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Mouse (Murine) OMG ELISA Kit for Competition ELISA - ABIN1882533
deLuca, Pikor, OLeary, Galicia-Rosas, Ward, Defreitas, Finlay, Ousman, Osborne, Gommerman: Substrain differences reveal novel disease-modifying gene candidates that alter the clinical course of a rodent model of multiple sclerosis. in Journal of immunology (Baltimore, Md. : 1950) 2010
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Human OMG ELISA Kit for Competition ELISA - ABIN1882530
Kezuka, Usui, Yamakawa, Matsunaga, Matsuda, Masuda, Utsumi, Tanaka, Goto: Relationship between NMO-antibody and anti-MOG antibody in optic neuritis. in Journal of neuro-ophthalmology : the official journal of the North American Neuro-Ophthalmology Society 2012
Mouse (Murine) OMG ELISA Kit for Competition ELISA - ABIN1882535
Hatterer, Touret, Belin, Honnorat, Nataf: Cerebrospinal fluid dendritic cells infiltrate the brain parenchyma and target the cervical lymph nodes under neuroinflammatory conditions. in PLoS ONE 2008
Retinal nerve fiber layer may be better preserved in MOG-IgG versus AQP4 (show AQP4 ELISA Kits)-IgG optic neuritis.
Data show that aquaporin-4 (AQP4) and myelin-oligodendrocyte glycoprotein (MOG) autoantibodies double positive neuromyelitis optica spectrum disorder (NMOSD) patients had a multiphase disease course with a high annual relapse rate.
MOG antibody associated optic neuritis tends to involve the anterior optic pathway.
This study demonstrated that increased expression of mRNA of OLIG1 (show OLIG1 ELISA Kits) in ventral prefrontal white matter in major depressive disorder.
In patients with idiopathic optic neuritis, 27.6% (8/29) were positive for MOG antibodies. Three of the eight MOG-positive patients showed significantly high CSF (show CSF2 ELISA Kits) levels of myelin basic protein (show MBP ELISA Kits). Five had optic pain and three had prodromal infection.
Immune modulation by a tolerogenic myelin oligodendrocyte glycoprotein (MOG)10-60 containing fusion protein in the marmoset experimental autoimmune encephalomyelitis model.
These data demonstrate a new role for myelin glycosylation in the control of immune homeostasis in the healthy human brain through the MOG-DC-SIGN (show CD209 ELISA Kits) homeostatic regulatory axis, which is comprised by inflammatory insults that affect glycosylation.
findings suggest that immune reactions toward MOG and in particular MOG-specific antibodies may play a functional role in multiple sclerosis
Patients with neuromyelitis optica spectrum disorders with MOG antibodies have distinct clinical features, fewer attacks, and better recovery than patients with AQP4 (show AQP4 ELISA Kits) antibodies or patients seronegative for both antibodies.
Bipolar I disorder and schizophrenia share a number of common genetic risk loci and susceptibility genes including the genes coding for myelin oligodendrocytes glycoprotein (MOG).
Identify nerve growth factor as a binding partner for MOG and demonstrate that this interaction is capable of sequestering NGF (show NGFB ELISA Kits) from TrkA (show NTRK1 ELISA Kits)-expressing neurons to modulate axon growth and survival.
Data indicate that in mixed chimeras with bone marrow-encoding myelin oligodendrocyte glycoprotein (MOG), the development of MOG-specific B cells was abrogated, resulting in a lack of MOG-specific B cells in all B cell compartments examined.
Identify immunogenic/encephalitogenic T-cell epitopes within MOG capable of inducing experimental autoimmune encephalomyelitis in C57BL/6 mice.
PLP significantly suppresses both models of experimental autoimmune encephalitis (EAE)even when there is some evidence of epitope spreading in the myelin oligodendrocyte (MOG)38-50-induced EAE model.
CNTF may play a role in the process of remyelination by inducing the MOG expression.
MOG-specific CD4 (show CD4 ELISA Kits)-positive T cells accumulate within the chemokine (show CCL1 ELISA Kits)-expressing draining lymph nodes, rather than within the brain or spinal cord during induction of autoimmune encephalomyelitis.
This study provides valuable information about requirements of anti-myelin oligodendrocyte glycoprotein reactivity for being regarded as a prognostic biomarker in a subtype of MS.
Mice immunized with encephalitogenic myelin oligodendrocyte glycoprotein MOG(35-55) peptide develop significant serum levels of anti-MOG antibodies in parallel with disease progression.
B cell-deficient mice exhibit reduced medullary thymic epithelial cell numbers and reduced MOG mRNA expression.
Cell surface targeting of MOG is not disrupted in the absence of the endoplasmic reticulum chaperones.
Cell adhesion molecule contributing to the interactive process required for myelination in the central nervous system.
, MOG alpha-5
, myelin-oligodendrocyte glycoprotein
, oligodendrocyte-myelin glycoprotein
, dystonia 2, torsion (autosomal recessive)
, myelin oligodendrocyte glycoprotein
, Myelin-oligodendrocyte glycoprotein
, myelin-oligodendrocyte glycoprotein-like
, MOG alpha-7Cj
, leucine-rich glycoprotein homolog
, oligodendrocyte myelin glycoprotein
, oligodendrocyte-myelin glycoprotein-like
, myelin-oligodendrocyte glycoprotein isoform beta1