Use your antibodies-online credentials, if available.
No Products on your Comparison List.
Your basket is empty.
Find out more
Acts as an anti-apoptotic factor and its absence increases cell death under normal and stress conditions. Additionally we are shipping C11orf82 Kits (4) and and many more products for this protein.
Showing 10 out of 13 products:
these findings indicate that the stability of the DDIAS protein is regulated by CHIP/HSP70 (show HSP70 Antibodies)-mediated proteasomal degradation and that CHIP overexpression stimulates the apoptosis of lung cancer cells in response to DNA-damaging agents
Noxin facilitated the expression of Cyclin D1 (show CCND1 Antibodies) and Cyclin E1 (show CCNE1 Antibodies) through activating P38 (show CRK Antibodies)-activating transcription factor 2 (show ATF2 Antibodies) signaling pathway, thus enhanced cell growth of breast cancer
EGF (show EGF Antibodies) activates the ERK5 (show MAPK7 Antibodies)/MEF2 (show MEF2A Antibodies) pathway, which in turn induces DDIAS expression to promote cancer cell invasion by activating beta-catenin (show CTNNB1 Antibodies) target genes
DDIAS is a target of NFATc1 (show NFATC1 Antibodies) and is associated with cisplatin resistance in lung cancer cells.
NOXIN overexpression, as a result of genomic DNA gain or amplification, promotes HCC (show FAM126A Antibodies) tumorigenesis by accelerating DNA synthesis and cell cycle progression, where NOXIN functions as a cofactor of DNA polymerase (show POLB Antibodies)-primase complex
Noxin may be a component of the cell defense system: it is activated by various stress stimuli, helps cells to withdraw from cycling, and opposes apoptosis.
Acts as an anti-apoptotic factor and its absence increases cell death under normal and stress conditions. Can induce cell cycle arrest in the G1 or early S phase and this activity is independent of p53/TP53 (By similarity).
nitric oxide-inducible gene protein