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Browse our anti-ENOS (NOS3) Antibodies

Full name:
anti-Nitric Oxide Synthase 3 (Endothelial Cell) Antibodies (NOS3)
On are 469 Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Antibodies from 30 different suppliers available. Additionally we are shipping ENOS Kits (92) and ENOS Proteins (23) and many more products for this protein. A total of 598 ENOS products are currently listed.
2310065A03Rik, cNOS, EC-NOS, ecNOS, ENOS, NOS, Nos-3, NOS3, NOSIII

All available anti-ENOS Antibodies

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Top referenced anti-ENOS Antibodies

  1. Amphibian Polyclonal ENOS Primary Antibody for IHC (fro), IHC (p) - ABIN152659 : Guo, Comhair, Zheng, Dweik, Eissa, Thomassen, Calhoun, Erzurum: Molecular mechanisms of increased nitric oxide (NO) in asthma: evidence for transcriptional and post-translational regulation of NO synthesis. in Journal of immunology (Baltimore, Md. : 1950) 2000 (PubMed)
    Show all 7 references for ABIN152659

  2. Human Polyclonal ENOS Primary Antibody for EIA, FACS - ABIN953739 : Yanamandra, Napper, Pramanik, Bocchini, Dhanireddy: Endothelial nitric oxide synthase genotypes in the etiology of retinopathy of prematurity in premature infants. in Ophthalmic genetics 2010 (PubMed)
    Show all 4 references for ABIN953739

  3. Human Polyclonal ENOS Primary Antibody for IF, WB - ABIN319319 : Wadman: GM advisory panel is slanted, say critics. in Nature 1999 (PubMed)
    Show all 3 references for ABIN319319

  4. Human Polyclonal ENOS Primary Antibody for IF - ABIN401626 : Moreno: Genetic polymorphisms and haplotypes of eNOS in breast cancer. in Breast cancer research and treatment 2008 (PubMed)
    Show all 3 references for ABIN401626

  5. Human Monoclonal ENOS Primary Antibody for IHC, ELISA - ABIN969097 : Dimmeler, Fleming, Fisslthaler, Hermann, Busse, Zeiher: Activation of nitric oxide synthase in endothelial cells by Akt-dependent phosphorylation. in Nature 1999 (PubMed)
    Show all 3 references for ABIN969097

  6. Human Polyclonal ENOS Primary Antibody for IF, IHC (p) - ABIN319231 : Harris, Ju, Venema, Liang, Zou, Michell, Chen, Kemp, Venema: Reciprocal phosphorylation and regulation of endothelial nitric-oxide synthase in response to bradykinin stimulation. in The Journal of biological chemistry 2001 (PubMed)
    Show all 3 references for ABIN319231

  7. Human Polyclonal ENOS Primary Antibody for FACS, IF - ABIN655773 : Ren, Sun, Deng, Zhang, Wu, Wei, Mani, Dou, Wang: The anti-inflammatory effect and potential mechanism of cardamonin in DSS-induced colitis. in American journal of physiology. Gastrointestinal and liver physiology 2015 (PubMed)
    Show all 2 references for ABIN655773

  8. Dog (Canine) Monoclonal ENOS Primary Antibody for WB - ABIN968916 : Michell, Chen Zp, Tiganis, Stapleton, Katsis, Power, Sim, Kemp: Coordinated control of endothelial nitric-oxide synthase phosphorylation by protein kinase C and the cAMP-dependent protein kinase. in The Journal of biological chemistry 2001 (PubMed)

  9. Human Polyclonal ENOS Primary Antibody for EIA, WB - ABIN453769 : Rikova, Guo, Zeng, Possemato, Yu, Haack, Nardone, Lee, Reeves, Li, Hu, Tan, Stokes, Sullivan, Mitchell, Wetzel, Macneill, Ren, Yuan, Bakalarski, Villen, Kornhauser, Smith, Li, Zhou, Gygi, Gu et al.: Global survey of phosphotyrosine signaling identifies oncogenic kinases in lung cancer. ... in Cell 2007 (PubMed)

  10. Human Polyclonal ENOS Primary Antibody for EIA - ABIN453267 : Greif, Kou, Michel: Site-specific dephosphorylation of endothelial nitric oxide synthase by protein phosphatase 2A: evidence for crosstalk between phosphorylation sites. in Biochemistry 2002 (PubMed)

More Antibodies against ENOS Interaction Partners

Human Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. eNOS does not play an important role in this cerebrocortical blood flow redistribution after carotid artery occlusion.

  2. there were significant decreases in intercellular adhesion molecules 1 (ICAM1 (show ICAM1 Antibodies)), ezrin (EZR (show EZR Antibodies)), mitogen-activated protein kinase kinase 2 (MAP2K2 (show MAP2K2 Antibodies)), and nitric oxide synthase (show NOS Antibodies) 3 (NOS3 (show NANOS3 Antibodies)) gene expressions in metabolic syndrome patients.

  3. Activation of eNOS by HDL (show HSD11B1 Antibodies) is decreased in MetS (show ETV3 Antibodies) patients before the appearance of diabetes mellitus and that sphingosine-1-phosphate depletion of HDL (show HSD11B1 Antibodies) is the main factor responsible for this defect.

  4. The thrombotic predisposition to arterial ischemia of the lower limbs in type 2 diabetic patients is assessed by evaluating polymorphisms in the three major platelet membrane adhesion receptors and endothelial nitric oxide synthase.

  5. In conclusion, exenatide significantly improves coronary endothelial function in patients with newly diagnosed type 2 diabetes. The effect may be mediated through activation of AMPK (show PRKAA1 Antibodies)/PI3K (show PIK3CA Antibodies)-Akt (show AKT1 Antibodies)/eNOS pathway via a GLP-1R (show GLP1R Antibodies)/cAMP-dependent mechanism.

  6. The downregulation of beta-arrestins 1/2 in saphenous vein endothelial cells (SVECs) prevented the shear stress-induced rise in levels of phosphorylation of Akt (show AKT1 Antibodies) and endothelial nitric oxide synthase (eNOS, Serine 1177).

  7. PGC-1alpha (show PPARGC1A Antibodies) overexpression improves AngII-induced eNOS dysfunction via activated PI3K (show PIK3CA Antibodies)/Akt (show AKT1 Antibodies) pathway, impaired PP2A (show PPP2R4 Antibodies) activity and reduced PP2A (show PPP2R4 Antibodies)-A/eNOS association.

  8. eNOS G894T and T-786C SNPs were both significantly correlated with hypertension. Additionally, the T allele of G894T SNP and C allele of T-786C SNP may serve as potential biological markers for hypertension susceptibility in Asians. [Meta-Analysis}

  9. Results suggest a relation between different endothelial nitric oxide synthase geno-types and risk of acute rejection episodes in liver transplant recipients.

  10. The presence of the eNOS allele had a significantly negative impact on responsiveness to a selective alpha1 -blocker in benign prostatic hyperplasia related lower urinary tract symptoms treatment, suggesting that eNOS G894T gene polymorphism may be a genetic susceptibility factor for alpha1 -blocker efficacy in men with benign prostatic hyperplasia related lower urinary tract symptoms

Pig (Porcine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. NOS3 was lowest in kidneys removed from live pigs, greater in kidneys from pigs with brain death, and greatest in kidneys from pigs with cardiac arrest.

  2. Rapid atrial pacing increases ADMA and down-regulates eNOS expression in an ADMA-independent manner.

  3. Icariin and icariside II may increase the eNOS expression through activating EGF-EGFR (show EGFR Antibodies) pathway in porcine aortic endothelial cells.

  4. Data suggest that pig sperm contain bNOS (show NOS1 Antibodies), iNOS (show NOS2 Antibodies), and eNOS; up-regulation of NOS (show NOS Antibodies) by leptin (show LEP Antibodies) during acrosome reaction and inhibition of acrosome reaction by inhibitors of nitric oxide synthases suggests these enzymes are involved in acrosome reaction.

  5. Periodic acceleration (pGz) acutely increases endothelial and neuronal nitric oxide synthase (show NOS1 Antibodies) expression in endomyocardium of normal swine.

  6. Data suggest that angiotensin II regulates nNOS (show NOS1 Antibodies) and eNOS expression and NOS (show NOS Antibodies) activity in afferent arterioles of the developing kidney via angiotensin 1 and 2 receptors.

  7. Endothelial nitric oxide synthase mRNA expression was elevated in gestational day 50 intrauterine growth retardation placenta and areola compared to gd50 control.

  8. Oligonol prevented the impairment of eNOS activity induced by high glucose through reversing altered eNOS phosphorylation status.

  9. Exercise training significantly increased total eNOS and phosphorylated levels of eNOS (pSer(1179)) in collateral-dependent arteries of experimental minipigs.

  10. Data show that wall shear stress increases with a decrease in artery diameter; eNOS protein contents decrease with an increase in diameter.

Cow (Bovine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. TNFalpha (show TNF Antibodies)reduces eNOS activity in bovine endothelial cells through serine 116 phosphorylation and Pin1 binding.

  2. Pomanox supplementation hinders hyperlipemia-induced coronary endothelial dysfunction by activating the Akt (show AKT1 Antibodies)/endothelial nitric oxide-synthase pathway and favorably counteracting vascular inflammation and oxidative damage.

  3. Signals that activate and phosphorylate eNOS are transmitted through distinct membrane domains in endothelial cells. Cholesterol domains, but not individual caveolae, mediate HDL stimulation of eNOS. VEGF and shear stress may act through caveolae.

  4. eNOS serine 1179 phosphorylation, in addition to enhancing NO production, also profoundly affects superoxide generation

  5. In addition to the heme center of eNOS oxygenase domain, we confirmed another O2.- generation site in the eNOS reductase domain and characterized its regulatory properties.

  6. A dimer-destabilized mutant of bovine eNOS where cysteine 101 was replaced by alanine, cloned and introduced into an eNOS-deficient mouse strain and that results provide the first in vivo evidence that eNOS-dependent oxidative stress is unlikely to be an initial cause of impaired endothelium-dependent vasodilation and/or a pathologic factor promoting intimal hyperplasia.

  7. Data show that resveratrol (Res) reversed caveolin-1 (Cav-1 (show CAV1 Antibodies))/endothelial nitric oxide synthase (eNOS) expressions in high glucose cultured bovine aortic endothelial cells (BAECs).

  8. Fluorescence decays of fluorescently labeled CaM (show KRIT1 Antibodies) bound to eNOS reveal four distinct conformational states and single-molecule fluorescence trajectories show multiple fluorescence states with transitions between states

  9. Endothelial nitric oxide synthase is regulated by ERK (show MAPK1 Antibodies) phosphorylation at Ser602.

  10. key regulatory role of CaM (show KRIT1 Antibodies) involves the stabilization of structural intermediates and precise positioning of the pivot for the FMN (show FMN1 Antibodies) domain tethered shuttling motion to accommodate efficient and rapid electron transfer in the homodimer of eNOS.

Rabbit Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. VEGFR2 (show KDR Antibodies) activation was not affected by Slit2 (show SLIT2 Antibodies), but eNOS phosphorylation was diminished

  2. Data suggest distinct localizations of eNOS at the spiral arteries/arterial sinuses and iNOS (show NOS2 Antibodies) along the radial arteries in the developing placenta.

  3. Pulmonary ischaemia-reperfusion up-regulates inducible nitric oxide synthesis and/activity, which coincides with reduced endothelial nitric oxide synthase activity.

  4. eNOS dysregulation may be a central mechanism of impaired cardioprotection during hyperglycemia.

  5. Quercetin inhibited myocardial ischemia-reperfusion-induced NOS3 mRNA and protein expression.

Mouse (Murine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. CAV1 (show CAV1 Antibodies) KO mice have elevated IOP and reduced conventional outflow facility when compared with WT mice. CAV2 (show CAV2 Antibodies) expression was absent in CAV1 (show CAV1 Antibodies) KO mice, but we observed increased expressions of eNOS

  2. TNF-alpha (show TNF Antibodies) does not regulate eNOS activity in murine endothelial cells through serine 116 phosphorylation and Pin1 (show PIN1 Antibodies) binding.

  3. The eNOS expressed in smooth muscle cells in FAs (show FAS Antibodies) attenuates alpha-adrenergic vasoconstriction.

  4. Thioredoxin (show TXN Antibodies)-mediated deglutathionylation of eNOS in the coronary artery in vivo protected against reperfusion injury, even in the presence of normal levels of glutathione.

  5. Eph (show EPHA1 Antibodies)-B4 stimulates eNOS phosphorylation in vitro and may mediate vein graft adaptation by regulation of eNOS activity in vivo.

  6. Loss of endothelial NO plays an important role in the generation of p25 (show CDK5R1 Antibodies) and resulting tau phosphorylation in neuronal tissue. Endothelial nitric oxide synthase (eNOS)-deficient (eNOS-/-) mice display increased levels of p25 (show CDK5R1 Antibodies), an aberrant activator of cyclin-dependent kinase 5 (show CDK5 Antibodies), which is one of the primary kinases responsible for tau hyperphosphorylation, and a statistically higher p25/p35 (show CDK5R1 Antibodies) ratio.

  7. Pulse pressure finely regulates eNOS, controlling cerebral artery reactivity.

  8. TNF-alpha (show TNF Antibodies) induces vascular insulin (show INS Antibodies) resistance by mechanisms that involve positive modulation of PTEN (show PTEN Antibodies) and inhibition of Akt (show AKT1 Antibodies)/eNOS/NO signaling.

  9. Beta2AR (show ADRB2 Antibodies) overexpression enhances endothelial progenitor cells (EPC (show TCF21 Antibodies)) functions in vitro and enhances the vascular repair abilities of EPCs in vivo via the beta2AR (show ADRB2 Antibodies)/Akt (show AKT1 Antibodies)/eNOS pathway.

  10. Through the abrogation of NAD(P)H (show NQO1 Antibodies) oxidase-driven eNOS uncoupling.

Guinea Pig Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. The present evidence indicated that the customary HBOT protocol may increase constitutive NOS expression.

ENOS (NOS3) Antigen Profile

Antigen Summary

Nitric oxide is a reactive free radical which acts as a biologic mediator in several processes, including neurotransmission and antimicrobial and antitumoral activities. Nitric oxide is synthesized from L-arginine by nitric oxide synthases. Variations in this gene are associated with susceptibility to coronary spasm. Multiple transcript variants encoding different isoforms have been found for this gene.

Alternative names and synonyms associated with ENOS (NOS3)

  • nitric oxide synthase 3 (endothelial cell) (NOS3) antibody
  • nitric oxide synthase 3 (endothelial cell) (nos3) antibody
  • endothelial nitric oxide synthase (23B) antibody
  • nitric oxide synthase 3, endothelial cell (Nos3) antibody
  • 2310065A03Rik antibody
  • cNOS antibody
  • EC-NOS antibody
  • ecNOS antibody
  • ENOS antibody
  • NOS antibody
  • Nos-3 antibody
  • NOS3 antibody
  • NOSIII antibody

Protein level used designations for NOS3

nitric oxide synthase 3 (endothelial cell) , nitric oxide synthase, endothelial-like , EC-NOS , NOS type III , NOSIII , cNOS , constitutive NOS , endothelial NOS , nitric oxide synthase, endothelial , eNOS , endothelial nitric oxide synthase , endothelial nitric oxide synthase NOS3 , endothelial nitric oxide synthase 3

714231 Macaca mulatta
100018035 Monodelphis domestica
100486016 Xenopus (Silurana) tropicalis
443077 Ovis aries
4846 Homo sapiens
403784 Canis lupus familiaris
397557 Sus scrofa
287024 Bos taurus
443073 Ovis aries
100063339 Equus caballus
100009498 Oryctolagus cuniculus
18127 Mus musculus
24600 Rattus norvegicus
100135577 Cavia porcellus
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