Tenascin XB (TNXB) ELISA Kits

TNXB encodes a member of the tenascin family of extracellular matrix glycoproteins. Additionally we are shipping TNXB Antibodies (44) and TNXB Proteins (4) and many more products for this protein.

list all ELISA KIts Gene Name GeneID UniProt
TNXB 7148 P22105
TNXB 81877  
TNXB 415089  
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Top TNXB ELISA Kits at antibodies-online.com

Showing 10 out of 24 products:

Catalog No. Reactivity Sensitivity Range Images Quantity Delivery Price Details
Human 0.061 ng/mL 0.15 ng/mL - 10 ng/mL 96 Tests 13 to 16 Days
Mouse 0.094 ng/mL 0.156-10 ng/mL Typical standard curve 96 Tests 12 to 14 Days
Rat 0.188 ng/mL 0.313-20 ng/mL Typical standard curve 96 Tests 12 to 14 Days
Rabbit 1.0 ng/mL 5.0-100 ng/mL   96 Tests 15 to 18 Days
Guinea Pig 1.0 ng/mL 5.0-100 ng/mL   96 Tests 15 to 18 Days
Pig 0.938 ng/mL 1.563-100 ng/mL   96 Tests 12 to 14 Days
Chicken 1.875 ng/mL 3.125-200 ng/mL   96 Tests 12 to 14 Days
Monkey 1.0 ng/mL 5.0-100 ng/mL   96 Tests 15 to 18 Days
Dog 1.0 ng/mL 5.0-100 ng/mL   96 Tests 15 to 18 Days
  96 Tests 26 to 36 Days

Top referenced TNXB ELISA Kits

  1. Human TNXB ELISA Kit for Sandwich ELISA - ABIN415102 : Kim, Woo, Suh, Ahn, Park, Hong, Lee, Ahn, Hwang, Kim, Park, Lee: Identification of vinculin as a potential plasma marker for age-related macular degeneration. in Investigative ophthalmology & visual science 2014 (PubMed)

More ELISA Kits for TNXB Interaction Partners

Human Tenascin XB (TNXB) interaction partners

  1. Data demonstrates gene expression changes in differentially methylated TNXB gene in patients with age-related macular degeneration.

  2. TNXB is a novel diagnostic biomarker for Malignant Mesothelioma

  3. mRNA for tenascin-X gene values was higher in ventricular septal defects.

  4. Hypermethylated sites at TNXB are associagted with response to starvation in anorexia nervosa.

  5. Study describes a biallelic TNXB variants in patients with congenital adrenal hyperplasia due to CYP21A2 deletions resulting in a classical Ehlers-Danlos syndrome phenotype with skin hyperextensibility, widened atrophic scars and joint hypermobility.

  6. patients with the TNX-deficient type EDS typically have generalized joint hypermobility, skin hyperextensibility and easy bruising. In contrast to the classical type, the inheritance pattern is autosomal recessive and atrophic scarring is absent. Molecular analysis of TNXB in a diagnostic setting is challenging.

  7. the identification of a rare missense variant in TNXB in combination with a positive family history of VUR and joint hypermobility may represent a non-invasive method to diagnose PVUR and warrants further evaluation in other cohorts

  8. We then quantified the tenascin-X level in serum of patients and identified tenascin-X as potent marker for ovarian cancer, showing that secretomic analysis is suitable for the identification of protein biomarkers when combined with protein immunoassay.

  9. It plays regulatory roles in collagen functions such as fibril organization and fibrillogenesis in calcific aortic valves.

  10. these results suggest that mutations in TNXB can cause hereditary primary vesicoureteral reflux .

  11. Noticeable decreased expression of tenascin-X in calcific aortic valves.

  12. Tenascin-X haploinsufficiency was associated with Ehlers-Danlos syndrome in patients with congenital adrenal hyperplasia

  13. no difference in genotype frequency was detected between patients who experienced a re-dislocation after the initial surgery and patients who did not sustain a re-dislocation.

  14. Genome-wide association study of age-related macular degeneration identifies TNXB, FKBPL and NOTCH4 as candidate susceptibility genes.

  15. Combined analysis of tenascin-C expression and the nodule size improved the prediction of malignancy in this patient cohort.

  16. rs204887 itself or a nearby variant is unlikely to play a major role in the development of schizophrenia although a cumulative contribution of rare variants in the TNXB gene cannot be ruled out.

  17. Three point mutations in TNX gene were found to be associated with hypermobility type Ehlers-Danlos syndrome (EDS) . The phenotypic effects of V1195M mutation on 7th fibronectin Type III domain (TNXfn7) with regards to EDS were investigated.

  18. molecular studies on RCCX haplotypes revealing a unique recombination giving rise to a TNXB/TNXA hybrid gene, CYP21A deletion and CYP21B duplication on one chromosome

  19. localization and analysis of the principal promoter for human tenascin-X

  20. chromosomal mapping of a de novo unequal crossover causing a deletion of the steroid 21-hydroxylase gene and a non-functional hybrid tenascin-X gene

Mouse (Murine) Tenascin XB (TNXB) interaction partners

  1. Tnx gene expression contributes to promoting TGFbeta1 mRNA expression in ocular fibroblasts and VEGF-A in macrophages, macrophage invasion, up-regulation of VEGF-A expression and neovascularization in an injured corneal stroma.

  2. these wound healing-related properties are affected in a TNX-deficient extracellular environment

  3. Results suggest that tenascin-X secreted from Schwann cells and/or fibroblasts is involved in blood vessel formation in peripheral nerves.

  4. Identification of the novel localization of tenascinX in the monkey choroid plexus and comparison with the mouse.

  5. Tenascin-x is an initiator of myocardial fibrosis and ACM development via upregulation of TGFbeta(1) and downregulation of PPARgamma.

  6. Altered properties of the force transmission pathways of muscle due to TNX deficiency directly affect muscle function in TNX KO mice. Such effects are likely to contribute to muscle weakness experienced by patients with Ehlers-Danlos syndrome.

  7. Tenascin-X deficiency mimics Ehlers-Danlos syndrome in mice through alteration of collagen deposition

  8. TNX has either a redundant or a very subtle function in the macromolecular organization in the peripheral nerve

  9. Tnx plays a role in the regulation of cell-cell and cell-matrix interactions: Tnx-null fibroblasts exhibit weaker adhesive properties to fibronectin and B16 melanoma cells than do wild-type fibroblasts.

  10. Induction of MMP-2 by Tnx deficiency is mediated through the c-Jun N-terminal kinase and protein tyrosine kinase phosphorylation pathway.

  11. TNX is unlikely to be involved in matrix deposition in the early phase of wound healing, but it is required in the later phase when remodeling and maturation of the matrix establishes and improves its biomechanical properties.

  12. TNX knockout mice have mild pregnancy-related abnormalities.

  13. localizations of Tn-X in the leptomeningeal trabecula (TB) of adult mice and in the connective tissue of the choroid plexus (CP) in the brains of mice

Cow (Bovine) Tenascin XB (TNXB) interaction partners

  1. Tenascin-X promotes activation of latent TGF-beta1 and subsequent epithelial to mesenchymal transition in mammary epithelial cells.

  2. mechanical analysis of collagen gels showed an increased compressive resistance of the gels containing tenascin-X, indicating that this protein might be directly involved in determining the mechanical properties of collagen-rich tissues in vivo.

  3. tenascin-X, via trimerization and multiple interactions with components of collagenous fibrils, plays a crucial role in the organisation of extracellular matrices.

  4. Tenascin-X is an elastic protein and the fibronectin type III (FnIII) domains can unfold under a stretching force and refold to regain their mechanical stability upon the removal of the stretching force.

TNXB Antigen Profile

Antigen Summary

This gene encodes a member of the tenascin family of extracellular matrix glycoproteins. The tenascins have anti-adhesive effects, as opposed to fibronectin which is adhesive. This protein is thought to function in matrix maturation during wound healing, and its deficiency has been associated with the connective tissue disorder Ehlers-Danlos syndrome. This gene localizes to the major histocompatibility complex (MHC) class III region on chromosome 6. It is one of four genes in this cluster which have been duplicated. The duplicated copy of this gene is incomplete and is a pseudogene which is transcribed but does not encode a protein. The structure of this gene is unusual in that it overlaps the CREBL1 and CYP21A2 genes at its 5' and 3' ends, respectively. Multiple transcript variants encoding different isoforms have been found for this gene.

Gene names and symbols associated with Tenascin XB (TNXB) ELISA Kits

  • tenascin XB (TNXB) antibody
  • tenascin XB (Tnxb) antibody
  • avian tenascin X (TNX) antibody
  • tenascin-X (LOC100434533) antibody
  • EDS3 antibody
  • HXBL antibody
  • TENX antibody
  • Tn-mhc antibody
  • TN-X antibody
  • TN-Y antibody
  • Tnx antibody
  • TNXB antibody
  • TNXB1 antibody
  • TNXB2 antibody
  • TNXBS antibody
  • XB antibody
  • XBS antibody

Protein level used designations for Tenascin XB (TNXB) ELISA Kits

growth-inhibiting protein 45 , hexabrachion-like protein , tenascin XB1 , tenascin XB2 , tenascin-X , tenascin X , tenascin XB , tenascin Y , tenascin X B , tenascin X pseudogene , tenascin-X-like

7148 Homo sapiens
81877 Mus musculus
282654 Bos taurus
396106 Gallus gallus
415089 Rattus norvegicus
462578 Pan troglodytes
100434533 Pongo abelii
100597679 Nomascus leucogenys
100682877 Canis lupus familiaris
445520 Sus scrofa
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