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The Golgi complex plays a key role in the sorting and modification of proteins exported from the endoplasmic reticulum. Additionally we are shipping GOLPH3 Antibodies (88) and GOLPH3 Proteins (5) and many more products for this protein.
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Golph3 is a target gene of miR34a. miR34a/GOLPH3 axis abrogates cancer cell stemness involved in urothelial bladder cancer chemoresistance.
The current study found that GOLPH3, an outer membrane protein of the Golgi complex, was significantly upregulated in N2A cells upon oxygen-glucose deprivation and reoxygenation.
High GOLPH3 expression is associated with breast cancer.
This study provides a brand new evidence that GOLPH3 promotes glioma cell proliferation by facilitating Wls recycling and Wnt (show WNT2 ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits) signaling.
High GOLPH3 expression is associated with glioma progression.
Proteomic-based screens identified GOLPH3 as a resident protein of the Golgi apparatus, and second as an oncogene (show RAB1A ELISA Kits) product in a screen for genes amplified in cancer. A third screen uncovered the association of GOLPH3 with the Golgi resident phosphatidyl inositol 4 phosphate to maintain the characteristic ribbon structure of the Golgi apparatus favoring vesicular transport of secretory proteins. [review]
Overexpression of GOLPH3 resulted in the downregulation of p21 protein (show NRAS ELISA Kits), upregulation of cyclin D1 (show CCND1 ELISA Kits) and increased retinoblastomaassociated protein phosphorylation, consequently leading to accelerated cell cycle progression.
Results indicate that GOLPH3 is an independent predictive factor for neoadjuvant chemoradiotherapy (nCRT) sensitivity and prognosis in locally advanced rectal cancer (LARC (show CCL20 ELISA Kits)).
Results provide evidences that overexpression of GOLPH3 stimulates EMT (show ITK ELISA Kits) via the Wnt (show WNT2 ELISA Kits)/beta-catenin (show CTNNB1 ELISA Kits) signaling pathway, which further promotes metastasis of epithelial ovarian cancer.
GOLPH3 levels may be a novel prognostic biomarker of tobacco related lung disease.
Authors confirm that the decrease of GOLPH3 that promotes the apoptosis of glioma cells may be regulated by the activation of NDRG1 and cleaved capcase 3. There was a inverse association between GOLPH3 and NDRG1 in glioma samples.
Data found that miR (show MLXIP ELISA Kits)-134 was highly downregulated in gastric cancer tissues and cell lines and inversely correlated with GOLPH3. These results suggest that miR (show MLXIP ELISA Kits)-134 regulates gastric cancer cell proliferation, at least potentially, through downregulation of the GOLPH3 gene, implicating a candidate tumor-suppressor miRNA in the pathogenesis of gastric cancer.
The selective S-nitrosylation of EMMPRIN and GOLPH3 at the Golgi apparatus in endothelial cells results from the physical proximity to eNOS (show NOS3 ELISA Kits)-derived nitric oxide.
The Golgi complex plays a key role in the sorting and modification of proteins exported from the endoplasmic reticulum. The protein encoded by this gene is a peripheral membrane protein of the Golgi stack and may have a regulatory role in Golgi trafficking. Several alternatively spliced transcript variants of this gene have been described, but the full-length nature of these variants has not been determined.
golgi phosphoprotein 3
, Golgi phosphoprotein 3
, coat protein GPP34
, golgi peripheral membrane protein 1, 34 kDa
, golgi protein
, golgi-associated protein
, mitochondrial DNA absence factor
, trans-Golgi protein GMx33