Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Show all species
Show all synonyms
Select your species
The authors demonstrate that HIV and specifically Nef and/or Vpu do not modulate CD155 on infected primary T cells and both CD155 and NKG2D (show KLRK1 ELISA Kits) ligands synergize as a natural killer cell receptor to trigger natural killer cell lysis of the infected cell.
Data show that gastric cancer cells inhibit T-cell metabolism through CD155/TIGIT (show TIGIT ELISA Kits) signaling.
Studies showed that CD155 was frequently overexpressed in human malignant tumors. Its overexpression promotes tumor cell invasion and migration, and is associated with tumor progression. [review]
soluble CD226 (show CD226 ELISA Kits) elevated in sera of CTCL (show TSPYL2 ELISA Kits) patients would be important for tumor immunity by interacting with CD155 on tumor cells.
data reveal that MICA (show MICA ELISA Kits) and PVR are directly regulated by human cytomegalovirus immediate early (show JUN ELISA Kits) proteins, and this may be crucial for the onset of an early host antiviral response
The SNP detection assay was successfully developed for identification of Ala67Thr polymorphism in human PVR/CD155 gene. The SNP assay will be useful for large scale screening of DNA samples.
implying that TIGIT (show TIGIT ELISA Kits) exerts immunosuppressive effects by competing with DNAM-1 (show CD226 ELISA Kits) for the same ligand, CD155
The present study provides evidence that regulation of the PVR/CD155 DNAM-1 (show CD226 ELISA Kits) ligand expression by nitric oxide may represent an additional immune-mediated mechanism and supports the anti-myeloma activity of nitric oxide donors.
Our findings suggest that loss of CD155 expression may play an important role in the immune escape of HCC (show FAM126A ELISA Kits) cells and thus CD155 may serve as a prognostic marker as well as a potential therapeutic target for HCC (show FAM126A ELISA Kits).
CD155 may play a critical role through both immunological and non-immuno logical mechanisms in pancreatic cancer and may be a therapeutic target for this intractable malignancy.
Data show that CD155 protein/CD226 antigen (show CD226 ELISA Kits) mainly mediates the interaction between NK cells and leukemic cells in acute myeloid leukemia (show BCL11A ELISA Kits). To investigate the interaction between leukemic cells
absence of either CD155 or CD226 (show CD226 ELISA Kits) in BALB/c mice causes a profound shift in the Natural Killer T-Cells subtype composition in thymus, expanding the frequency and numbers of iNKT1 cells at the expense of iNKT2 cells, as well as iNKT17 cells.
Results suggest that modulation of the expression of receptors and CD112 (show PVRL2 ELISA Kits) compensates for CD155 deficiency in immune surveillance against methylcholanthrene-induced tumors.
CD155 (PVR/Necl5) mediates a costimulatory signal in CD4 (show CD4 ELISA Kits)+ T cells and regulates allergic inflammation.
Sertoli cells recognize the excess cytoplasm of elongated spermatids through the PVR-CEACAM2-L interaction in mouse testis
TIGIT (show TIGIT ELISA Kits)/PVR (show PVRL2 ELISA Kits) ligation signaling mediates suppression of IFN-gamma (show IFNG ELISA Kits) production via the NF-kappaB (show NFKB1 ELISA Kits) pathway.
Our data suggest that CD155 regulates T(h)2 differentiation
These observations establish a firm link between the functions of CD155 and CD226 (show CD226 ELISA Kits) in several T cell differentiation steps.
Necl-5/poliovirus receptor interacts with VEGFR2 (show KDR ELISA Kits) and regulates VEGF (show VEGFA ELISA Kits)-induced angiogenesis.
The TLR3 (show TLR3 ELISA Kits)-TRIF (show RNF138 ELISA Kits) mediated antiviral response is important for protection against poliovirus infection in poliovirus receptor transgenic mice.
The protein encoded by this gene is a transmembrane glycoprotein belonging to the immunoglobulin superfamily. The external domain mediates cell attachment to the extracellular matrix molecule vitronectin, while its intracellular domain interacts with the dynein light chain Tctex-1/DYNLT1. The gene is specific to the primate lineage, and serves as a cellular receptor for poliovirus in the first step of poliovirus replication. Multiple transcript variants encoding different isoforms have been found for this gene.
, nectin-like 5
, nectin-like protein 5
, tumor-associated antigen 1
, tumor-associated glycoprotein pE4