Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species and application
anti-Human PI4K2A Antibodies:
anti-Mouse (Murine) PI4K2A Antibodies:
anti-Rat (Rattus) PI4K2A Antibodies:
Go to our pre-filtered search.
phosphatidylinositol 4-kinase (show PI4KB Antibodies) IIalpha knockdown inhibited vesicle-associated membrane protein 3 (show VAMP3 Antibodies) trafficking to perinuclear membranes and impaired the rate of VAMP3 (show VAMP3 Antibodies)-mediated recycling of the transferrin receptor
Mining oncogenomic databases revealed that loss of the PI4K2B (show PI4K2B Antibodies) allele and underexpression of PI4KIIbeta (show PI4K2B Antibodies) mRNA are associated with human cancers. This finding supports the cell data and suggests that PI4KIIbeta (show PI4K2B Antibodies) may be a clinically significant suppressor of invasion.
RNA interference of the type II phosphatidylinositol 4-kinases PI4KIIa and PI4KIIa in primary human endothelial cells leads to formation of an increased proportion of short WPB with perturbed packing of VWF (show VWF Antibodies), as exemplified by increased exposure of antibody-binding sites.
Here two crystal structures are presented: the structure of human PIK42A and the structure of PIK42B (show PI4K2B Antibodies) containing a nucleoside analogue.
PI4KIIalpha is a specific downstream effector of GABARAP (show GABARAP Antibodies) and it generates PI4P, which has a key role in the final stage of autophagy.
phosphatidylinositol 4-kinase (show PI4KA Antibodies) IIalpha knockdown inhibited vesicle-associated membrane protein 3 (show VAMP3 Antibodies) trafficking to perinuclear membranes and impaired the rate of VAMP3 (show VAMP3 Antibodies)-mediated recycling of the transferrin receptor
PI4K2A is a novel regulator of vesicular trafficking and neurotransmission in the brain.
The Hermansky-Pudlak syndrome complex BLOC-1 and its cargo PI4KIIalpha interact with regulators of the actin cytoskeleton.
Knockdown of PI4KIIalpha inhibits EGF (show EGF Antibodies)-stimulated Akt (show AKT1 Antibodies) phosphorylation.
Results suggest that the phosphatidylinositol 4-kinase 2-alpha gene may not be a cause of autosomal recessive hereditary spastic paraplegia in humans.
PI4KIIalpha is a novel regulator of tumor growth by its action on angiogenesis and HIF-1alpha (show HIF1A Antibodies) regulation.
Itch directly associates with and ubiquitinates PI4KIIalpha, and both proteins colocalize on endosomes containing Wnt (show WNT2 Antibodies)-activated frizzled 4 (Fz4 (show FZD4 Antibodies)) receptor
PI4KIIalpha is present on adaptor protein (AP) -3 organelles where it regulates AP-3 function
The unique N terminus of PI4K type IIalpha is required for the targeting of the enzyme to cellugyrin (show SYNGR2 Antibodies)-positive vesicles.
AP-3 (show AP3B1 Antibodies) and BLOC-1 (show PLDN Antibodies) act, either in concert or sequentially, to specify sorting of PI4KIIalpha along the endocytic route.
Phosphatidylinositolpolyphosphates (PtdInsPs) are centrally involved in many biologic processes, ranging from cell growth and organization of the actin cytoskeleton to endo- and exocytosis. PI4KII phosphorylates PtdIns at the D-4 position, an essential step in the biosynthesis of PtdInsPs (Barylko et al., 2001
phosphatidylinositol 4-kinase type 2 alpha
, phosphatidylinositol 4-kinase type II
, phosphatidylinositol 4-kinase type 2-alpha
, phosphatidylinositol 4-kinase type II-alpha
, similar to phosphatidylinositol 4-kinase type II
, phosphatidylinositol 4-kinase type 2-alpha-like
, phosphatidylinositol 4-kinase type II (PI4KII)
, 55 kDa type II phosphatidylinositol 4-kinase
, phosphatidylinositol 4-kinase II alpha