Browse our anti-ENOS (NOS3) Antibodies

Full name:
anti-Nitric Oxide Synthase 3 (Endothelial Cell) Antibodies (NOS3)
On www.antibodies-online.com are 478 Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) Antibodies from 32 different suppliers available. Additionally we are shipping ENOS Kits (97) and ENOS Proteins (23) and many more products for this protein. A total of 612 ENOS products are currently listed.
Synonyms:
2310065A03Rik, cNOS, EC-NOS, ecNOS, ENOS, NOS, Nos-3, NOS3, NOSIII

All available anti-ENOS Antibodies

Go to our pre-filtered search.

Top referenced anti-ENOS Antibodies

  1. Amphibian Polyclonal ENOS Primary Antibody for IHC (fro), IHC (p) - ABIN152659 : Guo, Comhair, Zheng, Dweik, Eissa, Thomassen, Calhoun, Erzurum: Molecular mechanisms of increased nitric oxide (NO) in asthma: evidence for transcriptional and post-translational regulation of NO synthesis. in Journal of immunology (Baltimore, Md. : 1950) 2000 (PubMed)
    Show all 7 references for 152659

  2. Human Polyclonal ENOS Primary Antibody for IF (p), IHC (p) - ABIN725690 : Li, Han, Guo, Li, Sang: Oxidative stress, endothelial dysfunction and inflammatory response in rat heart to NO? inhalation exposure. in Chemosphere 2011 (PubMed)
    Show all 7 references for 725690

  3. Human Polyclonal ENOS Primary Antibody for IF (p), IHC (p) - ABIN746468 : Ikemura, Yamamoto, Motomura, Yamaguchi, Zhao, Iwasaki, Iwamoto: Preventive effects of the anti-vasospasm agent via the regulation of the Rho-kinase pathway on the development of steroid-induced osteonecrosis in rabbits. in Bone 2013 (PubMed)
    Show all 4 references for 746468

  4. Human Polyclonal ENOS Primary Antibody for EIA, FACS - ABIN953739 : Yanamandra, Napper, Pramanik, Bocchini, Dhanireddy: Endothelial nitric oxide synthase genotypes in the etiology of retinopathy of prematurity in premature infants. in Ophthalmic genetics 2010 (PubMed)
    Show all 4 references for 953739

  5. Human Polyclonal ENOS Primary Antibody for IF, WB - ABIN319319 : Wadman: GM advisory panel is slanted, say critics. in Nature 1999 (PubMed)
    Show all 3 references for 319319

  6. Human Polyclonal ENOS Primary Antibody for FACS, IF - ABIN655773 : Ren, Sun, Deng, Zhang, Wu, Wei, Mani, Dou, Wang: The anti-inflammatory effect and potential mechanism of cardamonin in DSS-induced colitis. in American journal of physiology. Gastrointestinal and liver physiology 2015 (PubMed)
    Show all 2 references for 655773

  7. Dog (Canine) Monoclonal ENOS Primary Antibody for WB - ABIN968916 : Michell, Chen Zp, Tiganis, Stapleton, Katsis, Power, Sim, Kemp: Coordinated control of endothelial nitric-oxide synthase phosphorylation by protein kinase C and the cAMP-dependent protein kinase. in The Journal of biological chemistry 2001 (PubMed)

  8. Human Polyclonal ENOS Primary Antibody for IF (p), IHC (p) - ABIN703315 : Papinska, Mordwinkin, Meeks, Jadhav, Rodgers: Angiotensin-(1-7) administration benefits cardiac, renal and progenitor cell function in db/db mice. in British journal of pharmacology 2015 (PubMed)

  9. Human Polyclonal ENOS Primary Antibody for EIA, WB - ABIN453769 : Rikova, Guo, Zeng, Possemato, Yu, Haack, Nardone, Lee, Reeves, Li, Hu, Tan, Stokes, Sullivan, Mitchell, Wetzel, Macneill, Ren, Yuan, Bakalarski, Villen, Kornhauser, Smith, Li, Zhou, Gygi, Gu et al.: Global survey of phosphotyrosine signaling identifies oncogenic kinases in lung cancer. ... in Cell 2007 (PubMed)

  10. Human Polyclonal ENOS Primary Antibody for EIA - ABIN453267 : Greif, Kou, Michel: Site-specific dephosphorylation of endothelial nitric oxide synthase by protein phosphatase 2A: evidence for crosstalk between phosphorylation sites. in Biochemistry 2002 (PubMed)

More Antibodies against ENOS Interaction Partners

Human Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. In the subgroup analysis based on ethnicity, both T786C and 4a4b eNOS polymorphisms could influence the risk of male infertility in Asian and Caucasian.[meta-analysis].

  2. Phosphocreatine exerts an antiapoptotic effect in HUVECs exposed to oxidative stress by methylglyoxal through the mitochondrial pathway and the modulation of PI3K (show PIK3CA Antibodies)/Akt (show AKT1 Antibodies)/eNOS and NF-kappaB (show NFKB1 Antibodies) signaling.

  3. NOS2 (show NANOS2 Antibodies) rs2779248, NOS2 (show NANOS2 Antibodies) rs1137933, and NOS3 (show NANOS3 Antibodies) rs3918188 genetic polymorphisms are potentially related to the susceptibility to type 2 diabetes mellitus (T2DM), and the rs1800783 polymorphism might be considered as genetic risk factors for diabetic nephropathy.

  4. This study determined the genetic polymorphisms of the eNOS (Glu298Asp) and Cav-1 (show CAV1 Antibodies) (G14713A and T29107A) genes in association with susceptibility risk in patients who had suffered from a large artery atherosclerotic stroke. It demonstrated that the eNOS Glu298Asp in combination with 14713A/29107A at the CAV1 (show CAV1 Antibodies) locus was associated with a significant risk of an large artery atherosclerotic stroke.

  5. The eNOS T786C polymorphism was associated with ISR in Chinese Han patients treated with DES (show DES Antibodies). Genotyping may be helpful to identify patients with higher risks of ISR after drug-eluting stent implantation.

  6. Meta-analysis. eNOS gene G894T polymorphism was related to pregnancy-induced hypertension risk, especially for Asians.

  7. Data suggest that, in T-lymphocytes, nitric oxide generated by eNOS S-nitrosylates Cys374 on ACTB (show ACTB Antibodies) and thus regulates activation/recruitment of PRKCQ (show PRKCQ Antibodies) at immune synapse; S-nitrosylation of beta-actin (show ACTB Antibodies) impairs actin binding to PFN1 (show PFN1 Antibodies) and regulates protein transport in lamellipodia. (eNOS = nitric oxide synthase (show NOS Antibodies) 3; ACTB (show ACTB Antibodies) = beta-actin (show ACTB Antibodies); PRKCQ (show PRKCQ Antibodies) = protein kinase C-theta (show PRKCQ Antibodies); PFN1 (show PFN1 Antibodies) = profilin-1 (show PFN1 Antibodies))

  8. The GG eNOS allele showed greater susceptibility to atherosclerosis.

  9. NOS3 (show NANOS3 Antibodies) TT894 genotype protected against doxorubicin cardiotoxicity in survivors of childhood acute lymphoblastic leukemia.

  10. These findings indicated that miR (show MLXIP Antibodies)-126 protects HCMECs from H/R-induced injury and inflammatory response by activating the PI3K (show PIK3CA Antibodies)/Akt (show AKT1 Antibodies)/ eNOS signaling pathway.

Pig (Porcine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. NOS3 was lowest in kidneys removed from live pigs, greater in kidneys from pigs with brain death, and greatest in kidneys from pigs with cardiac arrest.

  2. Rapid atrial pacing increases ADMA and down-regulates eNOS expression in an ADMA-independent manner.

  3. Icariin and icariside II may increase the eNOS expression through activating EGF-EGFR (show EGFR Antibodies) pathway in porcine aortic endothelial cells.

  4. Data suggest that pig sperm contain bNOS (show NOS1 Antibodies), iNOS (show NOS2 Antibodies), and eNOS; up-regulation of NOS (show NOS Antibodies) by leptin (show LEP Antibodies) during acrosome reaction and inhibition of acrosome reaction by inhibitors of nitric oxide synthases suggests these enzymes are involved in acrosome reaction.

  5. Periodic acceleration (pGz) acutely increases endothelial and neuronal nitric oxide synthase (show NOS1 Antibodies) expression in endomyocardium of normal swine.

  6. Data suggest that angiotensin II regulates nNOS (show NOS1 Antibodies) and eNOS expression and NOS (show NOS Antibodies) activity in afferent arterioles of the developing kidney via angiotensin 1 and 2 receptors.

  7. Endothelial nitric oxide synthase mRNA expression was elevated in gestational day 50 intrauterine growth retardation placenta and areola compared to gd50 control.

  8. Oligonol prevented the impairment of eNOS activity induced by high glucose through reversing altered eNOS phosphorylation status.

  9. Exercise training significantly increased total eNOS and phosphorylated levels of eNOS (pSer(1179)) in collateral-dependent arteries of experimental minipigs.

  10. Data show that wall shear stress increases with a decrease in artery diameter; eNOS protein contents decrease with an increase in diameter.

Cow (Bovine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. These data define the mechanism responsible for the repressive effects of nitric oxide (NO) on the transcriptional activity of beta-catenin (show CTNNB1 Antibodies) and link eNOS-derived NO to the modulation by VEGF (show VEGFA Antibodies) of Wnt (show WNT2 Antibodies)/beta-catenin (show CTNNB1 Antibodies)-induced endothelial cell proliferation.

  2. Endothelial cell autophagy maintains shear stress-induced nitric oxide generation via glycolysis-dependent ATP/P2Y1 receptor (show P2RY1 Antibodies) signaling to endothelial nitric oxide synthase.

  3. TNFalpha (show TNF Antibodies)reduces eNOS activity in bovine endothelial cells through serine 116 phosphorylation and Pin1 binding.

  4. Pomanox supplementation hinders hyperlipemia-induced coronary endothelial dysfunction by activating the Akt (show AKT1 Antibodies)/endothelial nitric oxide-synthase pathway and favorably counteracting vascular inflammation and oxidative damage.

  5. Signals that activate and phosphorylate eNOS are transmitted through distinct membrane domains in endothelial cells. Cholesterol domains, but not individual caveolae, mediate HDL stimulation of eNOS. VEGF and shear stress may act through caveolae.

  6. eNOS serine 1179 phosphorylation, in addition to enhancing NO production, also profoundly affects superoxide generation

  7. In addition to the heme center of eNOS oxygenase domain, we confirmed another O2.- generation site in the eNOS reductase domain and characterized its regulatory properties.

  8. A dimer-destabilized mutant of bovine eNOS where cysteine 101 was replaced by alanine, cloned and introduced into an eNOS-deficient mouse strain and that results provide the first in vivo evidence that eNOS-dependent oxidative stress is unlikely to be an initial cause of impaired endothelium-dependent vasodilation and/or a pathologic factor promoting intimal hyperplasia.

  9. Data show that resveratrol (Res) reversed caveolin-1 (Cav-1 (show CAV1 Antibodies))/endothelial nitric oxide synthase (eNOS) expressions in high glucose cultured bovine aortic endothelial cells (BAECs).

  10. Fluorescence decays of fluorescently labeled CaM (show KRIT1 Antibodies) bound to eNOS reveal four distinct conformational states and single-molecule fluorescence trajectories show multiple fluorescence states with transitions between states

Rabbit Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. Kidneys from circulation-restricted fetuses showed endothelial nitric oxide synthase phosphorylation inhibition.

  2. Antidiabetic drug pioglitazone protects the heart via activation of endothelial nitric oxide synthase (eNOS/Nos3) pathway in a rabbit model of myocardial infarction.

  3. VEGFR2 (show KDR Antibodies) activation was not affected by Slit2 (show SLIT2 Antibodies), but eNOS phosphorylation was diminished

  4. Data suggest distinct localizations of eNOS at the spiral arteries/arterial sinuses and iNOS (show NOS2 Antibodies) along the radial arteries in the developing placenta.

  5. Pulmonary ischaemia-reperfusion up-regulates inducible nitric oxide synthesis and/activity, which coincides with reduced endothelial nitric oxide synthase activity.

  6. eNOS dysregulation may be a central mechanism of impaired cardioprotection during hyperglycemia.

  7. Quercetin inhibited myocardial ischemia-reperfusion-induced NOS3 mRNA and protein expression.

Mouse (Murine) Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. these data suggest that CD NOS3 may be involved in the diuretic response to a water load in a sex-specific manner; the mechanism of this effect remains to be determined.

  2. CAV1 (show CAV1 Antibodies) KO mice have elevated IOP and reduced conventional outflow facility when compared with WT mice. CAV2 (show CAV2 Antibodies) expression was absent in CAV1 (show CAV1 Antibodies) KO mice, but we observed increased expressions of eNOS

  3. TNF-alpha (show TNF Antibodies) does not regulate eNOS activity in murine endothelial cells through serine 116 phosphorylation and Pin1 (show PIN1 Antibodies) binding.

  4. The eNOS expressed in smooth muscle cells in FAs (show FAS Antibodies) attenuates alpha-adrenergic vasoconstriction.

  5. Thioredoxin (show TXN Antibodies)-mediated deglutathionylation of eNOS in the coronary artery in vivo protected against reperfusion injury, even in the presence of normal levels of glutathione.

  6. Eph (show EPHA1 Antibodies)-B4 stimulates eNOS phosphorylation in vitro and may mediate vein graft adaptation by regulation of eNOS activity in vivo.

  7. Loss of endothelial NO plays an important role in the generation of p25 (show CDK5R1 Antibodies) and resulting tau phosphorylation in neuronal tissue. Endothelial nitric oxide synthase (eNOS)-deficient (eNOS-/-) mice display increased levels of p25 (show CDK5R1 Antibodies), an aberrant activator of cyclin-dependent kinase 5 (show CDK5 Antibodies), which is one of the primary kinases responsible for tau hyperphosphorylation, and a statistically higher p25/p35 (show CDK5R1 Antibodies) ratio.

  8. Pulse pressure finely regulates eNOS, controlling cerebral artery reactivity.

  9. TNF-alpha (show TNF Antibodies) induces vascular insulin (show INS Antibodies) resistance by mechanisms that involve positive modulation of PTEN (show PTEN Antibodies) and inhibition of Akt (show AKT1 Antibodies)/eNOS/NO signaling.

  10. Beta2AR (show ADRB2 Antibodies) overexpression enhances endothelial progenitor cells (EPC (show TCF21 Antibodies)) functions in vitro and enhances the vascular repair abilities of EPCs in vivo via the beta2AR (show ADRB2 Antibodies)/Akt (show AKT1 Antibodies)/eNOS pathway.

Guinea Pig Nitric Oxide Synthase 3 (Endothelial Cell) (NOS3) interaction partners

  1. The present evidence indicated that the customary HBOT protocol may increase constitutive NOS expression.

ENOS (NOS3) Antigen Profile

Antigen Summary

Nitric oxide is a reactive free radical which acts as a biologic mediator in several processes, including neurotransmission and antimicrobial and antitumoral activities. Nitric oxide is synthesized from L-arginine by nitric oxide synthases. Variations in this gene are associated with susceptibility to coronary spasm. Multiple transcript variants encoding different isoforms have been found for this gene.

Alternative names and synonyms associated with ENOS (NOS3)

  • nitric oxide synthase 3 (endothelial cell) (NOS3) antibody
  • nitric oxide synthase 3 (endothelial cell) (nos3) antibody
  • endothelial nitric oxide synthase (23B) antibody
  • nitric oxide synthase 3, endothelial cell (Nos3) antibody
  • 2310065A03Rik antibody
  • cNOS antibody
  • EC-NOS antibody
  • ecNOS antibody
  • ENOS antibody
  • NOS antibody
  • Nos-3 antibody
  • NOS3 antibody
  • NOSIII antibody

Protein level used designations for NOS3

nitric oxide synthase 3 (endothelial cell) , nitric oxide synthase, endothelial-like , EC-NOS , NOS type III , NOSIII , cNOS , constitutive NOS , endothelial NOS , nitric oxide synthase, endothelial , eNOS , endothelial nitric oxide synthase , endothelial nitric oxide synthase NOS3 , endothelial nitric oxide synthase 3

GENE ID SPECIES
714231 Macaca mulatta
100018035 Monodelphis domestica
100486016 Xenopus (Silurana) tropicalis
443077 Ovis aries
4846 Homo sapiens
403784 Canis lupus familiaris
397557 Sus scrofa
287024 Bos taurus
443073 Ovis aries
100063339 Equus caballus
100009498 Oryctolagus cuniculus
18127 Mus musculus
24600 Rattus norvegicus
100135577 Cavia porcellus
Selected quality suppliers for anti-ENOS (NOS3) Antibodies
Did you look for something else?