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guanylyl cyclase\; functions as receptor for heat-stable enterotoxin responsible for acute diarrhea [RGD, Feb 2006].. Additionally we are shipping GUCY2C Antibodies (34) and GUCY2C Proteins (10) and many more products for this protein.
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Results demonstrate that GC-C and its ligands, guanylin (show GUCA2A ELISA Kits) and uroguanylin (show GUCA2B ELISA Kits) are downregulated in ulcerative colitis (UC), and this downregulation is more significant with aggravation of the clinical condition. Therefore, the GC-C signaling pathway may be implicated in the progression of UC.
the expression of GCC is maintained throughout the process of tumor progression and formation of metastatic disease.
To investigate gut (show GUSB ELISA Kits) motility and hormones before and after a meal in Familial GUCY2C diarrhea syndrome patients and compare with healthy controls
Mutations in GUCY2C indicate a role for this receptor in the pathogenesis of sporadic Congenital secretory diarrhea.
these data support Ad5 (show PSEN2 ELISA Kits)-GUCY2C-PADRE as a safe and effective vaccination strategy in preclinical models and position Ad5 (show PSEN2 ELISA Kits)-GUCY2C-PADRE for Phase I clinical testing in colorectal cancer patients.
Familial GUCY2C diarrhoea syndrome is caused by an activating mutation in the GUCY2C gene, which causes impaired contractility and fluid stagnation in the small bowel
Findings show how caloric suppression of the guanylin (show GUCA2A ELISA Kits)-GUCY2C signaling axis links obesity to negation of a universal tumor suppressor pathway in colorectal cancer.
Data show that individuals affected with meconium ileus (MI) had either homozygous or compound heterozygous variants in enterotoxin receptor GUCY2C.
High guanylyl cyclase C expression is associated with colon cancer recurrence.
Transmission disequilibrium tests (TDT (show DNTT ELISA Kits)), case-control studies and quantitative analyses indicated association between GUC2C with Attention-deficit/hyperactivitydisorder and its core symptoms.
Loss of Gucy2c expression is associated with Acute Radiation-Induced GI Syndrome.
The GC-C signaling pathway blunts colonic mucosal inflammation that is initiated by systemic cytokine burst or loss of mucosal immune cell immunosuppression.
GC-C signaling is an essential component of host defense during murine enteric infection by reducing bacterial load and preventing systemic dissemination of attaching/effacing-lesion forming bacterial pathogens such as C. rodentium.
Intestinal cell proliferation and senescence are regulated by receptor guanylyl cyclase C and p21.
GUCY2C opposes systemic genotoxic tumorigenesis by regulating AKT (show AKT1 ELISA Kits)-dependent intestinal barrier integrity
The role for GUCY2C-directed CD8 (show CD8A ELISA Kits)(+) T cells targeting specific epitopes in antitumor efficacy.
Data show that silencing of GUCY2C in mice disrupts satiation, resulting in hyperphagia and subsequent obesity and metabolic syndrome.
a novel role for GC-C signaling in facilitating mucosal wounding and inflammation, and further suggest that this may be mediated, in part, through control of RELMbeta (show RETNLB ELISA Kits) production
guanylyl cyclase\; functions as receptor for heat-stable enterotoxin responsible for acute diarrhea
, STA receptor
, guanylyl cyclase C
, heat-stable enterotoxin receptor
, intestinal guanylate cyclase
, heat stable enterotoxin receptor
, Guanylate cyclase 2C (heat stable enterotoxin receptor)
, guanylate cyclase 2C
, guanylate cyclase 2C (heat stable enterotoxin receptor)
, heat-stable enterotoxin receptor-like