2010 (PubMed)
    Show all 4 references for ABIN953739

  3. Human Polyclonal ENOS Primary Antibody for IF, WB - ABIN319319 : Wadman: GM advisory panel is slanted, say critics. in Nature 1999 (PubMed)
    Show all 3 references for ABIN319319

  4. Human Polyclonal ENOS Primary Antibody for IF - ABIN401626 : Moreno: Genetic polymorphisms and haplotypes of eNOS in breast cancer. in Breast cancer research and treatment 2008 (PubMed)
    Show all 3 references for ABIN401626

  5. Human Polyclonal ENOS Primary Antibody for IF (p), IHC (p) - ABIN746468 : Ikemura, Yamamoto, Motomura, Yamaguchi, Zhao, Iwasaki, Iwamoto: Preventive effects of the anti-vasospasm agent via the regulation of the Rho-kinase pathway on the development of steroid-induced osteonecrosis in rabbits. in Bone 2013 (PubMed)
    Show all 2 references for ABIN746468

  6. Human Polyclonal ENOS Primary Antibody for FACS, IF - ABIN655773 : Ren, Sun, Deng, Zhang, Wu, Wei, Mani, Dou, Wang: The anti-inflammatory effect and potential mechanism of cardamonin in DSS-induced colitis. in American journal of physiology. Gastrointestinal and liver physiology 2015 (PubMed)
    Show all 2 references for ABIN655773

  7. Dog (Canine) Monoclonal ENOS Primary Antibody for WB - ABIN968916 : Michell, Chen Zp, Tiganis, Stapleton, Katsis, Power, Sim, Kemp: Coordinated control of endothelial nitric-oxide synthase phosphorylation by protein kinase C and the cAMP-dependent protein kinase. in The Journal of biological chemistry 2001 (PubMed)

  8. Human Polyclonal ENOS Primary Antibody for EIA, WB - ABIN453769 : Rikova, Guo, Zeng, Possemato, Yu, Haack, Nardone, Lee, Reeves, Li, Hu, Tan, Stokes, Sullivan, Mitchell, Wetzel, Macneill, Ren, Yuan, Bakalarski, Villen, Kornhauser, Smith, Li, Zhou, Gygi, Gu et al.: Global survey of phosphotyrosine signaling identifies oncogenic kinases in lung cancer. ... in Cell 2007 (PubMed)

  9. Human Polyclonal ENOS Primary Antibody for EIA - ABIN453267 : Greif, Kou, Michel: Site-specific dephosphorylation of endothelial nitric oxide synthase by protein phosphatase 2A: evidence for crosstalk between phosphorylation sites. in Biochemistry 2002 (PubMed)

  10. Human Polyclonal ENOS Primary Antibody for IF (p), IHC (p) - ABIN703315 : Papinska, Mordwinkin, Meeks, Jadhav, Rodgers: Angiotensin-(1-7) administration benefits cardiac, renal and progenitor cell function in db/db mice. in British journal of pharmacology 2015 (PubMed)

More Antibodies against ENOS Interaction Partners

Human Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. Our results demonstrated that GRP94 (show HSP90B1 Antibodies) is a key molecule in Hepatocellular carcinoma (HCC (show FAM126A Antibodies)) progression that modulates the AKT (show AKT1 Antibodies) pathway and eNOS levels

  2. eNOS-T786C and PAI-1 (show SERPINE1 Antibodies)(4G/5G) are important polymorphisms in developing Buerger's disease

  3. eNOS G894T polymorphism might protect men against erectile dysfunction risk.

  4. The G894T eNOS polymorphism was associated with increased intima-media thickness in the right carotid in patients with systemic sclerosis.

  5. The findings of the present study suggest that polymorphism in G894T position of eNOS gene might be a risk factor for ischemic stroke mainly for large vessel disease stroke subtype in North Indian population

  6. We analyzed TNFalpha (show TNF Antibodies), NOS 3 (show NANOS3 Antibodies) and MDR1 (show TBC1D9 Antibodies) in 77 patients with MM and 77 healthy controls. The genotyping was performed with PCR and/or PCR-RFLP.

  7. We investigated NOS3 (show NANOS3 Antibodies) T-786C, G894T, and 4b/a polymorphisms in 102 patients with varicocele and 100 healthy controls. The 4b/a polymorphism may have a protective effect for varicocelem and G894T polymorphism may contribute to varicocele occurrence by lowering the level of NO. The higher NOS3 (show NANOS3 Antibodies) expression levels in the patient group may be a kind of dilator compensatory mechanism to protect vascular anatomy in varicocele.

  8. Results suggest that variants in intron 4 of NOS3 (show NANOS3 Antibodies), with pre-transcriptional effects as susceptibility factors, influencing the risk thalidomide embryopathy development.

  9. Results suggested that G894T and T-786C polymorphisms of the nitric oxide synthase (show NOS Antibodies) 3 gene, but not VNTR 4b/a, were associated with an increased risk of preeclampsia.

  10. Uric acid induced HUVEC apoptosis and endothelial dysfunction by triggering oxidative and endoplasmic reticulum stress through protein kinase C (show PKC Antibodies)/eNOS-mediated eNOS activity and NO production.

Pig (Porcine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. NOS3 was lowest in kidneys removed from live pigs, greater in kidneys from pigs with brain death, and greatest in kidneys from pigs with cardiac arrest.

  2. Rapid atrial pacing increases ADMA and down-regulates eNOS expression in an ADMA-independent manner.

  3. Icariin and icariside II may increase the eNOS expression through activating EGF-EGFR (show EGFR Antibodies) pathway in porcine aortic endothelial cells.

  4. Data suggest that pig sperm contain bNOS (show NOS1 Antibodies), iNOS (show NOS2 Antibodies), and eNOS; up-regulation of NOS (show NOS Antibodies) by leptin (show LEP Antibodies) during acrosome reaction and inhibition of acrosome reaction by inhibitors of nitric oxide synthases suggests these enzymes are involved in acrosome reaction.

  5. Periodic acceleration (pGz) acutely increases endothelial and neuronal nitric oxide synthase (show NOS1 Antibodies) expression in endomyocardium of normal swine.

  6. Data suggest that angiotensin II regulates nNOS (show NOS1 Antibodies) and eNOS expression and NOS (show NOS Antibodies) activity in afferent arterioles of the developing kidney via angiotensin 1 and 2 receptors.

  7. Endothelial nitric oxide synthase mRNA expression was elevated in gestational day 50 intrauterine growth retardation placenta and areola compared to gd50 control.

  8. Oligonol prevented the impairment of eNOS activity induced by high glucose through reversing altered eNOS phosphorylation status.

  9. Exercise training significantly increased total eNOS and phosphorylated levels of eNOS (pSer(1179)) in collateral-dependent arteries of experimental minipigs.

  10. Data show that wall shear stress increases with a decrease in artery diameter; eNOS protein contents decrease with an increase in diameter.

Cow (Bovine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. Pomanox supplementation hinders hyperlipemia-induced coronary endothelial dysfunction by activating the Akt (show AKT1 Antibodies)/endothelial nitric oxide-synthase pathway and favorably counteracting vascular inflammation and oxidative damage.

  2. Signals that activate and phosphorylate eNOS are transmitted through distinct membrane domains in endothelial cells. Cholesterol domains, but not individual caveolae, mediate HDL stimulation of eNOS. VEGF and shear stress may act through caveolae.

  3. eNOS serine 1179 phosphorylation, in addition to enhancing NO production, also profoundly affects superoxide generation

  4. In addition to the heme center of eNOS oxygenase domain, we confirmed another O2.- generation site in the eNOS reductase domain and characterized its regulatory properties.

  5. A dimer-destabilized mutant of bovine eNOS where cysteine 101 was replaced by alanine, cloned and introduced into an eNOS-deficient mouse strain and that results provide the first in vivo evidence that eNOS-dependent oxidative stress is unlikely to be an initial cause of impaired endothelium-dependent vasodilation and/or a pathologic factor promoting intimal hyperplasia.

  6. Data show that resveratrol (Res) reversed caveolin-1 (Cav-1 (show CAV1 Antibodies))/endothelial nitric oxide synthase (eNOS) expressions in high glucose cultured bovine aortic endothelial cells (BAECs).

  7. Fluorescence decays of fluorescently labeled CaM (show KRIT1 Antibodies) bound to eNOS reveal four distinct conformational states and single-molecule fluorescence trajectories show multiple fluorescence states with transitions between states

  8. Endothelial nitric oxide synthase is regulated by ERK (show MAPK1 Antibodies) phosphorylation at Ser602.

  9. key regulatory role of CaM (show KRIT1 Antibodies) involves the stabilization of structural intermediates and precise positioning of the pivot for the FMN (show FMN1 Antibodies) domain tethered shuttling motion to accommodate efficient and rapid electron transfer in the homodimer of eNOS.

  10. radiation-induced eNOS activation in bovine aortic endothelial cells is regulated by ATM (show ATM Antibodies) and HSP90 (show HSP90 Antibodies)

Rabbit Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. VEGFR2 (show KDR Antibodies) activation was not affected by Slit2 (show SLIT2 Antibodies), but eNOS phosphorylation was diminished

  2. Data suggest distinct localizations of eNOS at the spiral arteries/arterial sinuses and iNOS (show NOS2 Antibodies) along the radial arteries in the developing placenta.

  3. Pulmonary ischaemia-reperfusion up-regulates inducible nitric oxide synthesis and/activity, which coincides with reduced endothelial nitric oxide synthase activity.

  4. eNOS dysregulation may be a central mechanism of impaired cardioprotection during hyperglycemia.

  5. Quercetin inhibited myocardial ischemia-reperfusion-induced NOS3 mRNA and protein expression.

Mouse (Murine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. Beta2AR (show ADRB2 Antibodies) overexpression enhances endothelial progenitor cells (EPC (show TCF21 Antibodies)) functions in vitro and enhances the vascular repair abilities of EPCs in vivo via the beta2AR (show ADRB2 Antibodies)/Akt (show AKT1 Antibodies)/eNOS pathway.

  2. Through the abrogation of NAD(P)H (show NQO1 Antibodies) oxidase-driven eNOS uncoupling.

  3. The in vitro concordance of early Nos3(-/-) disease signatures supports the utility of iPSCs as a cellular model of developmental heart defects

  4. Findings do not support the hypothesis that reduced NO production from eNOS contributes to obesity-related adipose tissue inflammation.

  5. Normal and high eNOS levels are detrimental in both mild and severe cardiac pressure-overload.

  6. eNOS may be necessary to maintain podocyte integrity, especially mitochondrial function

  7. Disruption of the eNOS-NO signaling pathway exacerbates peritoneal fibrosis by delaying wound healing.

  8. BMP4 (show BMP4 Antibodies) has a role in inducing NOX1 (show NOX1 Antibodies)-dependent eNOS uncoupling in T2DM, which may promote development of novel therapeutics restoring endothelial function in T2DM

  9. The eNOS expression was induced with adipocyte differentiation and inhibition of eNOS/NO enhanced lipolysis in vitro and in vivo.

  10. Diet-induced obesity leads to l-arginine (show GATM Antibodies) deficiency and eNOS uncoupling in perivascular adipose tissue.

Guinea Pig Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. The present evidence indicated that the customary HBOT protocol may increase constitutive NOS expression.

ENOS (NOS3) Antigen Profile

Antigen Summary

Nitric oxide is a reactive free radical which acts as a biologic mediator in several processes, including neurotransmission and antimicrobial and antitumoral activities. Nitric oxide is synthesized from L-arginine by nitric oxide synthases. Variations in this gene are associated with susceptibility to coronary spasm. Multiple transcript variants encoding different isoforms have been found for this gene.

Alternative names and synonyms associated with ENOS (NOS3)

  • nitric oxide synthase 3 (endothelial cell) (NOS3) antibody
  • nitric oxide synthase 3 (endothelial cell) (nos3) antibody
  • endothelial nitric oxide synthase (23B) antibody
  • nitric oxide synthase 3, endothelial cell (Nos3) antibody
  • 2310065A03Rik antibody
  • cNOS antibody
  • EC-NOS antibody
  • ecNOS antibody
  • ENOS antibody
  • NOS antibody
  • Nos-3 antibody
  • NOS3 antibody
  • NOSIII antibody

Protein level used designations for NOS3

nitric oxide synthase 3 (endothelial cell) , nitric oxide synthase, endothelial-like , EC-NOS , NOS type III , NOSIII , cNOS , constitutive NOS , endothelial NOS , nitric oxide synthase, endothelial , eNOS , endothelial nitric oxide synthase , endothelial nitric oxide synthase NOS3 , endothelial nitric oxide synthase 3

GENE ID SPECIES
714231 Macaca mulatta
100018035 Monodelphis domestica
100486016 Xenopus (Silurana) tropicalis
443077 Ovis aries
4846 Homo sapiens
403784 Canis lupus familiaris
397557 Sus scrofa
287024 Bos taurus
443073 Ovis aries
100063339 Equus caballus
100009498 Oryctolagus cuniculus
18127 Mus musculus
24600 Rattus norvegicus
100135577 Cavia porcellus
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