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SCARA3 encodes a macrophage scavenger receptor-like protein. Additionally we are shipping SCARA3 Antibodies (26) and SCARA3 Proteins (7) and many more products for this protein.
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CSR1 is SUMOylated at K582 and rapid ubiquitinated and degradated in prostate cancer cells.
demonstrated that CSR1 inhibited hepatocellular carcinoma cell proliferation, migration and invasion through inactivation of hematopoietic PBX interacting protein (HPIP) and its downstream PI3K/AKT signaling pathway.
The interaction between CSR1 and SF3A3 led to migration of SF3A3 from nucleus to cytoplasm. The cytoplasmic redistribution of SF3A3 significantly reduced the splicing efficiency of epidermal growth factor receptor and platelet-derived growth factor receptor.
Suppression of CSR1 expression is a novel mechanism critical for the oncogenic activity of miR-650.
Findings regarding the interaction of NADPH-P450 reductase (NPR) with cellular stress response (CSR) indicated function of NPR in hypoxic response.
Data indicate that the clinical samples showed an inverse correlation between SCARA3 gene expression, myeloma progression, and favorable clinical prognosis.
The binding of CSR1 with XIAP enhanced caspase-9 and caspase-3 protease activities.
The consistently high SCARA3 levels in both primary carcinomas and metastatic cells in effusions, and its up-regulation along disease progression from diagnosis to recurrence, suggest a role in ovarian cancer biology.
Results demonstrate the involvement of SCARA3 and SCARA5 in the uptake of PF14-oligonucleotide nanocomplexes.
Down-regulation of CSR1 protein expression by promoter methylation is associated with tumor growth and metastasis of prostate cancer
CSR1 appears to induce cell death through a novel mechanism by hijacking a critical RNA processing enzyme CPSF3.
This gene encodes a macrophage scavenger receptor-like protein. This protein has been shown to deplete reactive oxygen species, and thus play an important role in protecting cells from oxidative stress. The expression of this gene is induced by oxidative stress. Alternatively spliced transcript variants encoding distinct isoforms have been described.
cellular stress response gene protein
, cellular stress response protein
, macrophage scavenger receptor-like 1
, scavenger receptor class A member 3
, scavenger receptor class A, member 3
, scavenger receptor class A member 3-like