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anti-Human GLP1R Antibodies:
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Human Polyclonal GLP1R Primary Antibody for DB, ICC - ABIN4314326
Kedees, Guz, Grigoryan, Teitelman: Functional activity of murine intestinal mucosal cells is regulated by the glucagon-like peptide-1 receptor. in Peptides 2013
Show all 5 references for ABIN4314326
Human Polyclonal GLP1R Primary Antibody for FACS, IF (p) - ABIN731333
Zhou, Yang, Xin, Li, Guo, Hu, Zhou, Zhang, Zhang, Han, Chen: Exendin-4 protects adipose-derived mesenchymal stem cells from apoptosis induced by hydrogen peroxide through the PI3K/Akt-Sfrp2 pathways. in Free radical biology & medicine 2014
Show all 2 references for ABIN731333
Human Polyclonal GLP1R Primary Antibody for IHC, IHC (p) - ABIN270557
Matveyenko, Dry, Cox, Moshtaghian, Gurlo, Galasso, Butler, Butler: Beneficial endocrine but adverse exocrine effects of sitagliptin in the human islet amyloid polypeptide transgenic rat model of type 2 diabetes: interactions with metformin. in Diabetes 2009
Show all 2 references for ABIN270557
Human Polyclonal GLP1R Primary Antibody for IF, ELISA - ABIN1535693
Thorens, Porret, Bühler, Deng, Morel, Widmann: Cloning and functional expression of the human islet GLP-1 receptor. Demonstration that exendin-4 is an agonist and exendin-(9-39) an antagonist of the receptor. in Diabetes 1993
Show all 2 references for ABIN1535693
Human Monoclonal GLP1R Primary Antibody for FACS - ABIN4899829
Thompson, Stephens, Bain, Kanamarlapudi: Molecular Characterisation of Small Molecule Agonists Effect on the Human Glucagon Like Peptide-1 Receptor Internalisation. in PLoS ONE 2016
Role of cysteine 341 and arginine 348 of GLP-1 receptor in G-protein coupling.
In glucagon (show GCG Antibodies)-like peptide receptor (GLP-1R) expressing cells, small molecule agonists induced cAMP production but caused no intracellular Ca2 (show CA2 Antibodies)+ accumulation, ERK (show EPHB2 Antibodies) phosphorylation or hGLP-1R internalisation.
We aimed to investigate whether genetic variations in glucagon (show GCG Antibodies)-like peptide receptor are associated with responses to dipepdityl peptidase-4 (show PEPD Antibodies) inhibitors in patients with type 2 diabetes. Polymorphism in the GLP-1 receptor may influence DPP-4 (show DPP4 Antibodies) inhibitor response.
Results suggest that pancreatic ductal adenocarcinoma (PDAC) cells or their precursor lesions do not overexpress glucagon-like peptide-1 receptor (GLP-1R) compared with non-neoplastic pancreatic cells.
The molecular dynamics simulations of wild-type and mutant GLP (show RCBTB1 Antibodies)-1R.ligand complexes provided molecular insights into GLP-1R-specific recognition mechanisms for the N terminus of GLP-1 (show GCG Antibodies) by residues in the 7TM pocket and explained how glucagon (show GCG Antibodies)-mimicking GLP-1 (show GCG Antibodies) mutants restored binding affinity for (glucagon receptor (show GCGR Antibodies) -mimicking) GLP-1R mutants.
NMR-determined structure of a high-potency cyclic conformationally-constrained 11-residue analogue of GLP-1 (show GCG Antibodies) was also docked into the receptor-binding site.
Lack of association of rs6923761 GLP-1 R polymorphism with weight loss.
An association was found between the rs6923761 GLP-1 receptor polymorphism and basal GLP-1 (show GCG Antibodies) levels in diabetes mellitus type 2 patients.
although GLP-1R is not an independent prognostic factor in PDAC patients, it appears to have some implications for pancreatic ductal adenocarcinoma metastatic ability
Retinal GLP1R expression was similar in patients with diabetes and healthy controls.
GLP-1R rs10305420 polymorphism explained some of the inter-individual differences in response to liraglutide regarding weight loss in obese PCOS women.
Genetic deletion of both GLP-1 (show GCG Antibodies) and GIP (show GIP Antibodies) receptors reveals that they are required to maintain an adequate islet number in adulthood and to maintain normal beta cell responses to glucose.
present study provides critical insights regarding the nature by which GLP-1 (show GCG Antibodies) signaling controls reinforced behaviors and underscores the importance of both peripheral and central GLP-1R signaling for the regulation of addictive disorders.
CB1 (show CNR1 Antibodies) activation negatively impacts GLP-1R-mediated insulin (show INS Antibodies) secretion.
These differential effects of exogenous Glp1r in nondiabetic and diabetic mice suggest that downregulation of Glp1r might be required to slow the progression of beta-cell failure under diabetic conditions.
although endogenous GLP-1R signalling contributes to increased brown adipose tissue thermogenesis, this mechanism does not play a significant role in the food intake-independent body weight lowering effect of the GLP-1 (show GCG Antibodies) mimetic liraglutide in obese mice
GLP-1R is present in pancreatic acinar cells and that GLP-1 (show GCG Antibodies) can regulate secretion through its receptor and cAMP signaling pathway.
GLP1R is expressed in mouse retina. GLP-1R protein abundance was independent of the presence of diabetes.
The data of this study reveal a novel role of GLP-1R in dorsal lateral septum function driving behavioral responses to cocaine.
GLP-1R agonism significantly reduced alcohol consumption in a mouse model of alcohol dependence.
these data provide novel evidence that lipotoxicity decreases the mesangial GLP-1R expression in intact cells and in vivo.
induces insulin secretion\; mediates neuroendocrine signaling of feeding behavior\; mediates cardiovascular response and increased blood pressure
glucagon-like peptide 1 receptor
, glucagon-like peptide 1 receptor-like
, GLP-1 receptor
, pancreatic beta cell receptor for the gluco-incretin hormone glucagon-like peptide 1