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APOO is a member of the apolipoprotein family.
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It is likely that apoO participates in fatty acid metabolism and the inflammatory response in HepG2 cells.
APOO represents a link between impaired mitochondrial function and cardiomyopathy onset, and targeting APOO-dependent metabolic remodeling has potential as a strategy to adjust heart metabolism and protect the myocardium from impaired contractility.
relationships between plasma apoO levels and high-sensitive C-reactive protein (show CRP ELISA Kits) (hs-CRP (show CRP ELISA Kits)) levels, as well as other lipid parameters in healthy subjects and patients with established acute coronary syndrome
apolipoprotein O is the first chondroitin sulfate chain containing apolipoprotein
The liver function indices (serum ALT (show ALT ELISA Kits) and AST) were clearly elevated, and the ultrastructure of cellular mitochondria was distinctly altered in the liver after apoO overexpression. Further, mitochondrial membrane potential decreased with hApoO treatment in L02 cells.
Overexpression of apolipoprotein O (show APOOL ELISA Kits) does not impact on plasma HDL (show HSD11B1 ELISA Kits) levels or functionality in human apolipoprotein A-I (show APOA1 ELISA Kits) transgenic mice.
apolipoprotein O (show APOOL ELISA Kits) is the first chondroitin sulfate chain containing apolipoprotein
This gene is a member of the apolipoprotein family. Members of this protein family are involved in the transport and metabolism of lipids. The encoded protein associates with HDL, LDL and VLDL lipoproteins and is characterized by chondroitin-sulfate glycosylation. This protein may be involved in preventing lipid accumulation in the myocardium in obese and diabetic patients. Alternative splicing results in multiple transcript variants. Pseudogenes of this gene are found on chromosomes 3, 4, 5, 12 and 16.
, family with sequence similarity 121B
, brain my025
, Apolipoprotein O