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anti-Human TEAD4 Antibodies:
anti-Mouse (Murine) TEAD4 Antibodies:
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Human Monoclonal TEAD4 Primary Antibody for IP, RNAi - ABIN563135
Benhaddou, Keime, Ye, Morlon, Michel, Jost, Mengus, Davidson: Transcription factor TEAD4 regulates expression of myogenin and the unfolded protein response genes during C2C12 cell differentiation. in Cell death and differentiation 2012
Show all 3 Pubmed References
Cow (Bovine) Polyclonal TEAD4 Primary Antibody for IHC, WB - ABIN2780491
Chen, Baty, Maeda, Brooks, Baker, Ueyama, Gursoy, Saba, Salama, London, Stewart: Transcription enhancer factor-1-related factor-transgenic mice develop cardiac conduction defects associated with altered connexin phosphorylation. in Circulation 2004
Show all 2 Pubmed References
Cow (Bovine) Polyclonal TEAD4 Primary Antibody for IF, WB - ABIN2777935
Appukuttan, McFarland, Davies, Atchaneeyasakul, Zhang, Babra, Pan, Rosenbaum, Acott, Powers, Stout: Identification of novel alternatively spliced isoforms of RTEF-1 within human ocular vascular endothelial cells and murine retina. in Investigative ophthalmology & visual science 2007
Show all 2 Pubmed References
the transcriptional regulators in the Hippo pathway, Tead4 and Yap1 (show YAP1 Antibodies), are required for general vertebrate epimorphic regeneration as well as for organ size control in appendage regeneration
Osmotic stress promotes TEAD4 cytoplasmic translocation via p38 MAPK (show MAPK14 Antibodies) in a Hippo-independent manner. Stress-induced TEAD inhibition predominates YAP (show YAP1 Antibodies)-activating signals and selectively suppresses YAP (show YAP1 Antibodies)-driven cancer cell growth.
The transcription factor TEAD4 regulates a pro-metastasis transcription program in a YAP (show YAP1 Antibodies)-independent manner in CRC (show CALR Antibodies), thus providing a novel mechanism of TEAD4 transcriptional regulation and its oncogenic role in CRC (show CALR Antibodies), independently of the Hippo pathway.
our work provides a structural basis for understanding the regulatory mechanism of TEAD4-mediated gene transcription
Our results suggest that TEAD4 plays a role in the pathophysiology of atypical teratoid/rhabdoid tumor, which represents a new insight into the biology of this aggressive tumor
It was found that the TEAD4-YAP (show YAP1 Antibodies) complex in the nuclei may be related closely to transcriptions of G1 arrest-related genes.
Collectively, these results indicate that human papillomavirus 16 E6 induces upregulation of APOBEC3B (show APOBEC3B Antibodies) through increased levels of TEADs, highlighting the importance of the TEAD-APOBEC3B (show APOBEC3B Antibodies) axis in carcinogenesis.
Tead4 cooperates with AP1 (show FOSB Antibodies) transcription factors to coordinate target gene transcription.
TEAD4 and KLF5 (show KLF5 Antibodies), in collaboration, promoted triple negative breast cancer cell proliferation and tumor growth in part by inhibiting p27 (show PAK2 Antibodies) gene transcription
potential anti-oxidation gene and can prevent H2O2-induced endothelial cell oxidative damage by activating Klotho (show KL Antibodies)
TEAD4 overexpression induced p16 (show CDKN2A Antibodies) in HAoSMCs homozygous for the nonrisk coronary disease allele, but not for the risk allele.
AP-1 (show JUN Antibodies)- and TEAD4-associated cis (show CISH Antibodies)-regulatory elements form hubs for multiple signalling-responsive transcription factors and define the cistrome that regulates vascular and hematopoietic development by extrinsic signals.
Data show that TEAD family of transcription factors Tead1 (show TEAD1 Antibodies) and Tead4-regulated gene expression in differentiating primary myoblasts.
Dual-luciferase reporter gene analysis showed that RTEF-1 is a direct target of mir-125a-5p, which regulates angiogenesis by repressing RTEF-1 expression and modulating eNOS and VEGF expression.
TEAD4 establishes the energy homeostasis essential for blastocoel formation.
These results show that RTEF-1-stimulated IGFBP-1 (show IGFBPI Antibodies) expression may be central to the mechanism by which RTEF-1 attenuates blood glucose levels.
Vgll1 (show VGLL1 Antibodies) interacts with TEAD4 in a manner similar to the transcription coactivators, as well as oncogenes YAP (show YAP1 Antibodies) and TAZ (show TAZ Antibodies), despite having a varied primary sequence. Vgll1 (show VGLL1 Antibodies) has the potential to promote cancer progression.
endothelial-specific RTEF-1 overexpressing mice had enhanced angiogenic sprouting and vascular structure remodeling, resulting in the formation of a denser and more highly interconnected superficial capillary plexus
Data suggest that altered subcellular localization of TEAD4 in blastomeres dictates first mammalian cell fate specification.
TEAD factors directly induce Myogenin (show MYOG Antibodies), CDKN1A (show CDKN1A Antibodies) and Caveolin 3 (show CAV3 Antibodies) expression to promote myoblast differentiation.
Gata3 (show GATA3 Antibodies) and Cdx2 (show CDX2 Antibodies) can act in parallel pathways downstream of Tead4 to induce the expression of common and independent targets in the trophoblast lineage, whereas Oct4 (show POU5F1 Antibodies) is required for continued repression of trophoblast fate in the embryonic lineage
This gene product is a member of the transcriptional enhancer factor (TEF) family of transcription factors, which contain the TEA/ATTS DNA-binding domain. It is preferentially expressed in the skeletal muscle, and binds to the M-CAT regulatory element found in promoters of muscle-specific genes to direct their gene expression. Alternatively spliced transcripts encoding distinct isoforms, some of which are translated through the use of a non-AUG (UUG) initiation codon, have been described for this gene.
TEA domain family member 4
, M-CAT binding factor
, M-CAT-binding factor
, transcriptional enhancer factor TEF-3
, related transcription enhancer factor 1B
, transcription factor 13-like 1
, transcription factor RTEF-1
, transcriptional enhancer factor 1-related
, transcriptional enhancer factor 3
, transcriptional enhancer factor 1-related protein
, ETF-related factor 2
, ETF-related factor-2
, FGF-regulated 19
, TEF-1-related factor 1
, TEF-1-related factor FR-19