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radiation decreases SATB1 expression and sensitizes cancer cells to confer clinical benefit of patients, suggesting that SATB1 is predictive of response to preoperative RT and clinical outcome in rectal cancer.
Findings indicate that high Ki67 (show MKI67 Proteins) antigen/ SATB1 protein ratio was an independent predictor of worse overall survival (OS), while Ki67 (show MKI67 Proteins) and SATB1 did not reach statistical significance as single predictors.
SATB1 reprograms the expression of tumor growth- and metastasis-associated genes to promote tumorigenesis and functionally overlaps with Wnt (show WNT2 Proteins) signaling critical for colorectal cancer tumorigenesis.
SATB1 as a Dual Regulator of Anti-Apoptotic BCL2 (show BCL2 Proteins) and Pro-Apoptotic NOXA (show PMAIP1 Proteins) Genes
SATB1 protein expression showed an increasing trend in advancing stages of breast cancer development.
replicative oncolytic adenovirus armed with SATB1 shRNA exhibits effective antitumor effect in human prostate cancer. Our study provides the basis for the development of ZD55-SATB1 for the treatment of prostate cancer
Immunohistochemical expression of SATB1 and SATB2 (show SATB2 Proteins) was analysed in tissue microarrays with primary tumours and a subset of paired lymph node metastases from 175 patients operated with pancreaticoduodenectomy for periampullary adenocarcinoma.
expression of SATB1 may increase the size of the BCSC population via the activation of the Notch (show NOTCH1 Proteins) signaling pathway and by increasing expression levels of Snail1 (show SNAI1 Proteins) and Twist1 (show TWIST1 Proteins).
SATB1 and HER2 (show ERBB2 Proteins) expression correlated with poorly differentiated breast cancer and indicated an unfavorable prognosis.
SATB1 is overexpressed in pancreatic cancer, promoting cancer cell proliferation and invasion through the activation of MYC (show MYC Proteins).
These results suggest that SATB1 plays an essential role in establishment of immune tolerance.
Data show that RNA binding protein HuD and special adenine-thymine (AT)-rich DNA-binding protein 1 (SATB1) form a positive regulatory loop that enhances NeuroD1 protein transcription and subsequent neuronal differentiation.
Fluorosed mouse ameloblasts have increased SATB1 retention and Galphaq (show GNAQ Proteins) activity.
The results indicated that the expression of SATB1 in both mRNA and protein levels was significantly decreased after cells transferred with siRNA sequence for 48 h, the proliferation of MF9 cells was significantly inhibited.
SATB1 binds to the ASE (show ARSE Proteins) and Rag promoters, facilitating inclusion of Rag2 (show RAG2 Proteins) in the chromatin hub and the loading of RNA polymerase II to both the Rag1 (show RAG1 Proteins) and Rag2 (show RAG2 Proteins) promoters.
SATB1 is indispensable for lymphocyte differentiation and stem cell development. (Review)
The ubiquitin-like domain-CUT repeat-like tandem of SATB1 are required for DNA binding.
Satb1 is a regulator that promotes hematopoietic stem cells quiescence and represses lineage commitment.
SATB1 as a critical regulator of interneuron maturation and terminal differentiation in the mammalian cortex.
These data suggest that Satb1 is required for medial ganglionic eminence-derived interneuron differentiation, connectivity, and survival.
This gene encodes a matrix protein which binds nuclear matrix and scaffold-associating DNAs through a unique nuclear architecture. The protein recruits chromatin-remodeling factors in order to regulate chromatin structure and gene expression. Multiple transcript variants encoding different isoforms have been found for this gene.
SATB homeobox 1
, special AT-rich sequence binding protein 1
, AT-rich binding protein-1
, DNA-binding protein SATB1-like
, DNA-binding protein SATB1
, special AT-rich sequence binding protein 1 (binds to nuclear matrix/scaffold-associating DNA)
, special AT-rich sequence-binding protein 1